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Post by lilsissy on Mar 24, 2010 19:38:30 GMT -5
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Post by Jill on Mar 25, 2010 8:57:30 GMT -5
Thanks for that link lilsissy- confirms the work of Mark Purdey. Prions! Mangansese was found world-wide by Mark Purdey as relates to various conditions/diseases that are, in fact, one and the same as Alzheimers'/Madcow. IOW- manganese + other variables created the condition. So now we see that manganese nodules and crusts are triggered by bacteria and algae? That is the source- the reason that Alzheimers' is running rampant these days. Could Morgellons be just another name for it? We see in the Great Lakes area that algae has taken over. What triggered that? Global dimming? So one of the 'ingredients' (manganese) is produced by the process that is happening NOW- worldwide? More here: lymebusters.proboards.com/index.cgi?action=display&board=rash&thread=9496&page=1lymebusters.proboards.com/index.cgi?action=display&board=rash&thread=12563&page=1Snippet: The Manganese produces the 'abnormal' prion- in Mad Cow/Alzheimers- so I would see where they could relate this to Lyme disease. www.markpurdey.com/Scientific support for empirical observations Purdey then found that his work connected very well with the work of Dr. David Brown of Cambridge University. Dr. Brown had demonstrated that normal, healthy prion protein bonds to copper in the normal, healthy brain and nervous system. Remember, this prion protein is a normal constituent of a normal brain; it is abnormal or deformed prion proteins that are associated with spongiform encephelopathy disease.Is it the abnormal prion that causes the disease or is it some unhealthy factor in the environment that causes the prion to become deformed and abnormal and this leads to the disease? Purdey published a paper proposing that: Manganese could, in the presence of copper deficiency in the animal or human, substitute for copper in the prion protein, thus producing the abnormal prion found in the brains of spongiform encephelopathy disease victims.
This was a hypothesis based on his field work in the areas where clusters of mad cow and CJD were occurring and where, he observed, all these areas had unusually low levels of copper in the soil and unusually high levels of manganese in either the soil or the atmosphere. Purdey reports that, “David Brown ran the necessary cell-culture experiments, in which he introduced manganese into copper-depleted prion protein cell cultures.” Behold the mutated prion www.markpurdey.com/articles_educatingrida_5.htmExcerpt: It seemed that the basic cornerstone of the environmental theory was beginning to establish itself, and an overall picture of the pathogenesis of this disease was panning out. That a high manganese/low copper imbalance somehow compromised the brain’s ability to deal with acute shock bursts of sound and light – the other common characteristics of TSE ecosystems.www.ourcivilisation.com/madcow/madcow.htmAn Explanation Of Mad Cow Disease by Mark Purdey Fair use Excerpt: Hard evidence has been amassed so far which indicates that vCJD and BSE could both result from separate exposure of cattle and humans to the same set of toxic environmental factors—excess manganese and oxidizing agents—and not from the ingestion of beef by humans and animal by-products by cows. If this hypothesis continues to accumulate corroborative evidence, a radical upheaval of the status quo mindset can be expected.end excerpt Excerpt: Prion protein is expressed in other tissues of the body which are also interconnected to the network that conducts electromagnetic energy, for instance in the spleen, lymphatic system, glial cells and nerve-growth-factor-mediated stem cells that proliferate during the growth and repair of neurons.In this respect, the suggestion of an electro-conducting function of the copper prion protein may turn out to give further scientific substance to the existence of the electromagnetic meridians recognized by Chinese medicine, where the healthy copper prion performs a regulatory role in maintaining the electro-homeostasis along these meridians. The hypothesis was falling into place: copper prions as the conductors and manganese prions as the blockers of electromagnetic energy flow. The fact that copper is used in wires that carry electric currents, whereas manganese is used in batteries and light bulb filaments that store electrical energy, helps explain the underlying cause of prion diseases: healthy copper prions conduct the vital electro-energy of sunlight along the circadian pathways that innervate deep into the brain—in order to maintain the balanced cycles of sleep, sex and behavior whilst aberrant manganese-contaminated prions blockade and store up that incoming UV energy to an explosive flash point—to a level that detonates off neuropathogenic cluster bombs of free radical chain reactions along the circadian pathways.With an overabundance of manganese prions and loss of copper prions, the oxidative impact of UV energy received at the retina can no longer be quenched. Consequently, the energy flow of UV piles up, finding itself misappropriated into converting the accumulated store of innocuous manganese 2+ ( antioxidant ) into its lethal manganese 3+ or 4+ form (pro-oxidant). So any accumulations of abnormal manganese prion protein in the retina finds itself transformed from a safe to a lethal form. In this respect eco-oxidants such as UV serve to unleash a kind of "Jekyll and Hyde" effect in the manganese-contaminated, copper-depleted mammal, which, in turn, kicks off a whole chain reaction of free radical assault on the central nervous system—ultimately resulting in a neurodegenerative meltdown that leads to spongiform disease.
