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Post by fritolay66 on Jan 31, 2010 1:02:02 GMT -5
For those of us whom either have positive fungal petris, known or suspected environmental exposures to mold, or have Lyme in which induce biotoxin illness as well as the previously mentioned, then it is my hope you will consider the information I have posted here.
In biotoxin exposure and illness due to that exposure, a pathway in which exists would be the MSH hormone, hemolysins, cytokiene response, leptin receptors, negative feedback pathways, positive feedback pathways, and MARCoNS with biofilm formation and antibiotic resistence within the sinuses. So in whole, this post is about whole body toxins to include the sinuses and why including them is important in treatment for those with biotoxin illness.
Some sources I have read state that up to 80% of mold exposure patients and thus biotoxin exposure exhibit MARConS colonization of the deep sinuses. Colonization in which causes symptoms due to the biotoxins it produces. Colonization due to the fact that it is considered a normal occupant except for those whom are immunocompromised and so is not considered an infection causing disease, but colonization contributing to the ability of disease to take place. CoNS is Coagulase Negative Staphylococcus. MARCoNS, in my own personal opinion, is Methicillin Resistant Co-agulase Negative Staphylococus and is not normal. Any of us whom have been exposed to numerous antibiotics should consider this information carefully and especially if they are not responding to detoxification and treatments.
MARCoNS is not commonly found in the deep sinuses of normal subjects with intact immune systems. MARCoNS is found in many patients with mold exposure, Chronic Lyme, and biotoxin illnesses. It becomes Methicillin resistant due to the exposures to antibiotics. It is a very crafty organism in developing resistence.
(MSH) Melanocyte Stimulating Hormone, protects the mucous membranes in the nose from colonization of MARCoNS. Fungal exposure, Chronic Lyme, and Biotoxin illnesses deplete MSH. so with the depletion of MSH and resulting MSH deficiency, the sinuses are no longer protected from colonization of these organisms. In MSH deficient patients, I found in one source that most will display multiple resistences to more than one level of antibiotics.
They believe there are two mechanisms in which these bacteria can cause damage to MSH. One is by the production of hemolysins. These are proteins in which lyse red blood cells. The MARCoNS secrete this and cause leakage of iron in the red blood cells, of which it needs to sustain its own life. So not only does a fungal infection within the skin in most cases use iron, but the bacteria do as well. The hemolysins this bacteria creates are then sent out into the bloodstream in which the body then recognizes as foreign and stimulates a cytokiene response. Cytokienes then destroy the leptin receptors on fat cells. This is why many of us experience weight gain instead of the normal disease state of weight loss. Damaging the leptin receptors would then also contribute to the deficiency of MSH. As MSH production depends on Leptin.
The other mechanism is suspected to be Exotoxin A in which are now becoming associated with MARCoNS and are associated with Staph Aureus, and other coagulase positive staph. This toxin is capable of splitting MSH and there for totally destroying its function.
Another important aspect of MARCoNS is their ability to create biofilms. These biofilms are what contain the antibiotic resistence mechanisms of the bacteria and are co-operative in many functions. This biofilm is becoming associated with (CFS) Chronic Fatigue Syndrome. And it is here, that genotypes, the HLA's I have posted on previously are important, because the pathways are markers for the individual whom also may be predisposed to these bacterial biofilm formations. Hence multiple infections over the course of a lifetime and especially when biotoxin illness develops.
MARCoNS thrive in the absence of MSH deficiency caused by fungal exposure, Chronic Lyme, and biotoxin illnesses. MSH is incedibly important to us because it induces a very specific pathway of cytokiene response to the bacteria and kills it or prohibits it from forming in the first place. It is this in which is an example of a NORMAL regulatory pathway in which no longer can function due to the fungal exposure, Chronic Lyme, and Biotoxin illness.
In normal human functioning, this control is through the normal negative feedback pathways. But in mold exposure, Chronic Lyme, and Biotoxin illnesses, the pathway becomes a positive feedback pathway and thus the more MSH becomes deficient, the more problems that will develop. completely abnormal responses controlled by the organisms and not our normal physiologic functions.
The presence of MARCoNS and if it is not addressed, will prevent one in which has experienced mold exposure, Chronic Lyme, and Biotoxin illness from attaining a treatment response and will most certainly prevent one from attaining a cure. Is it the only roadblock, in all probability, no, but I haven't heard anyone talk about this before. Please consider this.
