|
Post by godsgrace on Sept 3, 2008 9:26:49 GMT -5
perhaps the MTBE in the air is responsible for the depletion of the ozone causing global warming? Have a hard time believing it is carbon dioxide which we release when we breathe and plants use!!!!! BS gg
|
|
|
Post by Jill on Sept 4, 2008 20:31:56 GMT -5
There is a man that lived in California that claims that the MTBE's- while they were in use- did alter the weather. He had an elaborate theory-
I don't know if it did alter the weather (when consumed in cars/jets) but I am learning what it is doing now.
As to the Global warming- don't know about that either. According to scientists from various places- worldwide- it's more like Global Dimming- due to the metals in the atmosphere that won't let the suns rays come thru.
A special that I watched made claims that various cities had nearly 10 % dimming (reduction in sunlight).
That makes sense to me.
|
|
|
Post by godsgrace on Sept 4, 2008 22:45:08 GMT -5
the dimming is caused from the chemtrails.
heavy metals being sprayed as well as chemicals, viruses, sedatives and many other components.
barium aluminum titanium
just a few off the top of my head..
gg
|
|
|
Post by Jill on Sept 6, 2008 15:44:31 GMT -5
Exactly, Godsgrace- right as rain!
|
|
|
Post by godsgrace on Sept 6, 2008 16:55:18 GMT -5
;D
love ya babe
keep thinking keep reading keep posting
godsgrace
|
|
|
Post by lilsissy on Sept 6, 2008 23:54:18 GMT -5
|
|
|
Post by Jill on Sept 7, 2008 12:07:09 GMT -5
Could be- Lilsissy- but I'd bet that the stuff in our drinking water, soil (ergo:food) and air have more to do with the fibers than filaments in the upper atmosphere. If it were proven that these filaments could find their way to earth- and then proven that they contained bacteria/fungus/agrobacterium and the other components found in the Morgellons patients by researchers- then I would say it was so- but so far, we just know there are filaments out there.
EMF effects everything- much less filaments
We are ingesting something- that is then processed- in our bodies.
Via- water/food/air
The only thing that makes total sense is the MTBE/TBA and the remediation products. They are in our water/food/air
And- they contain: bacteria/fungus (filamentous- no less)/agrobacterium/the whole 9-
It is a fit- a match- 100%
|
|
|
Post by Jill on Sept 7, 2008 15:48:22 GMT -5
MTBE- Evaluation of evidence for infection as a mode of action for induction of rat lymphoma Jane C. Caldwell *, Jennifer Jinot, Danielle DeVoney, Jeff S. Gift National Center for Environmental Assessment, U.S. Environmental Protection Agency, Washington, DC email: Jane C. Caldwell (caldwell.jane@epa.gov) *Correspondence to Jane C. Caldwell, National Center for Environmental Assessment, U.S. Environmental Protection Agency, Maildrop B105-07, 109 T.W. Alexander Drive, Research Triangle Park, North Carolina 27711 This manuscript has been reviewed by the U.S. Environmental Protection Agency and approved for publication. The views expressed in this study are those of the author and do not necessarily reflect the views or policies of the U.S. Environmental Protection Agency. This article is a US Government work and, as such, is in the public domain in the United States of America.
Keywords methyl tertiary-butyl ether • lymphoma • lymphoblastic leukemia • mode of action • risk assessment
Abstract The European Food Safety Authority (EFSA) released a 2006 report questioning the relationship of aspartame exposure with increased incidence of lymphomas/leukemias in a European Ramazzini Foundation (ERF) rat study. The EFSA report suggested that the lymphoma/leukemia findings were most likely explained by infection in the rat colony. The ERF has also conducted the only available long-term oral study of methyl tertiary-butyl ether (MTBE). Thus, using the EFSA report as support, some have now raised questions about the human relevance of MTBE-associated hemolymphoreticular tumors reported by the ERF in female rats as well as whether their incidence was elevated above background levels. In this report, we discuss the hypothesized mode of action (MOA) of infection-induced lymphoma and its relevance to MTBE-associated lymphomas. We address the relationship of rat strain and study duration to lymphoma susceptibility and review evidence of low background rates of this