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Post by fritolay66 on Jan 30, 2009 22:21:17 GMT -5
Hi Guys.
I found this on another forum and for awhile now I have been wondering. It has been said several times that those Morgellons patients tested for Lyme and company, 90% or higher according to which quote you get, are testing positive.
Then add in MCS from the failures Lyme and company cause in our detox pathways, and one could very well develop lesions just like the ones in the first link at the bottom.
My son and I are walking billboards for Bartonella manifestations on the skin. If you look at the link with the rash and the depigmentation, that is me. The purple river strech marks, my son. The pimples, the lesions, the itching, the burning, the pin pricking, the rashes, stacked clam shells, we have a little of it all. You will also notice the direct reference to Morgellons.
I am curious, how many of us here are Lyme and company positive? What happens if the reason for the sluggish progress on Morgellon's is because they already know its connected to Lyme and company, also emphatically denied as well.
You know, our Morgellon's didn't improve until we started treating heavily for Lyme, Bartonella, Babesia, Candida, and MCS by treating our liver detox pathways and the P-450 enzymes.
Bartonella has been a pain in our a$$ and we continue to fight, but we have improved from a point where we could not function.
Love to All...
FritoBartonella Symptoms Bartonellosis Common symptoms of bartonellosis include: ___Fatigue (often with agitation, unlike Lyme disease, which is more exhaustion) ___Low grade fevers, especially morning and/or late afternoon, often associated with feelings of "coming down with the flu or a virus" ___Sweats, often morning or late afternoon (sometimes at night) - often described as "thick" or "sticky" in nature ___Headaches, especially frontal (often confused with sinus) or on top of head ___Eye symptoms including episodes of blurred vision, red eyes, dry eyes ___Ringing in the ears (tinnitus) and sometimes hearing problems (decreased or even increased sensitivity - so-called hyperacusis) ___Sore throats (recurring) ___Swollen glands, especially neck and under arms ___Anxiety and worry attacks; others perceive as "very anxious" ___Episodes of confusion and disorientation that are usually transient (and very scary); often can be seizure-like in nature ___Poor sleep (especially difficulty falling asleep); poor sleep quality ___Joint pain and stiffness (often both Left and Right sides as opposed to Lyme which is often on one side only with pain and stiffness that changes locations) ___Muscle pains especially the calves; may be twitching and cramping also ___Foot pain, more in the morning involving the heels or soles of the feet (sometimes misdiagnosed as plantar fasciitis) ___Nerve irritation symptoms which can be described as burning, vibrating, numb, shooting, etc. ___Tremors and/or muscle twitching ___Heart palpitations and strange chest pains ___Episodes of breathlessness ___Strange rashes recurring on the body often, red stretch marks, and peculiar tender lumps and nodules along the sides of the legs or arms, spider veins ___Gastrointestinal symptoms, abdominal pain and acid reflux ___Shin bone pain and tenderness Bartonella is a bacterium that causes illness, the most commonly known of which is a disease called "Cat Scratch Fever." Thousands of known cases of Bartonella occur in the U.S. each year, with the vast majority of known cases due to bites from fleas that infest cats or infected dogs (may also occur directly from bites and scratches from infected dogs or cats). Bartonella can also be transmitted by ticks that transmit Lyme Disease. In fact, in a study published recently, deer ticks from New Jersey had a higher prevalence of Bartonella organisms than of Lyme organisms. It is unclear whether the organism that we see transmitted along with Lyme disease is actually a Bartonella species (such as B. henselae or B. quintana) or is "Bartonella-Like Organism" (BLO) that is yet to be fully identified. While BLO has features similar to organisms in the Bartonella family, it also has features slimiar to the Mycoplasma and the Francisella (causes tularemia) families. publichealthalert.org/pdfnew/2008_05.pdfpublichealthalert.org/pdfnew/2007_08.pdf
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Post by fritolay66 on Jan 30, 2009 23:16:09 GMT -5
Found some more good stuff, authorship credit should go to TinCup.