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Post by lilsissy on Mar 25, 2010 22:58:06 GMT -5
You got me with that one jill, wow
did you know that Bush put prions on the bio terror list in 2003, if a scientist speaks about them he may face treason charges. Pancreatic cancer has undigested abnormally folded Ppr same as Scrapie. Scrapie prions can enter through the skin , that's why they call it scrapie from the scrapes on the skin of infected lambs.
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Post by lilsissy on Mar 25, 2010 23:16:27 GMT -5
With an overabundance of manganese prions and loss of copper prions, the oxidative impact of UV energy received at the retina can no longer be quenched. Consequently, the energy flow of UV piles up, finding itself misappropriated into converting the accumulated store of innocuous manganese 2+ ( antioxidant ) into its lethal manganese 3+ or 4+ form (pro-oxidant). So any accumulations of abnormal manganese prion protein in the retina finds itself transformed from a safe to a lethal form.
My daughter is having trouble, the U.V. lights at school bother her just like they have always bothered me. Last week the lights at school put her into a tizzy , she kept thinking her contact was not right and went into the bathroom to fix it and was astonished to see one huge pupil and one small pupil with one red cheek only. We rushed her into emergency , the did a cat scan but they where stumped and later called back and they want to get her in soon for an M.R.I. I am very concerned for her . The migraines she has may have caused her to have vessel damage , she has had these since 14 when she became ill with stomach problems . She has had that one red cheek for some time but I just recently learned about it. She has had two spontaneous miscarriages and enlarged ovary and a very rapid 40 pound weight gain.
The nurse at the emergency became angry with me for shutting the curtain as the light was to much for her and made me leave so I never had the chance to talk to the doctor.
Wow , now I understand the Florescent light , why they make some of us sleepy then confused and anxious, overload
wow Jill that is good information. I am going to save this thread.
Did you see the threads I did on prions?
Jen
I just walked over to her house and had her husband put the U.V. light sunglasses in her purse for school tomarrow. Please pray for her all.
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Post by lilsissy on Mar 25, 2010 23:45:18 GMT -5
I have been looking for this, lymebusters.proboards.com/index.cgi?board=rash&action=display&thread=9583&page=4-------------------------------------------------------------------------------- Medical Hypotheses : Elevated silver, barium and strontium in ...The resulting Ag, Ba, Sr or Mn based compounds seed piezoelectric crystals which ...... In: A. Foldes and R. Reiter, Editors, Advances pineal research vol. ... linkinghub.elsevier.com/retrieve/pii/S0306987704001331 - Similar pages - Note this I revised this to include title.Lilsissy here is the article, Abstract High levels of Silver (Ag), Barium (Ba) and Strontium (Sr) and low levels of copper (Cu) have been measured in the antlers, soils and pastures of the deer that are thriving in the chronic wasting disease (CWD) cluster zones in North America in relation to the areas where CWD and other transmissible spongiform encephalopathies (TSEs) have not been reported. The elevations of Ag, Ba and Sr were thought to originate from both natural geochemical and artificial pollutant sources – stemming from the common practise of aerial spraying with ‘cloud seeding' Ag or Ba crystal nuclei for rain making in these drought prone areas of North America, the atmospheric spraying with Ba based aerosols for enhancing/refracting radar and radio signal communications as well as the spreading of waste Ba drilling mud from the local oil/gas well industry across pastureland. These metals have subsequently bioconcentrated up the foodchain and into the mammals who are dependent upon the local Cu deficient ecosystems. A dual eco-prerequisite theory is proposed on the aetiology of TSEs which is based upon an Ag, Ba, Sr or Mn replacement binding at the vacant Cu/Zn domains on the cellular prion protein (PrP)/sulphated proteoglycan molecules which impairs the capacities of the brain to protect itself against incoming shockbursts of sound and light