tumor in control animals at the ERF, similar survival rates for female rats at the ERF and National Toxicology Program (NTP), and chemical- and gender-specificity of tumor induction for this type of tumor in studies at the ERF. We find that the background incidence of hemolymphoreticular tumors in female rats in the MTBE study is consistent with contemporaneous studies at the ERF and that there is an exposure-related effect, which is unlikely to be due to infections. We examine more recent tumor classification schemes for lymphomas, which support the combination of lymphoblastic leukemias and lymphomas reported by Belpoggi et al. ([1995] Toxicol Ind Health 11:119-149; [1998] Eur J Oncol 3:201-206). Environ. Mol. Mutagen., 2008. Published 2007 Wiley-Liss, Inc. Received: 29 May 2007; Revised: 12 September 2007; Accepted: 5 October 2007Digital Object Identifier (DOI) 10.1002/em.20356 About DOI
www3.interscience.wiley.com/journal/117869061/abstract?CRETRY=1&SRETRY=0
|
|
|
Post by Jill on Sept 7, 2008 17:20:28 GMT -5
Toxicity of MTBE to Freshwater Organisms Investigators: David E. Hinton, Ph.D, Principal Investigator School of Veterinary Medicine: Anatomy, Physiology & Cell Biology, UC Davis Cory Koger, Graduate Student Inge Werner, PhD, Postdoctoral Researcher School of Veterinary Medicine: Anatomy, Physiology & Cell Biology, UC Davis Abstract Text: Increased input of the fuel oxygenate MTBE into aquatic systems has led to concerns about its effect(s) on aquatic life. As part of a study conducted by University of California scientists for the State of California, the Aquatic Toxicology Laboratory, UC Davis, reviewed existing literature on toxicity of MTBE to freshwater organisms. Then, we derived information on potential toxicity of MTBE to California resident species, and conducted chronic, developmental toxicity assays in fish. Depending on time of exposure and endpoint measured, MTBE was found to be acutely toxic to various aquatic organisms at concentrations of 44 - >1000 mg/L in invertebrates, and 388 - >3000 mg/L in vertebrates. Developmental effects in medaka (Oryzias latipes) were not observed at concentrations up to 480 mg/l, and all fish hatched and performed feeding and swimming in a normal manner. Bacterial assays proved most sensitive with toxicity to Salmonella typhimurium measured at 7.4 mg/L within 48 hours. In microalgae, decreased growth was observed at 2,400 mg/L and 4,800 mg/L within 5 days. MTBE does not appear to bioconcentrate in fish and is rapidly excreted or metabolized. Collectively, the available bioassay data suggests that at the commonly observed environmental MTBE exposure levels found in surface waters (<100 parts per billion) the compound is likely not be toxic to aquatic life. [/b] MTBE Research Research Area: Funded By: Other TSR&TP Funding Abstract Source: 1999 Symposium Presenter: Inge Werner perc.ucdavis.edu/abs_search/FMPro?-db=abstracts.fp5&-format=record%5fdetail.htm&-lay=web%5fmain&-sortfield=title&-op=eq&bPublish%5fIt=1&-op=cn&Research%5fArea=MTBE%20Research&-max=20&-recid=34107&-find=
|
|
|
Post by Jill on Sept 7, 2008 17:24:39 GMT -5
per above:
Salmonella enterica From Wikipedia, the free encyclopedia (Redirected from Salmonella typhi) Jump to: navigation, search Salmonella enterica
S. enterica Typhimurium colonies on a Hektoen enteric agar plate Scientific classification Kingdom: Bacteria Phylum: Proteobacteria Class: Gamma Proteobacteria Order: Enterobacteriales Family: Enterobacteriaceae Genus: Salmonella Species: S. enterica Binomial name Salmonella enterica (ex Kauffmann & Edwards 1952) Le Minor & Popoff 1987
Salmonella enterica is a rod shaped, flagellated, Gram-negative bacterium, and a member of the genus Salmonella.[1] Serovars S. enterica has an extraordinarily large number of serovars or strains—up to 2000 have been described.[2] Salmonella enterica Serovar Typhi (historically elevated to species status as S. typhi) is the disease agent in typhoid fever. Other serovars such as Typhimurium (also known as S. typhimurium) can lead to a form of human gastroenteritis sometimes referred to as salmonellosis.The genome sequences of serovars Typhi[3] and Typhimurium LT2[4] have been established. Also an analysis of the proteome of Typhimurium LT2 under differing environmental conditions has been performed [5].