BARTONELLA SYMPTOMS
GENERAL: Fatigue, Restlessness, Combative behavior, Myalgias, Malaise, Liver and/or Spleen involvement, Abdominal pain, Infectious Mononucleosis-like Syndrome, Granulomatous Hepatitis
BRAIN: Encephalopathy may occur 1-6 weeks after the initial infection and is fairly common in patients with Bartonella. Note: Approximately 50 percent of patients who develop Encephalopathy can be affected by seizures (from focal to generalized, and from brief and self-limited to status epilepticus). Headaches, Cognitive Dysfunction, and CNS Lesions may be evident.
RASH AND LYMPHADENITIS: Erythematous papules (red splotches or slightly raised red spots) may develop. Such papules occasionally occur on the lower limbs but are more common on the upper limbs, the head, and neck. The papules may appear on the skin or mucous membranes. Bartonella may also cause subcutaneous nodules, with some bone involvement possible. The nodules may show some hyperpigmentation, be tender, fester, and/or be enlarged or swollen, but not always.
EYES: Conjunctivitis, Bartonella Neuroretinitis, Loss of Vision, Flame Shaped Hemorrhages, Branch Retinal Artery Occlusion with Vision Loss, Cotton Wool Exudates, Parinaud’s Oculoglandular Syndrome, and Papilledema.
BONES AND MUSCLES: Osteomyelitis, Myositis, Osteolytic Lesions (softening of bone), Myelitis, Radiculitis, Transverse Myelitis, Arthritis, Chronic Demyelinating Polyneuropathy.
HEART: Endocarditis, Cardiomegaly. Possible lab findings: The following may show up during standard testing: Thrombocytopenia, pancytopenia, anemia, elevated serum alkaline phosphatase level, elevated bilirubin, abnormal liver enzymes.
X-ray of the bone may show areas of lysis or poorly-defined areas of cortical destruction with periosteal reaction. Cardiomegaly may show up on a chest X-Ray.
Biopsies of lymph nodes reveal pathology often indistinguishable from sarcoidosis. Reports of biopsies strongly suggestive of lymphoma do occur.
Tests occasionally show an enlarged liver with multiple hypodense areas scattered throughout the parenchyma.
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Post by fritolay66 on Jan 30, 2009 23:22:07 GMT -5
BARTONELLA-LIKE ORGANISMS by Joseph Burrascano
It has been said that Bartonella is the most common of all tick-borne pathogens. Indeed, there seems to be a fairly distinct clinical syndrome when this type of organism is present in the chronic Lyme patient. However, several aspects of this infection seem to indicate that this tick-associated strain of Bartonella is different from that described as "cat scratch disease". For example, in patients who fit the clinical picture, standard Bartonella blood testing is commonly non-reactive. Furthermore, the usual Bartonella medications do not work for this- they suppress the symptoms but do not permanently clear them. For these reasons I like to refer to this as a "Bartonella-like organism" (BLO), rather than assume it is a more common species.
Indicators of BLO infection include symptoms involving the central nervous system that are out of proportion to the other systemic symptoms of chronic Lyme. There seems to be an increased irritability to the CMS, with agitation, anxiety, insomnia, and even seizures, plus symptoms of encephalitis, such as cognitive deficits and confusion. Other key symptoms may include gastritis, lower abdominal pain (mesenteric adenitis), sore soles, especially in the AM, tender subcutaneous nodules along the extremities, and red rashes. These rashes may have the appearance of red streaks like stretch marks that do not follow skin planes, spider veins, or red papular eruptions. Lymph nodes may be enlarged and the throat can be sore.
Because standard Bartonella testing, either by serology or PCR, may not pick up this BLO, the blood test is very insensitive. Therefore, the diagnosis is a clinical one, based on the above points. Also, suspect infection with BLO in extensively treated Lyme patients who still are encephalitic, and who never had been treated with a significant course of specific treatment.
The drug of choice to treat BLO is levofloxacin (Levaquin). Levofloxacin is usually never used for Lyme or Babesia, so many patients who have tick-borne diseases, and who have been treated for them but remain ill, may in fact be infected with BLO. Treatment consist of 500 mg daily (may be adjusted based on body weight) for at least one month. Treat for three months or longer in the more ill patient. It has been suggested that levofloxacin may be more effective in treating this infection if a proton pump inhibitor is added in standard doses.
