Eyes that glow in the pitch-black night make for many a scary tale. But why do some animals' eyes glow at night?
"A lot of the animals we see, especially the ones that go out at night, have a special, reflective surface right behind their retinas," says Dr. Cynthia Powell, a veterinary ophthalmologist at Colorado State University. That light-reflecting surface, called the tapetum lucidum, helps animals see better in the dark.
When light enters the eye, it's supposed to hit a photoreceptor that transmits the information to the brain, Powell explains. But sometimes the light doesn't hit the photoreceptor, so the tapetum lucidum acts as a mirror to bounce it back for a second chance.
A large number of animals have the tapetum lucidum, including deer, dogs, cats, cattle, horses and ferrets. Humans don't, and neither do some other primates. Squirrels, kangaroos and pigs don't have the tapeta, either.
I'm suspecting that most of us are now considered 'GM organisms'...?... we all need to get black lights and start looking at our world.
I am so angry at what has happened to mankind, that the evolutionist fools in charge of deciding what can go into our bodies do not consider us different from the apes and insects and have purposely created a modified human organism without our consent nor knowledge.
The fluorescence of the human lens is caused by accumulation of advanced glycation end products formed by non-enzymatic glycation on lens proteins. Advanced glycation end products are important for the pathogenesis of diabetic long-term complications. Lens fluorescence may thus serve as a biomarker of the extent of tissue-damage related to advanced glycation end products which may be of importance for the management of diabetic patients.
end cut, so still the question remains why does it glow green because it can glow red. Green glow is a marker for trans-genes. This article does suggest it can be from tissue damage but then why does my 2 year old grandson's eyes light up like a green Christmas tree bulb?
Does my 2 year old grandson already have advanced glycation of his eyes? His eyes displayed this trait rather strongly.
It also can be from protein modification which is I believe what we are looking at . I first came across this in an article saying it was a newly found compound from human lenses. It plays a role in breakdown abnormal calf proteins . This process results in crystallization of the human lens, I believe.
I once posted a thread here about color blindness, it effect the ability to see blue light properly . When I took an eye test that was what the results said, I had a rare type of color blindness to some dark shades of blue light. Blue light or the equivalent radio waves are what are being utilized in some viral brain control techniques . I do not know if that is a co-incidence but well hmmmmmmm???
Abstract A new fluorescent compound was isolated from human lens insoluble protein fraction, and was identified as 2-amino-3-hydroxyacetophenone-O-β-D-glucoside(1 Graphical Abstract A new fluorescent compound was isolated from the alkaline ethanol extract of human lens insoluble protein fraction, and was identified as 2-amino-3-hydroxyacetophenone-O-β-D-glucoside.
Porphyrias are a group of inherited or acquired disorders of certain enzymes in the heme biosynthetic pathway (also called porphyrin pathway). They are broadly classified as acute (hepatic) porphyrias and cutaneous (erythropoietic) porphyrias, based on the site of the overproduction and accumulation of the porphyrins (or their chemical precursors). They manifest with either neurological complications or with skin problems (or occasionally both). A clinically induced and histologically identical condition is called pseudoporphyria. Pseudoporphyria is characterized by normal serum and urine porphyrin levels.
The term derives from the Greek ðïñöýñá, porphyra, meaning "purple pigment".
The name is likely to have been a reference to the purple discolouration of feces and urine in patients during an attack. Although original descriptions are attributed to Hippocrates, the disease was first explained biochemically by Dr Felix Hoppe-Seyler in 1874, and acute porphyrias were described by the Dutch physician Prof B.J. Stokvis in 1889.
Cutaneous porphyria The cutaneous, or erythropoietic, porphyrias primarily affect the skin, causing photosensitivity (photodermatitis), blisters, necrosis of the skin and gums, itching, and swelling, and increased hair growth on areas such as the forehead. Often there is no abdominal pain, distinguishing it from other porphyrias.
Pathogenesis In humans, porphyrins are the main precursors of heme, an essential constituent of hemoglobin, myoglobin, catalase, peroxidase, respiratory and P450 liver cytochromes.