|
|
|
Post by Jill on Sept 7, 2008 17:33:07 GMT -5
Another method of remediation: Sorption for Removing Methyl Tertiary Butyl Ether (MTBE) from Drinking Water Fair use Investigators: Irwin (Mel) H. Suffet, Ph.D., Professor Environmental Sciences and Engineering Program, UC Los Angeles Irwin (Mel) H. Suffet, Ph.D., Professor Environmental Sciences and Engineering Program, UC Los Angeles Eakalak Khan, Ph.D., Postdoctoral Research Associate Xinling Ouyang, Master’s Student Wei Rong, Doctoral Student Department of Environmental Health Sciences, UC Los Angeles Abstract Text: Methyl tertiary butyl ether (MTBE), the most common oxygenated fuel additive, has been detected in several drinking water supplies in California. MTBE levels as high as 610 ppb in the groundwater of the City of Santa Monica were reported. An action level of 35 ppb for drinking water has been established by the State of California Department of Health Services. In addition to the risk associated with human exposure through drinking water, unpleasant taste and odor should not be detected under this action level. Removal of MTBE from drinking water can be achieved through several water treatment processes such as air stripping with adsorption of collected material, advanced oxidation, membrane separation, and sorption. Since MTBE is highly water soluble polar compound, high air to water ratios are required in air stripping process. Consequently, the use of the process may not be economical. Membrane separation is considered too expensive for the use in general water treatment applications. Advanced oxidation processes, such as ozone and ozone/peroxide, are very effective in removing MTBE from drinking water; however, bromate is a by-product of these processes. The objective of this research is to determine the cost and effectiveness of sorption process to remove MTBE from drinking water.[/color] Currently, isotherm studies at low to moderate concentrations of MTBE (< 1000 ppb) adsorption, are being conducted. Sorbents used in these studies include granular activated carbons (bituminous coal, wood, peat, and coconut), XAD-4 and XAD-8 resins (Amberlite), and XE-572 resin (Ambersorb). Non-traditional sorbents, such as modified clay materials, will also be tested. Competitive studies will be conducted with humic materials and two major oxidation by-products: tertiary butyl alcohol (TBA) and tertiary butyl formate (TBF). The evaluation of sorbents will also consider their use as a polishing method for advanced oxidation system. Based on the results from isotherm studies, effective sorbents will be studied using the laboratory scale rapid small-scale column tests (RSSCT). RSSCT can simulate the full scale granular activated carbon column performance and the RSSCT results can be used to scale up the columns as well as to evaluate the cost of the process. Research Area: MTBE Research Funded By: Toxic Mechanisms Lead Campus Program Abstract Source: 1998 Symposium Presenter: Eakalak Khan perc.ucdavis.edu/abs_search/FMPro?-db=abstracts.fp5&-format=record%5fdetail.htm&-lay=web%5fmain&-sortfield=title&-op=eq&bPublish%5fIt=1&-op=cn&Research%5fArea=MTBE%20Research&-max=20&-recid=34052&-find=
|
|
|
Post by Jill on Sept 7, 2008 17:34:38 GMT -5
Bromate- per the above- from the wiki:
Bromate From Wikipedia, the free encyclopedia Jump to: navigation, search A space-filling model of the bromate anionThe bromate anion, BrO3-, is a bromine-based oxoanion. A bromate is a chemical compound that contains this ion. Examples of bromates include sodium bromate, (NaBrO3), and potassium bromate, (KBrO3).
Bromates are formed many different ways in municipal drinking water. The most common is ozone and bromide ion react according to the following abbreviated reaction:
Br- + O3 ¡ú BrO3- Electrochemical processes, such as formation of hypochlorite, will also form bromate, when bromide ion is present in the brine solution used to make hypochlorite.
Photoactivation will also promote chlorine dioxide or (recently, see below) liquid or gaseous chlorine to also produce bromate in bromide-containing water, when exposed to sunlight
Bromate in drinking water is undesirable because it is a suspected human carcinogen.[1][2] The presence of it in Coca Cola's Dasani forced a recall of that product in the UK.[3] Proposals to reduce bromate formation include switching to enclosed atmospheric tank contact systems, lowering the water pH to between 5.9 - 6.3, and limiting the doses of ozone.