Another subtlety is that certain antibiotic combinations seem to inhibit the action of levofloxacin, while others seem to be neutral. I advise against combining Levaquin with an erythromycin-like drug, as clinically such patients do poorly. On the other hand, combinations with cephalosporins, penicillins and tetracyclines are okay. Alternatives to levofloxacin include rifampin, gentamicin and possibly streptomycin.
Levofloxacin is generally well tolerated, with almost no stomach upset. Very rarely, it can cause confusion and insomnia- this is usually temporary, and may be relieved by lowering the dose. There is, however, one side effect that would require it to be stopped- it may cause a painful tendonitis, usually of the largest tendons. If this happens, then the levofloxacin must be stopped or tendon rupture may occur. Unfortunately, levofloxacin and drugs in this family cannot be given to those under the age of 18, so other alternatives, such as azithromycin and/or rifampin, are used in children.
Incidentally, animal studies show that Bartonella may be transmitted across the placenta. No human studies have been done.
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Post by fritolay66 on Jan 30, 2009 23:38:19 GMT -5
www.cdc.gov/EID/content/13/12/1948.htmVolume 13, Number 12–December 2007
Dispatch Bartonella DNA in Dog Saliva
Ashlee W. Duncan,* Ricardo G. Maggi,* and Edward B. Breitschwerdt*
*North Carolina State University College of Veterinary Medicine, Raleigh, North Carolina, USA
Abstract
Bartonella species, transmitted by arthropods or animal bites and scratches, are emerging pathogens in human and veterinary medicine. PCR and DNA sequencing were used to test oral swabs collected from dogs. Results indicated the presence of 4 Bartonella species: B. bovis, B. henselae, B. quintana, and B. vinsonii subspecies berkhoffii.
Bartonella species are being recognized as increasingly important bacterial pathogens in veterinary and human medicine. These organisms can be transmitted by an arthropod vector or alternatively by animal scratches or bites (1). Among the 11 species or subspecies known or suspected to be pathogenic in humans, 8 have been detected in or isolated from pet dogs or cats, thereby highlighting the zoonotic potential of these bacteria (2).
In general, cats are implicated in the transmission of Bartonella henselae, typically resulting in cat-scratch disease; however, there have also been sporadic reports of Bartonella transmission by dogs (3–5). When B. henselae prevalence was evaluated in a population of 52 dogs, 4 dogs were seroreactive at reciprocal titers of 64 or 128, and Bartonella-positive PCR results were found in 3 of 52 blood samples, 5 of 9 oral swabs, and 5 of 9 nail clippings (5).
Based on these reports and the recent recognition of B. henselae and B. vinsonii subspecies berkhoffii bacteremia in veterinarians and veterinary technicians who experience frequent cat and dog scratches and bites (6), we speculated that Bartonella species may be present in the saliva of dogs. The purpose of this study was to determine whether Bartonella DNA could be detected in oral swabs collected from dogs.
The Study
As part of an ongoing study from November 2004 to December 2006 to investigate the prevalence of Anaplasma, Bartonella, and Ehrlichia infections in healthy golden retrievers and golden retrievers with lymphoma, a buccal swab was collected using a sterile cotton applicator. The swab was placed against the inside surface of the dog's cheek. Saliva and tissue were collected by rolling the swab firmly against the cheek. Subsequently, the swab was placed into a sterile, no additive, Vacutainer (Becton Dickinson, Franklin Lakes, NJ, USA) serum tube and allowed to air dry for 10 to 15 minutes at room temperature before the tube was recapped.
Cells on the air-dried swab were resuspended in 500 ìL of QuickExtract DNA Extraction Solution (EPICENTRE Biotechnologies, Madison, WI, USA), according to the manufacturer's instructions. Total DNA was isolated using 200 ìL of the QuickExtract resuspension, which was extracted through a QIAamp DNA Blood Mini-Kit (QIAGEN, Inc., Valencia, CA, USA) according to the manufacturer's instructions. Similarly, total DNA was extracted from 200 ìL of EDTA-anticoagulated whole blood using the QIAamp DNA Blood Mini-Kit.