Heme synthesis—note that some reactions occur in the cytoplasm and some in the mitochondrion (yellow)Deficiency in the enzymes of the porphyrin pathway leads to insufficient production of heme. Heme function plays a central role in cellular metabolism.
This is not the main problem in the porphyrias; most heme synthesis enzymes—even dysfunctional enzymes—have enough residual activity to assist in heme biosynthesis. The principal problem in these deficiencies is the accumulation of porphyrins, the heme precursors, which are toxic to tissue in high concentrations. The chemical properties of these intermediates determine the location of accumulation, whether they induce photosensitivity, and whether the intermediate is excreted (in the urine or feces).
There are eight enzymes in the heme biosynthetic pathway, four of which—the first one and the last three—are in the mitochondria, while the other four are in the cytosol. Defects in any of these can lead to some form of porphyria.
The hepatic porphyrias are characterized by acute neurological attacks (seizures, psychosis, extreme back and abdominal pain and an acute polyneuropathy), while the erythropoietic forms present with skin problems, usually a light-sensitive blistering rash and increased hair growth.
Variegate porphyria (also porphyria variegata or mixed porphyria), which results from a partial deficiency in PROTO oxidase, manifests itself with skin lesions similar to those of porphyria cutanea tarda combined with acute neurologic attacks.
All other porphyrias are either skin- or nerve-predominant.
Kam and I just checked, our eyes do not glow. We both have brown eyes though, mine have a lot of green in them. My kids told me, when I get really angry they turn bright green, does that count...?..lol
My brother just send me the information on the black lights he purchased 2 for us to use, we also have a James Bare machine coming for Karen. He wants to but a scope with the computer compatible feature. Which is the best one anyone know, Digital Blue?
I have to invest in another Eye-clops my Doctor kept mine.
The green fluorescent protein (GFP) is protein composed of 238 amino acids (26.9kDa), which exhibits bright green fluorescence when exposed to blue light. Although many other marine organisms have similar green fluorescent proteins, GFP traditionally refers to the protein first isolated from the jellyfish Aequorea victoria. The GFP from A. victoria has a major excitation peak at a wavelength of 395 nm and a minor one at 475 nm
GREEN FLUORESCENT PROTEIN (GFP) GFP (Green Fluorescent Protein) is a protein produced by a jellyfish Aequorea which fluoresces in the lower green portion of the visible spectrum. The gene for GFP has been isolated and has become a useful tool for making expressed proteinsfluorescent by creating chimeric genes composed of those of GFP and its different color variants linked to genes of proteins of interest. One may thus have an in vivo fluorescent protein which may be followed in a living system. There have been several recent developments for the use of GFPand several different color variants One initial problem with the use of GFP was the excitation and emission spectra of the wild type protein for fluorescence microscopy. Wild type GFP has two excitation peaks, a major one at 395 nm (in the long UV range) and a smaller one at 475 nm (blue) and its emission peak at 509 nm (green). For wild type GFP, it has been found that exciting the protein at 395 nm causes fairly rapid quenching of the fluorescence. Also most investigators have used FITC filtersets to observe GFP staining. To alleviate this problem, several mutants of the GFP gene were constructed which have increased fluorescence, but perhaps more important, the major excitation peak has been red-shifted to 490 nm with the emission staying at 509 nm. This is better for use of FITC filtersets as this mutant GFP has the same excitation range as FITC. Furthermore, the main laserline used for FITC excitation is from the argon laser at 488nm. There is no good commonly used laserline near 395 nm in most confocals. One of the mutant GFPs which had a 5-6 times greater amount of fluorescence has a serine to threonine substitution at position 65 in the protein (S65T: See Heim, et al., Nature, 373: 663-664 (1995). Since then, other mutations have given further improvements in the brightness of the emission (See Clontech's website).
Wow Jen... can anyone take a pic of you with the black light on and upload it here? I'd love to see that... not that I love that it's happening to you. I'm sure it's scary... but don't freak out ok? Morgs love it when we do... which I know you already know.