In laboratories bromates can be synthesized by dissolving Br2 in a concentrated solution of potassium hydroxide (KOH). The following reactions will take place:
Br2 + 2 OH- ¡ú Br- + BrO- + H2O 3 BrO- ¡ú BrO3- + 2 Br-
[edit] Reservoir pollution On December 14, 2007, it was announced by the Los Angeles Department of Water and Power (LADWP) that the Silver Lake and Elysian reservoirs were going to be drained due to bromate contamination. Bromate usually forms when bromide is purified using ozone, a method used at filtration plants. In the case of the Silver Lake and Elysian reservoirs, however, a combination of bromide from well water, chlorine and sunlight formed bromate. The decontamination took 4 months and resulted in the discharge of over 600 million gallons of contaminated water.[4]
On June 9, 2008 the LADWP began covering the surface of the 10 acre, 58 million gallon open Ivanhoe reservoir with black, plastic balls to block the sunlight which causes the naturally present bromide to react with the chlorine used in treatment. It will require 30 million of the 40 cent balls to cover the Ivanhoe and Elysian reserviors.[5]
[edit] References ^ "Potassium Bromate (Group 2B)". International Agency for Research on Cancer: Summaries and Evaluations. Canadian Centre for Occupational Health and Safety. Retrieved on 2008-03-09. ^ Kurokawa, Yuji (July 1990). "Toxicity and carcinogenicity of potassium bromate--a new renal carcinogen". Environmental Health Perspectives 87: 309¨C335. doi:10.2307/3431039. PMID 2269236. Retrieved on 2008-03-09. ^ "Coke recalls controversial water", BBC News (2004-03-19). Retrieved on 2008-03-09. ^ "DWP To Drain 2 Reservoirs After Potentially Harmful Chemical Found", KNBC News (2007-12-14). Retrieved on 2008-03-09. ^ "DWP drops 400,000 balls onto Ivanhoe Reservoir". Retrieved from "http://en.wikipedia.org/wiki/Bromate" Categories: Oxoanions | Bromates
|
|
|
Post by Jill on Sept 7, 2008 17:53:23 GMT -5
Biodegradation of MTBE and Toluene by Bacterial Strain PM1 Fair use Investigators: Jessica R. Hanson, EdM, Doctoral Student Department of Land, Air and Water Resources, UC Davis Kate M. Scow, PhD, Mentor Professor Department of Land, Air and Water Resources, UC Davis Corinne E. Ackerman, Undergraduate Student Department of Land, Air and Water Resources, UC Davis Abstract Text: The bacterial strain PM1, which is capable of utilizing methyl tert-butyl ether (MTBE) as its sole carbon and energy source, was isolated from a mixed microbial consortium obtained from the University of California, Davis Department of Civil and Environmental Engineering. The original parent mixed culture was enriched from a compost-filled biofilter at the Joint Water Pollution Control Plant in Los Angeles County, CA . Analysis of the 16S rDNA sequence of strain PM1 revealed that it is most closely related to the genus Sphingomonas. Strain PM1 was capable of degrading MTBE concentrations ranging from 5 - 500 mg MTBE/ml, and the bacterium mineralized 33% of an initial concentration of 50 mg 14C-MTBE/ml within 72 hours. Observed rates of MTBE degradation were 42 mg MTBE/mg cell proteins/hour with cell growth yields of 0.1 mg cell protein/mg MTBE. Strain PM1 was unable to utilize toluene as its sole carbon and energy source, however the organism could degrade toluene when it was first induced on MTBE. Varying MTBE concentrations from 5 to 100 mg/ml had little effect on the rate of degradation of 20 mg/ml toluene. Increasing toluene concentration while holding MTBE constant resulted in inhibition of both toluene and MTBE degradation. When added to pristine soils and contaminated groundwater matrix samples with little MTBE-degrading activity, strain PM1 was able to degrade MTBE at concentrations of 25 mg MTBE/ml solution. Manipulation of oxygen tensions in groundwater matrix microcosms showed that MTBE degradation by strain PM1 requires oxygen. These results suggest that PM1 has potential for use in the remediation of sufficiently aerated MTBE contaminated environmental sites. Research Area: MTBE Research Funded By: Graduate Student Fellowship (from the TSR&TP Director's Office) Abstract Source: 1999 Symposium Presenter: Jessica Hanson perc.ucdavis.edu/abs_search/FMPro?-db=abstracts.fp5&-format=record%5fdetail.htm&-lay=web%5fmain&-sortfield=title&-op=eq&bPublish%5fIt=1&-op=cn&Research%5fArea=MTBE%20Research&-max=20&-recid=34148&-find=
|
|
|
Post by Jill on Sept 7, 2008 17:56:35 GMT -5
Per above- from the Wiki:
Sphingomonas From Wikipedia, the free encyclopedia Jump to: navigation, search Sphingomonas Scientific classification Kingdom: Bacteria Phylum: Proteobacteria Class: Alpha Proteobacteria Order: Sphingomonadales Family: Sphingomonadaceae Genus: Sphingomonas
Sphingomonas was defined in 1990 as a group of Gram-negative, rod-shaped, chemoheterotrophic, strictly aerobic bacteria. They possess ubiquinone 10 as their major respiratory quinone, contain glycosphingolipids (GSLs) instead of lipopolysaccharide in their cell envelopes, and typically produce yellow-pigmented colonies.
By 2001, the genus included more than 20 species that were quite diverse in terms of their phylogenetic, ecological, and physiological properties. As a result, the Sphingomonas were subdivided into four genera: Sphingomonas, Sphingobium, Novosphingobium and Sphingopyxis.
These genera are commonly referred to collectively as "sphingomonads" . The sphingomonads are widely distributed in nature, having been isolated from many different land and water habitats, as well as from plant root systems, clinical specimens, and other sources.