Oral swabs and blood samples (n = 44 each) were screened for the presence of Bartonella by 2 previously described PCR methods (7). The first PCR targeted a fragment of the 16S-23S intergenic transcribed spacer (ITS) region; samples that were PCR positive for Bartonella DNA by the ITS primers were subsequently analyzed by a second PCR targeting the heme-binding protein gene, Pap31. Positive and negative controls were used in all processing steps, including DNA extraction. PCR amplicons were sequenced to identify species (Davis Sequencing, Davis, CA, USA). Sequence analysis and alignment with GenBank sequences were performed (AlignX, Vector NTI Suite 6.0, InforMax, Inc., Frederick, MD, USA). Additionally, serum samples were analyzed for IgG antibodies to B. henselae and B. vinsonii (berkhoffii) using an indirect immunofluorescence assay (IFA), as described previously (8). Reciprocal titers >64 were considered seroreactive.
Of the 44 dogs surveyed, oral swabs collected from 5 (11.4%) dogs were PCR-positive for Bartonella DNA. Sequencing indicated that 5 different Bartonella species or subtypes were present: B. bovis, B. henselae, B. quintana, and B. vinsonii subsp. berkhoffii types I and II (Table). PCR amplification and sequencing of blood samples from these 5 dogs showed B. henselae and B. vinsonii (berkhoffii) DNA in 2 dogs (Table). None of these 5 dogs was seroreactive to B. henselae or B. vinsonii (berkhoffii) antigens. Contamination was not detected in any of the negative control samples at any stage of processing or at any time during the study. As this work was part of an ongoing study of golden retrievers with and without lymphoma, dogs 1 and 2 had lymphoma; the remaining 3 dogs were clinically healthy (Table).
Conclusions These results demonstrate the presence of Bartonella DNA in oral swabs obtained from dogs. Notably, 3 Bartonella species and 2 B. vinsonii (berkhoffii) types were found in dog saliva. B. bovis, formerly referred to as B. weissii, was initially isolated from the blood of cats (9). Subsequently, this organism was isolated from the blood of cows in the United States, Europe, and Africa (10–12).
To our knowledge, this is only the second known report of the detection of B. bovis DNA in a sample obtained from a dog (13). All 5 dogs in this study lacked serologic evidence of Bartonella infection, a finding which has been previously reported in bacteremic dogs and humans (6,13,14).
Previous studies have shown that targeting multiple Bartonella genes provides molecular evidence of coinfection with more than 1 Bartonella species or strain type (6,7,13). In the current work, the inability to confirm the ITS PCR results with a second PCR target has been previously reported by our laboratory (6,13,14) and likely reflects differences in PCR sensitivity, interference or inhibition of the PCR reaction by oral bacteria that are present in greater numbers than the Bartonella, or the lack of a known heme-binding protein gene in various Bartonella species, such as B. bovis. The limit of detection (LOD) of Bartonella ITS PCR is 2 copies/reaction, while the LOD of Pap31 assay is 10 copies/reaction.
Further, although B. henselae has a detectable Pap31 protein (Table), several researchers in our laboratory have successfully isolated B. henselae strains that lack a PCR-detectable heme-binding protein (unpub. data). Upon recognition of the discordance between ITS and Pap31, additional genes such as 16S, gltA, and rpoB were targeted; however, these analyses were negative for Bartonella and resulted in nonspecific bacterial amplification. Because inhibition of ITS PCR was suspected due the presence of other oral bacteria, Bartonella-negative DNA extracts from oral swabs were spiked with B. henselae DNA at 1.5, 2.5, 5, and 10 (0.002 pg/ìL) copies/reaction. Inhibition was detected at up to 5 copies/reaction, while the 10 copies/reaction sample was consistently amplified by the ITS primers.