I know many of you out there have observed in the past about the propensity of whatever Morgellons is, that it seems to hit the face and neck really hard. I also know many of you out there also suffer ear infections, whether middle ear infections or external canal infections or are having conjunctivitis with granules or black specks. This is where the sinuses become very important in this aspect of this disease as well.
Importantly these bacteria and others including gram negative, can also be associated within the mouth and can get in between the teeth as well. All of these structures are connected. Through the teeth, often times the roots pentrate the maxillary sinuses of which then the invading or colonizing organism then has access to all of the sinuses and can disseminate from there. In the deep sinuses, its dark and comfy and incredibly hard to reach with any medication and especially true with biofilm producing organisms. The ears are connected to the sinsues by the Eustachian tube. The eyes are connected to the sinuses by the lacrimal ducts in which drain into the sinuses. And not only are fungii capable of producing granules, so are some bacterias. I am also aware that some of us have made the observation of bumps at the back of our throats, sore throats, diarhhea. All the structures and the connections I have spoken about connect to the esophagus by the Nasopharynx. Diarhhea? Yes, all that biofilm production contributes to the mucous and we swallow about 8 pints of it in a day, whether we are aware of it or not. I could make a point about intestinal cleansing and detox but I am sure you can see the connection. I am beginning to question the presence of both and hope those of you interested will also consider them as well.
How do you find out whether this organism is present?
It is called an API-staph culture.
Frito
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Post by Sidney on Jan 31, 2010 2:11:02 GMT -5
Frito, thank you so much for this. I will print it and study it again and again.
I'm particularly interested in the API Staph Culture and will add it to my growing list of planned lab work.
I'm pretty sure I had the CRP (C-Reactive Protein) test a few months ago, but these are just a few of the tests I'm going to ask about when I see my GP next visit.
Cytomegalovirus CD NK 57 or is it NK CD 57? I can never remember. Very interested in the Cytokines - Interleukins
Our "Morgellons" appears to be an immune complex disorder, but even as I write this I question so much as while some are very ill, others of us are rarely ill with colds, flu or whatever.
Anyway, all we can do is continue to plug along, hope and pray for answers.
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Post by freaky on Jan 31, 2010 3:57:30 GMT -5
Thanks so much fritolay. You are such a smart woman. I admire you. freaky
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Post by fritolay66 on Jan 31, 2010 10:27:05 GMT -5
Sidney,
You mentioned testing. I also recommend these in the context of mold exposure and disease.
HLA, Diagnosis Code: 279.10, 377.34, 279.8 MSH, Diagnosis Code: 253.2 Leptin, Diagnosis Code: 253.2 ADH, Diagnosis Code: 253.5 TNF, Diagnosis Code: 416.9 CRP, Diagnosis Code: 378.54 CBC, Diagnosis Code: 285.0 Api-Staph Culture, Diagnosis Code: 478.21 MMP-9, Diagnosis Code: 340 VEGF, Diagnosis Code: 416.9, 253.2, 710.0
There is more I found, but I don't want to violate copyright laws. These codes are from known and accepted diagnosis guidelines, and are there for covered by most insurance plans.
Frito
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Post by bannanny on Jan 31, 2010 17:48:02 GMT -5
So these are tests we should consider having, or are you saying there's something here we should be taking? Sorry, my brain hasn't come back yet.
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Post by fritolay66 on Jan 31, 2010 18:37:55 GMT -5
Yes, Bananny, these are tests and I included the footwork for physicians in their identifying codes, called diagnosis codes. Diagnosis codes are important in that there are identified disease entities associated with these codes, and so are then reimbursable or covered by insurance companies. This is very important in that if a test has a diagnosis code, it then has a reason to be performed. Most of our Non-Morgellons physicians are unfamiliar with these codes and I feel it is part of their resistance to order adequate testing. Normal blood laboratory values will not show the intricate variations associated with mold exposure and biotoxin illness. Also, you will notice a similarity of these tests to some of the tests performed by our LLMD's in the context of Lyme Disease, another biotoxin illness, and its co-infections, more biotoxin illnesses.
Do I recommend these tests? If one were to think they have an environmental mold illness and a resulting biotoxin illness, then yes.
Hugs Bananny....