Some of the sphingomonads (especially Sphingomonas paucimobilis) also play a role in human disease, primarily by causing a range of mostly nosocomial, non-life-threatening infections that typically are easily treated by antibiotic therapy. Due to their biodegradative and biosynthetic capabilities, sphingomonads have been utilised for a wide range of biotechnological applications, from bioremediation of environmental contaminants to production of extracellular polymers such as sphingans (eg. gellan, welan, and rhamsan) used extensively in the food and other industries[citation needed]. One strain, Sphingomonas sp. 2MPII, can degrade 2-methylphenanthrene[1]
In May 2008, Daniel Burd, a 16 year old Canadian, won the Canada-Wide Science Fair in Ottawa after discovering that Sphingomonas can degrade over 40% of the weight of plastic bags (Polyethylene) in less than three months. [2]
[edit] References ^ G.M. Ni'matuzahroh, M. Gilewicz, M. Guiliano & J.C. Bertrand (May 1999). "In-vitro study of interaction between photooxidation and biodegradation of 2-methylphenanthrene by Sphingomonas sp 2MPII". Chemosphere 38 (11): 2501–2507. doi:10.1016/S0045-6535(98)00456-1. ISSN 0045-6535. PMID 10204235. ^ TheRecord.com - CanadaWorld - WCI student isolates microbe that lunches on plastic bags [edit] External links Article describing the dicovery of Sphingomonas as a biodegrader of plastic bags Kawawada, Karen, The Record (May 22 2008). This Proteobacteria-related article is a stub. You can help Wikipedia by expanding it.
Retrieved from "http://en.wikipedia.org/wiki/Sphingomonas"
|
|
|
Post by Jill on Sept 27, 2008 5:24:16 GMT -5
|
|
|
Post by Jill on Mar 4, 2009 12:38:37 GMT -5
K marie, Agree - this topic should be researched and kept in on the front burner. It is the only real and factual (and documented) explanation of what is happening not only in the US but worldwide. Case in point- Google China and MTBE- you will find the Chinese are over the top in use of the substance. Right now. www.ethanolproducer.com/article.jsp?article_id=1307 China Ramps Up Toxic MTBE Use goliath.ecnext.com/coms2/browse_R_C003 browse prodution (MTBE) in China by year- chart goes up to 2008 (articles) phoenix-cfs.org/EdPgOriginalSin.htmExcerpt: Fair use ORIGINAL SIN: Incline Village, Chronic Fatigue Syndrome and Myalgic Encephalomyelitis by Cort Johnson Incline Village is ground zero for the modern era of CFS/ME. It is where CFS/ME first (re)emerged in a big way and made it onto the national scene. What a scene it was; we had a bucolic (upper class) community in a resort town in the Sierra’s beset by a bizarre epidemic. We have the embattled good guys (our doctors) struggling against their supposed allies (big, bad government bureaucracy – the CDC) and the rest of the medical establishment all the while defending their scorned and disabled patients. What a stirring story it was. Incline Village is a place where, paradoxically, things went right in a big way (CFS/ME finally gets attention) and wrong in a big way (CFS/ME called ‘yuppie disease’.) Not so long after the Incline Outbreak the name ‘chronic fatigue syndrome’ was coined and down the slippery slope of ‘fatigue’ this disease slid. Both ME and CFS advocates claim Incline Village as their own. Some believe it was our first big chance – perhaps our only chance - to fix this disease in the research community’s mind before it got tainted by the fatigue label. The facts were all there they say! All the powers that be had to do was open their eyes. Instead they turned them away and we have suffered the consequences ever since. The CDC, of course, was on the scene early but they weren’t the only ones. A group of committed CFS researchers/physicians were there also. This group came to this disease with open eyes and for several of them it was a career changing event. The pedigree of this group is amazing; many (Cheney, Peterson, Ablashi, Komaroff, Bastien, Buchwald) will go on to take central roles in CFS research or treatment. Early on they were able to garner a series of large NIH grants and over the next six or seven years they would come to publish at least five studies examining the Incline Village (and other cohorts). We know the CDC found nothing interesting but did ‘our side’ say? These researchers - so passionately struck by their patients suffering - knew they were facing a real disease. What did they report? What kinds of patients did they find? Were they CFS patients or ME patients? Were their conclusions listened to or were they dismissed? What kind of start did CFS research get off to? Twenty-two years later we take a look at Incline Village and what it meant and didn’t mean for CFS/ME patients. The Studies: These were not small efforts. Much of the information