These data, in conjunction with previous case reports (3–5), suggest that potentially viable Bartonella organisms may be transmitted to humans after a dog bite. The detection of DNA by PCR does not necessarily indicate the viability of Bartonella organisms. However, due to the extremely slow growth characteristics of Bartonella spp., isolation from the oral cavity does not seem feasible, because of competition with numerous other rapidly growing oral bacterial species. Recently, Bartonella DNA has been amplified from peripheral lymph nodes of healthy dogs (14). B. henselae was also amplified from salivary gland tissues from a dog with saladenitis (15).
There are several plausible routes by which a Bartonella sp. could gain entry to the oral cavity. Future studies should determine if the tonsilar lymphoid tissues, salivary glands, or periodontal, gingival, or other oral tissues can serve as sources of Bartonella spp. contamination of canine saliva. As Bartonella infection may represent an occupational risk for veterinary professionals and others with extensive animal contact (6), additional studies should address the risk of transmission from dogs to humans following bite wounds.
Acknowledgments We acknowledge the assistance of the veterinarians who provided samples and the owners who allowed participation of their dogs in this study.
This research was funded in part by the American Kennel Club-Canine Health Foundation, Bayer Animal Health, and the State of North Carolina.
Dr Duncan recently completed her PhD in epidemiology and biotechnology in the Intracellular Pathogens Laboratory at the College of Veterinary Medicine–North Carolina State University. Her primary research interests include Bartonella species in dogs and humans.
References Boulouis HJ, Chang CC, Henn JB, Kasten RW, Chomel BB. Factors associated with the rapid emergence of zoonotic Bartonella infections. Vet Res. 2005;36:383–410. Chomel BB, Boulouis HJ, Maruyama S, Breitschwerdt EB. Bartonella spp. in pets and effect on human health. Emerg Infect Dis. 2006;12:389–94. Kerkhoff FT, Ossewaarde JM, de Loos WS, Rothova A. Presumed ocular bartonellosis. Br J Ophthalmol. 1999;83:270–5. Keret D, Giladi M, Kletter Y, Wientroub S. Cat-scratch disease osteomyelitis from a dog scratch. J Bone Joint Surg Br. 1998;80:766–7. Tsukahara M, Tsuneoka H, Iino H, Ohno K, Murano I. Bartonella henselae infection from a dog. Lancet. 1998;352:1682. Breitschwerdt EB, Maggi RG, Duncan AW, Nicholson WL, Hegarty BC, Woods CW. Bartonella species in blood of immunocompetent persons with animal and arthropod contact. Emerg Infect Dis. 2007;13:938–41. Diniz PPVP, Maggi RG, Schwartz DS, Cadenas MB, Bradley JM, Hegarty BC, et al. Canine bartonellosis: serological and molecular prevalence in Brazil and evidence of co-infection with Bartonella henselae and Bartonella vinsonii subsp. berkhoffii. Vet Res. 2007;38:697–710. Solano-Gallego L, Bradley J, Hegarty B, Sigmon B, Breitschwerdt E. Bartonella henselae IgG antibodies are prevalent in dogs from southeastern USA. Vet Res. 2004;35:585–95. Regnery R, Marano N, Jameson P, Marston E, Jones D, Handley S, et al. A fourth Bartonella species, B. weissii species nova, isolated from domestic cats. In: Abstracts of the 15th Meeting of the American Society for Rickettsiology; Captiva Island, Florida; 2000 April 30–May 3; Abstract 4. American Society for Rickettsiology; 2000. Breitschwerdt EB, Sontakke S, Cannedy A, Hancock SI, Bradley JM. Infection with Bartonella weissii and detection of Nanobacterium antigens in a North Carolina beef herd. J Clin Microbiol. 2001;39:879–82. Bermond D, Boulouis HJ, Heller R, Van Laere G, Monteil H, Chomel BB, et al. Bartonella bovis sp. nov. and Bartonella capreoli sp. nov., isolated from European ruminants. Int J Syst Evol Microbiol. 2002;52:383–90. Raoult D, La Scola B, Kelly PJ, Davoust B, Gomez J. Bartonella bovis in cattle in Africa. Vet Microbiol. 2005;105:155–6. Duncan AW, Maggi RG, Breitschwerdt EB. A combined approach for the enhanced detection and isolation of Bartonella species in dog blood samples: pre-enrichment liquid culture followed by PCR and subculture onto agar plates. J Microbiol Methods. 2007;69:273–81. Duncan AW, Birkenheuer AJ, Maggi RG, Breitschwerdt EB. Bartonella DNA detected in the blood and lymph nodes of healthy dogs. In: Abstracts of the 20th Meeting of the American Society for Rickettsiology; Pacific Grove, California; 2006 Sept 2–7; Abstract 110. American Society for Rickettsiology; 2006. Saunders GK, Monroe WE. Systemic granulomatous disease and sialometaplasia in a dog with Bartonella infection. Vet Pathol. 2006;43:391–2. Table Table. PCR, DNA sequencing, and serologic results for the 5 dogs positive for Bartonella DNA in oral swabs.