energy. Ag/Ba/Sr chelation of free sulphur within the biosystem inhibits the viable synthesis of the sulphur dependent proteoglycans, which results in the overall collapse of the Cu mediated conduction of electric signals along the PrP-proteoglycan signalling pathways; ultimately disrupting GABA type inhibitory currents at the synapses/end plates of the auditory/circadian regulated circuitry, as well as disrupting proteoglycan co-regulation of the growth factor signalling systems which maintain the structural integrity of the nervous system. The resulting Ag, Ba, Sr or Mn based compounds seed piezoelectric crystals which incorporate PrP and ferritin into their structure. These ferrimagnetically ordered crystals multireplicate and choke up the PrP-proteoglycan conduits of electrical conduction throughout the CNS. The second stage of pathogenesis comes into play when the pressure energy from incoming shock bursts of low frequency acoustic waves from low fly jets, explosions, earthquakes, etc. (a key eco-characteristic of TSE cluster environments) are absorbed by the rogue ‘piezoelectric' crystals, which duly convert the mechanical pressure energy into an electrical energy which accumulates in the crystal-PrP-ferritin aggregates (the fibrils) until a point of ‘saturation polarization' is reached. Magnetic fields are generated on the crystal surface, which initiate chain reactions of deleterious free radical mediated spongiform neurodegeneration in surrounding tissues. Since Ag, Ba, Sr or Mn based piezoelectric crystals are heat resistant and carry a magnetic field inducing pathogenic capacity, it is proposed that these ferroelectric crystal pollutants represent the transmissible, pathogenic agents that initiate TSE.
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Post by Jill on Mar 26, 2010 15:35:20 GMT -5
Good article you posted- goes to Mark Purdey's findings. In fact, that IS Mark Purdeys' article. The crystals = organophosphates Med Hypotheses. 2004;63(2):211-25. Elevated silver, barium and strontium in antlers, vegetation and soils sourced from CWD cluster areas: do Ag/Ba/Sr piezoelectric crystals represent the transmissible pathogenic agent in TSEs? Purdey M. High Barn Farm, Elworthy, Taunton, Somerset TA4 3PX, UK. tsepurdey@aol.com Fair use Excerpt: High levels of Silver (Ag), Barium (Ba) and Strontium (Sr) and low levels of copper (Cu) have been measured in the antlers, soils and pastures of the deer that are thriving in the chronic wasting disease (CWD) cluster zones in North America in relation to the areas where CWD and other transmissible spongiform encephalopathies (TSEs) have not been reported. end The metals- Silver, Barium and the Strontium combined with low levels of copper (manganese and zinc displace copper in the liver**) combined with other components (such as organophosphates) create the the mutant prions of Madcow/Alzheimers. Mark described other combinations that create the 'mutant' prion but all contain high levels of manganese. Re: Manganese displaces copper in liver www.arltma.com/CopperToxDoc.htmKlinghardt offers help- addresses pineal issues- link below- component 2 tinyurl.com/y9fmy56Fair use Excerpt: Central inhibition of the pineal gland, hypothalamus and pituitary gland is almost always an issue that has to be resolved somewhat independently from treating the infection. Furthermore, biotoxins from the infectious agents have a synergistic effect with heavy metals, xenobiotics and thioethers from cavitations and NICO lesions in the jaw and from root filled teeth. My published neurotoxin elimination protocol can be downloaded for free (6). end excerpt on the above link- scroll down to the portion where Klinghardt states that mold and fungi are always a part of the picture. There is a mention of Dr Zhang's protocol in Klinghardt's links. The Coca Cola company dispersed wastewater into the fields surrounding their Paw Paw facility. It was found (years later) that there was a negative impact on the groundwater. Monitoring turned up high levels of iron, manganese and arsenic. www.michigan.gov/documents/deq/wb-gwdischarge-CCNA-FS_282752_7.pdfThe heavy metals in the air due to industry in my area- mostly car manufacture- has the chemicals in terms of #'s in the air- off the charts. Manganese @ 333,692 #'s Lead 23,642 Zinc 416,842 Barium 3,919 Chronium 15,145 and so on. Car manufacturing areas have the highest rates.