Frito
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Post by bannanny on Jan 31, 2010 18:45:27 GMT -5
Thank you so much frito... I'll copy and paste and bring it to my doc (if I ever find a new one!)
huggin ya back ~~ bannanny
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Post by fritolay66 on Jan 31, 2010 22:06:45 GMT -5
Sidney,
You mentioned CD 57. Can I ask, have you taken it and have those results, or is that on your to do list?
Anybody here have any immune function tests they would like to post?
Frito
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Post by fritolay66 on Feb 2, 2010 0:56:17 GMT -5
In the case of environmental mold, skin fungii, internal fungii, and candidiasis, all of these are closely associated with their own symbiont bacteria. All of these forms of fungii have them. This is called ubiquitous bacteria. So, in the case of our environmental mold suspects, Cladosporium would have certain strains of bacteria associated with it, T. Hazarium with its own, Penicillium with its own, Chaetomium with its own, and so on even though some commonalities could exist between them. MARCoNS is commonly being found in the nasal passages in the case of environmental mold and I would suspect with these others, or a similar resistant staph. The danger of environmental mold versus whats in the air all around us, is that the environmental mold in buildings including our homes have no other competition or little competition and so they grow hidden out of sight or in deep spaces unimpeded and in that manner become more dangerous to our health than what is just commonly found in the air. The reason this unimpeded growth is so dangerous, is there is nothing to keep it in check, and so when it creates its spores, it also creates its own bacterial world, toxins, etc. It is the concentration of this that makes us sick as opposed to what is "naturally found in the air". Frito Do any of you remember this chart?
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Post by Sidney on Feb 2, 2010 1:18:36 GMT -5
Sidney, You mentioned CD 57. Can I ask, have you taken it and have those results, or is that on your to do list? Anybody here have any immune function tests they would like to post? Frito Thank you for providing test names and supplying the codes as well. (Lots of effort on your part!!!) I have not had the CD 57 test. I think when I first became ill in 1991 there were immune tests. I remember something about ANA and blah blah, but I will have to go to my files. I remember my hands were so messed up at that time that I had some sort of imaging that would show if I had Carpal Tunnel Syndrome and that turned out negative. I had miserable numbness as well as pain in both hands and both feet. Hate even thinking about the early days, but thank you for asking. I will get into the files and take a look.
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Post by fritolay66 on Feb 2, 2010 8:08:48 GMT -5
Sidney,
That hand numbness and tingling, is a classic sign of mold biotoxin poisoning. Same with the feet as well. I remember my mom complaining of it as well, and you know that smell we have all talked about at one time? She had that smell in her clothes and hair. Her home had a sewage leak under the kitchen faucet and subsequent damage underneath the house throughout the crawl space.
I had a different home at that time and also experienced a roof crack and subsequent water damage. it was at this time I started having trouble with my hormones. Anywho, getting off track here.
Everyone, please consider the ubiquitous bacteria that comes with mold exposure and biotoxin illness. The biotoxin illness cannot be addressed fully until those bacteria are gotten rid of. These are resistant bacteria due to the nature of this mold beast, and just any antibiotics won't address it. Get the Api-staph culture along with the other labs.
Some of us have had our lives so destroyed by this illness, we are the ones that have fallen through the cracks. I am one. We need solutions to the lack of health care options.
When I experienced the flu the other month ago, I started doing nasal lavage. To the saline solution, I have added 10 ppm colloidal silver and a drop of gentian violet. Less is more in this case as the exposure is cumulative.
When I found the information on MARCoNS, it really coincides with what I feel I am experiencing in my own personal case. It has been a huge benefit as my treatment seemed to hit a barrier and although I wasn't sliding backwards, I wasn't moving forwards either.
I have more codes for anyone interested.
Frito
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Post by in tokyo on Feb 5, 2010 4:09:09 GMT -5
Wow Frito, That is a super post, thank you for all of the info and your time researching. It really makes sense.
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Post by fritolay66 on Feb 5, 2010 10:37:04 GMT -5
For those of you whom left me personal messages, I felt it would better serve your personal interests, to do a general post of those additional codes you asked for.