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Post by fritolay66 on Jan 30, 2009 23:54:03 GMT -5
www.cdc.gov/ncidod/EiD/vol10no7/03-0896.htmBartonella spp. DNA Associated with Biting Flies from California Crystal Y. Chung,* Rickie W. Kasten,* Sandra M. Paff,* Brian A. Van Horn,* Muriel Vayssier-Taussat,† Henri-Jean Boulouis,† and Bruno B. Chomel*Comments *University of California, Davis, California, USA; and †Unité Mixte de Recherche, Ecole Nationale Veterinaire d'Alfort, Maisons-Alfort, France Bartonella DNA was investigated in 104 horn flies (Haematobia spp.), 60 stable flies (Stomoxys spp.), 11 deer flies (Chrysops spp.), and 11 horse flies (Tabanus spp.) collected on cattle in California. Partial sequencing indicated B. bovis DNA in the horn fly pool and B. henselae type M DNA in one stable fly. Bartonella spp. are vector-borne bacteria associated with numerous emerging infections in humans and animals (1). Four Bartonella species have been isolated from wild and domestic ruminants. B. schoenbuchensis and B. capreoli were recovered from wild roe deer (Capreolus capreolus) (2,3) in Europe, whereas B. bovis (formerly B. weissii) was recovered from domestic cattle in the United States and Europe (3–5). Strains similar to B. bovis and B. capreoli were also isolated from mule deer (Odocoileus hemionus) and elk (Cervus elaphus) from California (3,4). Recently, B. chomelii was recovered from bacteremic cows in France (6). A high prevalence of infection with various Bartonella species has been reported in domestic and wild ruminants in North America and Europe (2–4). Of the herds investigated in California, 95% of beef cattle and 17% of dairy cattle were bacteremic for B. bovis and 90% of the mule deer were bacteremic for Bartonella spp. (4). The main vector of these ruminant-infecting Bartonella spp. has not been identified. The role of ticks as potential vectors for Bartonella in cattle was investigated (7,8). In Europe, >70% of 121 Ixodes ricinus ticks collected from roe deer had 16S rRNA gene sequences for Bartonella or other closely related species (7). In California, Bartonella DNA was detected in approximately 19% of 151 questing adult I. pacificus ticks (8), but the direct role of ticks in Bartonella transmission among ruminants has never been established. In a search for an efficient Bartonella vector, which could explain such high prevalence of infection in wild and domestic ruminants, we tested biting flies for Bartonella spp. DNA to establish the potential role of biting flies as vectors of Bartonella in cattle. . . . . Conclusions This identification of Bartonella DNA is the first associated with horn and stable flies and the first identification of B. henselae from a biting fly. It is also the first report of identification of Bartonella DNA from flies from North America. This finding demonstrates, as for ticks, that Bartonella DNA is present in various biting insects. We found a very low percentage of Bartonella DNA–positive flies, in contrast to the very high prevalence (57 [88%] of 65 observed in Hippoboscidae adult flies (Lipoptena cervi and Hippobosca equina) collected from domestic cattle and wild roe deer in France (H.J. Boulouis, pers. comm.). This low prevalence may be related to the fact that different fly species were tested but more likely could be associated with a low level of Bartonella bacteremia in our herds. In a previous study, only 17% of cows in a dairy herd were bacteremic (4), and prevalence was even lower in another dairy herd from Tulare, in the central valley of California (B.B. Chomel et al., unpub. data). A follow-up for this study would be to collect blood from herds at the University of California, Davis, and establish the status of Bartonella bacteremia. Future research should include collecting flies in different locations and herds in which high levels of bacteremia were previously detected. Inhibitory factors were unlikely to be associated with such a low prevalence because spiked controls were systematically detected. Identification of B. henselae DNA in a stable fly indicates the wide range of blood-sucking arthropods that can harbor this human pathogen. The partial gltA sequence was identical to that for B. henselae type Marseille, the most common type found in cats and humans in California (11). Fleas have been shown to be an efficient vector of B. henselae (12–14). More recently, B. henselae DNA was identified in adult questing I. pacificus ticks from California and from I. ricinus ticks collected on humans in Italy (8,15). The role of ticks as potential vectors of B. henselae in humans has also been suggested (16–18). Since Bartonella are likely to be present in biting flies, investigating the potential of biting flies as either mechanical or biologic vectors of Bartonella in cattle and possibly humans should be pursued.