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Post by kmarie on Mar 26, 2010 15:40:15 GMT -5
OMG, Jill and Lilsissy,....................... I am still in shock at all this and don't know why when I try to TELL PEOPLE the "TRUTH", they refuse to believe all this is taking place in this GREAT United States of America.
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Post by lilsissy on Mar 26, 2010 20:03:48 GMT -5
I was just told, My neighbor 2 doors down has had an aneurysm yesterday, She was operated on and is recovering . Jerry (lady) is 40 something. My daughter 19 lives directly across the street and appears to be threatening an aneurysm ( 1 week ). What is the odds 2 people in 1 week so young within 100 feet of each another having serious brain vessel complications . I was told by Jerry's sister who is a good friend that there was another lady from our neighborhood at the hospital yesterday with an aneurysm. Bush put prions on the bio terror list in 2003, is it in my water? www.medicalnewstoday.com/articles/181385.php
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Post by lilsissy on Mar 26, 2010 20:11:37 GMT -5
From Lifeboat Foundation, described here « Message sent Today at 2:27am »
-------------------------------------------------------------------------------- is verichip with flu sensor and next sounds like morgellons, W.H.O. agenda?
From your link
TECHNOLOGIES TO COMBAT BIOLOGICAL VIRUSES
One technology to develop is RNAi-based viral suppression. Also, further strategies for battling viral infections are being developed by biotech and pharma companies, such as research programs focusing on the use of decoy oligonucleotides, aptamers, and other small molecules such as peptides and glycopeptides to inhibit viral fusion with human cell membranes or function. These technologies are new and largely unproven so the important and definitive times are ahead.
Other technologies that should be developed include:
1) Development of rapid detection and identification technology: technologies such as these are being developed, based on electrostatic interactions with unmodified gold nanoparticles, silicon transistors (also described in DNA detection made easy), or DNA pairing with a single strand DNA tethered to an enzyme which becomes activated upon binding to the complementary strand.
2) Development of "smart" materials such as antiviral surface coatings that are being tested for use in face masks and other applications.
3) Further advances in sequencing technologies, ultimately reaching a target of full virus sequencing within hours. As mentioned, identification of the virus used for the development of a vaccine or other treatment for an unknown virus necessitates the rapid sequencing of its entire DNA or RNA genome. Sequencing technology is in widespread use and is constantly decreasing in cost per segment sequenced as well as in the time taken for the sequencing.
The relevant outcomes of development in this field will reduce sample preparation time as well as expand the diversity of materials useful for isolation of viruses to be sequenced (blood, saliva, skin, mucosa). As faster sequencing times and better sequence assembly software are constantly being developed the need for these measures to be specifically undertaken is of lesser importance. Examples for emerging rapid sequencing technologies include nanopore-based sequencing, sequencing based on nano-scale electronic and photonic effects, and sequencing performed using microarray-based fluorescently-tagged polymerase and nucleotides.
4) Software-based treatment design. A longer term and expensive (though ultimately valuable) avenue of research, which would be useful in a variety of medicinal applications, would be the development of a comprehensive software system able to analyze the genetic makeup of a virus as well as the proteins it expresses (its proteome), which could provide specific epitopic or conformational targets to interfere with the production, processing and function of these molecules.
The initial identification of viruses susceptibilities would help determine the most likely effective antiviral treatments based on DNA, RNA, or protein-based interference strategies. Software-based strategies should also allow the identification of the optimal protein sequences to be used as a vaccine, and be able to accelerate the "good guys" response in the "arms race" as further bioengineered, malicious pathogens are developed.
ENGINEERED BACTERIA AND PRIONS
Infectious human viruses are almost always either airborne or spread by direct person to person contact. The first mode of propagation of deadly rapidly infective agents would be potentially suicidal in the global sense for a terrorist group or nation. Such infections know no boundaries imposed by nations or ideologies. (It would be possible for a nation or terrorist group with sufficient resources to produce a vaccine that would protect them against such a release... A scary scenario was played out in fiction a few years ago in Rainbow Six by Tom Clancy.) The second mode would be far too slow in any event and good public health counter-measures already exist for slowly propagating infectious agents.