in this paper will be taken from a major paper published in 1992 (Buchwald, Cheney, Peterson, Ablashi, Komaroff et. al. 1992). It was one of the first major studies ever done on CFS/ME and it is still one of the largest. It involved cerebrospinal fluid draws, MRI’s, HHV-6, HTLV and lymphocyte testing in at least a portion of over 200 patients. Defining CFS: One of the first problems the investigators of the 1992 study faced was one researchers are still struggling with; how to define an often debilitating disease that is marked, for the most part, by rather common symptoms. As most definitions have done, these researchers focused on the predominating symptom, fatigue. They required each patient in the study to have at least three months of chronic, debilitating fatigue with at least two of eight other symptoms (fever, headache, sore throat, earache, rhinnorhea, cough, diarrhea or muscle pain. Aside from fatigue the symptom set was somewhat unusual; fever, earache, post-nasal drip, cough and diarrhea will not appear in the 1994 definition or be prominent in others. Headache, sore throat and muscle aches will. A perhaps special kind of fatigue – post-exertional fatigue – that some researchers believe characterizes CFS was not mentioned and had not yet been delineated. Neither cognitive problems nor joint pains were part of the definition but both were common (79-88/67-82%) in this group. They noted that a ‘few patients’ (8%) developed ‘transient periods’ of an encephalitis-like symptoms including confusion, gait problems, paralysis and seizures. These early symptoms are similar to some described in early ME epidemics. Was this an ‘ME’ subset? Like ME patients earlier studies described the most severe nervous system problems (paralysis, seizure) were described as ‘transient’. As the disease progressed these patients looked more and more like the other patients and they were not segregated out again. end excerpt In my study of MTBE- I found that the cause of the condition in the Lake Tahoe/Incline Village area was found to be MTBE in Lake Tahoe. There was a researcher/doctor at Incline Village who wrote a paper on the subject- he had worked for Dr Garth Nicholson for a time. Dr Vojdani. more later Garth Nicholson has the knowledge of all the aspects of these conditions/diseases- from the mycoplasma to the MTBE, et al The only reason that a cause was found (even though is is still kept under the radar) is because Lake Tahoe residents are affluent and evidently had enough pull to get help. Again, ironically, the CDC was there. I read reports about how the 'reps' from the CDC had lost blood samples that were taken from those ill with the CFS/ME. Also, how those CDC reps basically took the time at Incline Village as a vacation rather than a serious issue. lymebusters.proboards39.com/index.cgi?board=theories&action=display&thread=10536&page=2www.aptwater.com/assets/news_articles/Article-WC&P0904.pdfExcerpt: Lurking beneath the panoramic and pristine beauty of Lake Tahoe is an environmental crisis in the form of the gasoline additive MTBE. Vir- tually unknown in the common vernacu- lar a decade ago, MTBE has become a household term. An acronym for me- thyl tert-butyl ether, MTBE has been the subject of intense environmental, legal and legislative debates regarding its im- pact on the environment, especially wa- ter, and South Lake Tahoe, California, has been a focal point of these debates. Drinking water contamination The South Tahoe Public Utility STPU provides drinking water to the city and surrounding communities from under- ground aquifers. Within a one-year pe- riod, the STPU was forced to shut down more than one-third of its drinking wa- ter wells due to MTBE contamination. The sources of the contamination were leaking underground storage tanks (LUSTs) used by gasoline dispensing sta- tions. The crisis became economic as well as environmental. ** Treatments that WORK: This is from a Doctor present at Incline Village- treating victims www.mall-net.com/cathcart/cfids.htmlExcerpt: STATEMENT ON CFIDS Robert F. Cathcart, M. D. In retrospect, I saw the first patients with CFIDS in Incline Village about 1978. The epidemic officially started sometime in 1983 in Incline Village. I left Incline Village January 1, 1980 but continued to treat patients for chronic fatigue. My treatment was mainly with massive doses of vitamin C but it also included many other nutrients. The rationale has been that CFIDS is a free radical disease involving damaged mitochondria. My suspicion that chronic fatigue was a free radical disease involving mitochondria was because of the beneficial effect of massive doses of vitamin C. I was using the massive doses of vitamin C not for the vitamin C but for the electrons carried