The next time I go into town and visit my firends I'll try it on mine, they have a blacklight. I see now as I read on tho that you used a blacklight flashlight. I'll see if I can pick one up and take a pic with my camera if mine do the same.
Of the links you gave us to pics, this is normal of most animals...
This however is caused by genetic modifications...
Which of the two did your eyes look like the most?
I've held my friends blacklight over my head before and many particles show up that fluoresce. It takes a minute, but there were plenty of them in my hair. I also fluoresced at Dr. Hildy's... just never tried it on my eyes before. If I do it, I'll do it quickly as to not harm my eyes. I suppose it's the same as looking into the sun for a long time tho... not worried about it.
hugs ~~ bannanny
IF I COULD SIT ACROSS THE PORCH FROM GOD... I'D THANK HIM FOR GIVING ME YOU!
ddenial...: it feels like im loosing weight by the minute and can actually see that im getting thinner and thinner fast and i do mean scary visible fast
Dec 12, 2014 14:27:29 GMT -5
ddenial...: But nah honestly it looks and feels like im healing really really fast and that it has everything to do with the fact that my non healing lesion has been slowly but surelly been shrinking after ive been attacking it almost continiously these past 3 days
Dec 12, 2014 14:46:50 GMT -5
ddenial...: This is the first time that ive actually have come face to face with this disease and have clearly seen what damage its causing and the places that have been affected so far.
Dec 12, 2014 14:58:18 GMT -5
ddenial...: And more importantly it made it pretty clear to me what the hell kind of disease this actually is
Dec 12, 2014 15:07:56 GMT -5
ddenial...: personally im afraid that this is not limited to
Dec 12, 2014 15:09:39 GMT -5
ddenial...: my own situation but i really cannot pass any sensible judgment on this matter
Dec 12, 2014 15:14:54 GMT -5
ddenial...: but i believe its a skin disease at origin. The skin is where our health problems are formed but im not talking aout the obvious rsshes or other skin manifistations that are taking place also. Because that is actually also caused by the main problem
Dec 12, 2014 15:22:05 GMT -5
ddenial...: and thats skin growth. Caused by skin cancer in the form of melanoma.
Dec 12, 2014 15:25:42 GMT -5
ddenial...: its covering your body in thick layers of skin, grows on/in/ trough skin/mucles/fat/bone/organs if it gets the chance, causes skin damage and problems of any kind on top of that, can make skin changes, häir
Dec 12, 2014 15:31:08 GMT -5
ddenial...: changes and can cause certain aspects of the body to increase in size. For example it may severly alter the way u look when growing in the face and causing your feautures to grow out of balance etc
Dec 12, 2014 15:37:51 GMT -5
ddenial...: But i believe its likely that its
Dec 12, 2014 15:43:11 GMT -5
ddenial...: growing in a very slow tempo and that this is the reason why it gets failed to diagnose and that we fail to reckonize the changes it is making as it makes them continous in such a slow tempo we not perceive. And this also means its growth is much more
Dec 12, 2014 15:52:43 GMT -5
ddenial...: balanced and spread over big areas and will not quickly show clear signs of abnormal growths that are typical for cancer. But nonetheless it might just as well affect very large parts of the body and skin already and causing serious health problems or deat
Dec 12, 2014 16:00:57 GMT -5
Baraka Obam: This is not a SKIN disease, outward signs of disease are just that, signs that something very serious are going on inside. Cancer, do not JUMP to the last aspect of this disease so quickly, you have other things to face.
Dec 12, 2014 16:22:21 GMT -5
itchin4answers: The SKIN is the largest organ of the human body. Outward signs on the skin is an internal sign that there is something terribly wrong within - mind, body, and spirit. Since when was the "head" disconnected from the "physical body".
Dec 13, 2014 20:58:24 GMT -5
Cant read: Cant read, first blood, where did anyone say anything about a head, INSANE idiots they never stop
Dec 14, 2014 0:07:01 GMT -5