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Post by toni on Jan 31, 2009 9:13:18 GMT -5
Great thread and tons of info, thank you Frito!
Kmarie, yes you sure have been saying this, (about the different strains depending on where one lives) or may have contracted the infection/disease.
Sure makes alot of sense for being a big part in all this.
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Post by fritolay66 on Jan 31, 2009 18:42:57 GMT -5
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Post by ibzahp on Jun 26, 2010 15:47:36 GMT -5
Thanks for this Frito, I have been found positive for Bartonella, Mycoplama, Herpes virus 6 (respitory version)eleveted liver enzymes, eppstein Barr. old hep C. doc assumes I have Lyme as well as I had to go to a quest lab instead of Igenex. The headaches and body pains from Bart are wicked. Glad you posted this information. My doc is treating with Biaxin, Bactrim, Nystatin. and then enzymes,probiotics, vitamins and minerals
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Post by fritolay66 on Jun 30, 2010 17:48:55 GMT -5
Nezi, your absolutely welcome!! I know we have talked about all this but I just now saw this. Bartonella is a wicked animal. You are on a good start and I am so happy you have an LLMD now!!!
Frito
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Post by itchin4answers on Apr 17, 2012 20:26:32 GMT -5
My results from Igenex arrived in the mail this morning, being forwarded by my doctor. I am positive for Bartonella henselae. My body is producing antibiotics for Lyme (BB). The Babebsia results are somewhat confusing, though I couldn't see through my tears. My doctor will interpret during our phone consult coming up. We need to discuss treatment. This link will explain the treatment for Bartonella and I can prove and have that these drugs do NOT cure Bartonella. en.wikipedia.org/wiki/Bartonella_henselae
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Post by itchin4answers on Apr 17, 2012 20:31:45 GMT -5
My results from Igenex arrived in the mail this morning, being forwarded by my doctor. I am positive for Bartonella henselae. My body is producing antibiotics for Lyme (BB). The Babebsia results are somewhat confusing, though I couldn't see through my tears. My doctor will interpret during our phone consult coming up. We need to discuss treatment. This link will explain the treatment for Bartonella and I can prove and have that these drugs do NOT cure Bartonella. en.wikipedia.org/wiki/Bartonella_henselae ;D ;D Hahahahhahahaha! "antibiotics" as much as I hate this disease, there are times it gives me a laugh. I sure needed that laugh right now! Antibodies is what I meant to say.
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Post by itchin4answers on Apr 17, 2012 20:59:20 GMT -5
My results from Igenex arrived in the mail this morning, being forwarded by my doctor. I am positive for Bartonella henselae. My body is producing antibiotics for Lyme (BB). The Babebsia results are somewhat confusing, though I couldn't see through my tears. My doctor will interpret during our phone consult coming up. We need to discuss treatment. This link will explain the treatment for Bartonella and I can prove and have that these drugs do NOT cure Bartonella. en.wikipedia.org/wiki/Bartonella_henselaeMites demonstrated to transmit Lyme & Bartonella tinyurl.com/77cr5vy
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