by the vitamin C. Ordinarily, when a vitamin C molecule gives up its two extra electrons to scavenge two free radicals, the vitamin C is refueled with two more electrons from the mitochondria. When the mitochondria are damaged and cannot provide the electrons then the spent vitamin C is rapidly irreversibly lost. By giving massive doses of C, this loss is prevented, and the continuing supply of fresh vitamin C substitutes for the inability of the mitochondria to provide the electrons to refuel the spent vitamin C (dehydroascorbate.) Not incidentally, a major function of the mitochondria is to provide electrons in the form of ATP to the muscles. Without sufficient ATP to fuel the muscles, fatigue results. more on this later- ATP The mitochondria are damaged by either viruses, bacteria (sometimes cell wall deficient bacteria, L-forms), yeast toxins, sensitivity to chemicals (including some drugs), allergic reactions, etc. Probably, it usually involves two or more of the above. The damaged mitochondria become the major source of free radicals. A free radical cascade results. Fee radicals from a damaged mitochondria damage adjacent mitochondria and cause them to produce more free radicals. A domino effect results. Because all this up-regulates the immune system, various autoimmune phenomena frequently result which may include aching in muscles, trigger points, etc. (fibromyalgia). end excerpt MTBE- (Methanol) breaks down to benzene/formaldehyde/etc- deadly. Now in the soil and water supply. BENZENE- another key word.... This saga is tied to the Enron debacle- and much more.... lymebusters.proboards39.com/index.cgi?board=rash&action=display&thread=11889&page=1 As you hear in the above video- CFS leads to cancer- they knew this almost 30 years ago- they also knew the source/cause. www.youtube.com/watch?v=0Y_T5ylWUv4 Fibro/CFS- same/similar per many researchers- Not a 'new' discovery- but rather one coming to the media- spun, of course. Maybe so we don't get the connection- MTBE=CFS or Fibro (etc)and then CANCER
|
|
|
Post by Jill on Mar 4, 2009 12:47:23 GMT -5
Dr. Lapp:I had never heard of CFS or FM until 1984, when the VP of a Fortune 500 company came to me and described a debilitating illness that was triggered by the flu, and prevented him for working more than 1-2 days per week. Soon after I began researching his case, a similar case presented to my office, then another, and another. Before long it became clear that many of these cases were related, so I called the CDC to report a possible epidemic. ð@e CDC put me in touch with two other groups (Dan Peterson and Paul Cheney in Lake Tahoe, and David Bell in Lyndonville, NY) who were experiencing similar epidemics in their towns, and thus began my new career and ambition! www.immunesupport.com/Healthwatch/Healthwatch-Treatment-Guide-2002.pdfNote- the above link features treatment Protocols from 6 leading doctors- circa- 2002
|
|
|
Post by Jill on Mar 4, 2009 12:56:13 GMT -5
freespace.virgin.net/david.axford/me-refs.htm. Rosenbaum, ME., Vojdani, A., Susser M and Watson, CM. Improved immune activation markers in chronic fatigue and immune dysfunction syndrome (CFIDS) patients treated with thymic protein A. Journal of Nutritional and Environmental Medicine, 2001, 11, 241-247. Thymic protein A, an immune modulator, was tested in 23 patients with CFIDS (one year duration, abnormal CD8+ subpopulations and interferon pathway associated enzyme levels). Sixteen patients experienced normalisation of immune function with a corresponding improvement in symptoms.source of above on Google search: ***http://www.naturalhealthyconcepts.com/proboost-12pack-p-strengthen-immune-system.html REMEDY
|
|
|
Post by Jill on Mar 4, 2009 14:17:34 GMT -5
Dr Vojdani- who researched and wrote an abstract on CFS while on staff at Immed- Dr Garth Nicholson.s company (mycoplasma) comes up with a patent for a new Lyme/tick borne illness test: Ironic, eh? Think CFS and Lyme could be linked? tinyurl.com/bclnzdUnited States Patent 7,390,626 Vojdani June 24, 2008 -------------------------------------------------------------------------------- Methods and kit for diagnosing tick borne illnesses Abstract ELISA, Western Blot, and a peptide-based ELISA were applied to clinical specimens from patients with clinical symptoms of tick borne diseases, including Lyme disease. Peptides from different components of Borrelia during different cycles, including peptides from outer surface protein, leukocyte function associated antigens, immunodominant antigens, variable major proteins, and peptides from decorin-binding proteins of Borrelial subspecies (B. sensu stricto. B. afzelii, B. garinii) were used. Antibodies against specific peptides from Babesia and Ehrlichia were also measured.