Because it would be suicidal for a terrorist group or nation to use airborne infectious viruses, they may decide to use engineered bacteria or prions instead. (Although suicidal terrorists do exist!) To combat these threats, we propose frequent testing of the water supply, not just for known bacteria but for the biologically necessary consensus DNA sequences that would be present even in engineered organisms. All known toxin-producing sequences should be tested for as well.
We also propose more extensive testing of the meat supply for prion sequences and we are definitely against the current government regulations which prohibit meat processors from doing extra prion tests at their own expense! This testing would be expensive but we are currently doing way too little of it. Additionally, testing air in cities would be useful.
Note that technologies like PCR get cheaper every day and large scale testing of this kind would further reduce the per test cost.
We support development of the prion blood test being developed by Claudio A. Soto's group. This new test is a million times more sensitive than conventional antibody-based techniques for detecting prions.
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Post by lilsissy on Mar 26, 2010 20:15:20 GMT -5
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Post by lilsissy on Mar 26, 2010 20:18:50 GMT -5
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Post by Jill on Mar 26, 2010 22:54:34 GMT -5
Agree, Kmarie- things are not looking good. But then you look at the repression other places in the world and maybe it's not as bad ? Yet ? Howard Ruff, economist had stated many years ago that the next depression will make 1929 look like a walk in the park. I think he is right and I think we are on GO. Obvious there were pressing reasons for the health bill to be in place right now. Guess we know a few. On the bright side (?) - we know ways to treat for nanobacteria. EDTA Chelation therapy, tetracycline and Nutraceuticals. As to the prions- mutant or lab produced/engineered, that will take some time to figure out. There are new treatments for Alzheimers- just heard an ad the other day. NO cure yet and unlikely to ever be a cure in this lifetime. A prion is: en.wikipedia.org/wiki/PrionFair use Excerpt: A prion is an infectious agent that is composed primarily of protein.[2] To date, all such agents that have been discovered propagate by transmitting a mis-folded protein state; as with viruses the protein itself does not self-replicate, rather it induces existing polypeptides in the host organism to take on the rogue form.[3] The misfolded form of the prion protein has been implicated in a number of diseases in a variety of mammals, including bovine spongiform encephalopathy (BSE, also known as "mad cow disease") in cattle and Creutzfeldt–Jakob disease (CJD) in humans. All known prion diseases affect the structure of the brain or other neural tissue, and all are currently untreatable and universally fatal.[4] In general usage, prion refers to the theoretical unit of infection. In scientific notation, PrPC refers to the endogenous form of prion protein (PrP), which is found in a multitude of tissues, while PrPSc refers to the misfolded form of PrP, that is responsible for the formation of amyloid plaques[5] and neurodegeneration.[6] The precise composition of the prion is not known, though they can be formed by combining PrPC, polyadenylic acid, and lipids.[7] end excerpt What a dilemma. Yeah, lilsissy, seems every third person I know is sick with something really awful. I posted Klinghardts material above because in his Component #2, he mentions the pineal gland hypothalamus and pituitary gland and then mentions his neurotoxin elimination protocol that is free. If this protocol were to be followed, based on neurotoxin elimination (prions/Bb/whatever) it would sure be worth a try. It seems that his plan is like that of the Nanobacteria folk (above), less the chelation. Worth a try? tinyurl.com/y9fmy56Excerpt: Fair use Component #2: The illness producing effect of microbial exo- and endotoxins Most of these are neurotoxins, some appear to be carcinogenic as well, others block the T3 receptor on the cell wall, etc. Decreased hormonal output of the gonads and adrenals is a commonly observed neurotoxin mediated problem in Lyme patients. Central inhibition of the pineal gland, hypothalamus and pituitary gland is almost always an issue that has to be resolved somewhat independently from treating the infection. Furthermore, biotoxins from the infectious agents have a synergistic effect with heavy metals, xenobiotics and thioethers from cavitations and NICO lesions in the jaw and from root filled teeth. My published neurotoxin elimination protocol can be downloaded for free (6). We use toxin binding agents such as fiber rich ground up raw vegetables, chlorella, cholestyramine ( 13 ), beta-Sitosterol, propolis powder, apple pectin and mucuna bean powder ( 14 ). A solid heavy metal detoxification program should be used simultaneously with the first phases of the Lyme treatment. Safe toxic metal elimination is an art unto itself. However, the information is widely available now( 15 ). end excerpt
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