|
|
|
Post by Jill on Mar 4, 2009 14:20:27 GMT -5
Patent- Dr Vojdani- one of the Doctors at Incline Village- United States Patent Patent Number: 5,707,816 Date of Patent: January 13, 1998 IMMUNOLOGICAL CROSS REACTIVITY BETWEEN CANDIDA AND HUMAN TISSUE OR FOOD ANTIGENS Inventor: Assignee: Appl. No.: Filed: Aristo Vojdani Immunosciences Lab., Inc. 796,411 February 6, 1997
ENHANCEMENT OF NATURAL KILLER CELL ACTIVITY AND T AND B CELL FUNCTION BY BUFFERED VITAMIN C IN PATIENTS EXPOSED TO TOXIC CHEMICALS: THE ROLE OF PROTEIN KINASE – C[/size] Gunnar Heuser1 and Aristo Vojdani2,3 1Neuromed & Neurotox Associates, 3235 Moorpark Rd., Thousand Oaks, CA 91361 2Immunosciences Lab, Inc., 8730 Wilshire Blvd., #305, Beverly Hills, CA 90211 3Department of Medicine, Drew University School of Medicine and Science 12021 S. Wilmington Ave., Los Angeles, CA 90059 Abstract After exposure to many toxic chemicals, NK function can be decreased significantly. Weeks or months later, natural killer (NK) function can rebound to normal levels in some and can be suppressed for prolonged periods of time in other patients. In view of this, we decided to study the effect of buffered vitamin C on NK, T and B cell function in patients who had been exposed to toxic chemicals. After the first blood draw, 55 patients immediately ingested granulated buffered vitamin C in water at a dosage of 60 mg/Kg body weight. Exactly 24 hours later, blood was again drawn for a follow-up study of NK, T and B cell function. Vitamin C in high oral dose was capable of enhancing NK activity up to ten-fold in 78% of patients. Lymphocyte blastogenic responses to T and B cell mitogens were restored to the nomal level after vitamin C usage. Signal transduction enzyme protein kinase C (PKC) appeared to be involved in the mechanism of induction of NK activity by vitamin C. We conclude that immune functional abnormalities can be restored after toxic chemical exposure by oral usage of vitamin C. *** tinyurl.com/c98arxExcerpt from patent: Immunological cross reactivity between Candida and human tissue or food antigens Autoantibodies are frequently found in the sera of virus-infected individuals, both during and after infection. For example, after infection with Epstein-Barr virus (EBV), antibodies reacting with intermediate filaments of cells, immunoglobulin or thyroglobulin were detected (Oldstone, J. Autoimmunity, 2(Suppl.):187-194, 1989; Srinivasappa et al., J. Virology, 57:397-401, 1986; Talal et al., J Clin. Invest., 85:1886-1871). Candida is a heterogeneous genus of yeast. At least six Candida species have been implicated as human pathogens, although the majority of Candida infections are caused by Candida albicans and Candida tropicalis. Candida is most often found in the mouth, feces and vagina and results in substantial morbidity and mortality in immunosuppressed patients or in those patients superinfected with the fungus. By virtue of widespread mucosal contact with this organism, antibody formation against it is common and begins early in life. Candida antibodies have been shown to belong to the IgA, IgG, IgM and IgE immunoglobulin classes (Edge et al., Clin. Allergy, 10:47-58, 1980). The predominant antibody in patients with systemic infectious candidiasis is Candida IgG (Lehner, J. Med. Microbiol, 3:475-481, 1970), whereas in patients with recurring vaginal candidiasis, the antibody response is primarily local and consists mainly of secretory IgA (Mathur et al., Infect. Immun., 15:287-294, 1977). Circulating levels of Candida antigens, antibodies and immune complexes have been used for the diagnosis of candidemia (Gutierrez et al., J. Clin. Microbiol., 1:2550-2552, 1993). Candida albicans has been reported to be a pathogenic factor in selected cases of urticaria, asthma, irritable bowel disease and psoriasis. Candida has been implicated in polyglandular autoimmune disease in which high titers of organ-specific antibodies to endocrine organs and parietal cells of the stomach were discovered (Saenger et al., J. Clin. Endo. Metabol., 54:863-868, 1982). Mathur et al. (J. Reprod. Immunol, 2:247-262, 1980) found that patients with chronic vaginal candidiasis (CVC) had one or more cross-reactive antigens on ovarian follicles, T.sub.h cells and Candida since absorption of patient sera with Candida cells, ovarian follicle cells or thymocytes reduced all three antibody titers. Despite this indirect association between CVC and autoantibodies, immunological cross reactivity between Candida antigens and human antigens was not shown. Individuals with Candida infection often develop multiple food intolerances, making it difficult for them to maintain a normal lifestyle. There is general agreement that a low carbohydrate diet is mandatory, although most patients do well on a diet avoiding refined sugars and large amounts of honey, maple syrup and fruit juices. Milk should also be restricted due to its high lactose content. Elimination of yeast-containing foods is also recommended due to the high degree of apparent cross-sensitization between Candida albicans and brewer's/baker's yeast. However, not all patients sensitive to Candida albicans will be sensitive to yeast containing foods and ingestion of a yeast-containing food does not in itself augment Candida albicans growth. Thus, it is desirable to identify whether a particular mammalian tissue or food contains antigens which cross-react with Candida antigens since antibody production against such antigens may cause development of an autoimmune disease. The present invention provides such a method. end excerpt as an aside- another patent- Dr Vojdani tinyurl.com/dkhx3d
|
|