ACTH, Diagnosis Code: 255.4 Cortisol, Diagnosis Code: 255.4 DHEAS, Diagnosis Code: MALE 257.2 FEMALE 256.39 Testosterone, Diagnosis Code: MALE 257.2 FEMALE 256.39 Androsteneione, Diagnosis Code: MALE 607.84 FEMALE 256.39 CMP, Diangosis Code: 780.79 Anticardiolipin, Diagnosis Code: 710.0 Myelin Basic Protein, Diagnosis Code: 340 Antigliadin, 579.0 IL-1B, Diagnosis Code: 716.99 TSH, Diagnosis Code: 244.8 IgA, Diagnosis Code: 579.0 TTG IgA, Diagnosis Code: 579.0 IgE, Diagnosis Code: 493.01 GGT, Diagnosis Code: 250.00 Lipid with Phenotype, Diagnosis Code: 272.0
Immune Complexes, C3d, C1q binding, Profile code #82086, Diagnosis Code: 279.8
+ Immune Complexes, C2, C4, C4d, C3a, Profile code # 82118, Diagnosis Code: 279.8
+ C3a, factor B, C3, Profile code # 82100, Diagnosis Code: 279.8
Lyme Western Blot, Igenix Lab only. Some physicians will recommend using Lab Quest, I personally do not. I recommend Igenix, as I am somewhat familar with the differences in ability and techniques in identifying bands, and Igenix is surpassed by none at this time.
As with the other tests above, there were recommendations of the specific labs in which to use with these requests. I will follow with another post on those.
For those of you whom inquired about why these tests are ordered, I will also include a glossary of those terms in the next post.
For those in which wanted the diagnosis codes associated with a definition or explanation and correlation to disease, I will include those in a later post. That will take some leg work and time to do, and I am happy to do it.
Thank you all.
Frito
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Post by fritolay66 on Feb 5, 2010 13:00:08 GMT -5
The Glossary I mentioned for the testing I posted previously. This will help to explain the intent of the testing. In alphabetical Order: ACTH: Adrenocorticotrophic Hormone, released by the Anterior Pituitary Gland. It stimulates the relase of cortisol and other hormones by the adrenal gland. Earlier in MSH deficiency, high levels of ACTH compensate for the loss of MSH regulatory control. ACTH is often dysregulated and is to be measured simultaneously with Cortisol. ADH, Antidiuretic Hormone: Made the the Posterior portion of the pituitary Gland and is secreted in the form of Vasopressin acting to retain free water from the kidneys. Meause simultaneiously with Osmolality. Androgens: Male hormones found also in females, and is released following stimulation by the Anterior Pituitary Gland. These hormones are referred to as gonadotrophins. the measure of total testosterone, DHEA-s and androstenedione will identify abnormalities associated with the MSH controls of gonadotrophins. This abnormality is found in about fourty percent of biotoxin illness patients, and I also believe may explain why some of us have noticed our hormones being whacked, while others have not. Anticardiolipin: Are are a form of anti-mitochondrial antibodies. Cardiolipin is an important component of the inner mitochondrial membrane and is found almost exclusively in the inner mitochondrial membrane where it is essential for the optimal function of numerous enzymes that are involved in mitochondrial energy metabolism. Antiphospholipid syndrome (APS or APLS) or antiphospholipid antibody syndrome is a disorder of coagulation that causes blood clots (thrombosis) in both arteries and veins as well as pregnancy-related complications. The syndrome occurs due to the autoimmune production of antibodies against phospholipid (aPL), a cell membrane substance. In particular, the disease is characterised by antibodies against cardiolipin (anti-cardiolipin antibodies) and glycoprotein I. They are finding correlations of this and biotoxin mediated illnesses and those of mold exposures. en.wikipedia.org/wiki/Anti-cardiolipin_antibodiesen.wikipedia.org/wiki/Antibodies,_anticardiolipin en.wikipedia.org/wiki/Antiphospholipid_syndromeC3a: A by-product of the activation of the third component of the complement immune system. C3a can continue to activate C3 when the alternative pathway of complement is activated. This creates a high risk for Chronic Fatque and persistent symptoms in biotoxin patients. Cortisol: Is a powerful anti-inflammatory steroid hormone released by the adrenal glands upon stimulation of ACTH. Must measure or test simultaneously with ACTH to assess dysregulation. CRP, C-Reactive Protein: A marker of inflammation initiated by the release of IL-6 and is an innate immune response. DHEA-S, Dehydroepiandrosterone sulfate: Most abundant steroid hormone in the human body and is catalyzed in the liver, small intestine and the adrenals. Is a androgen Hormone and considered a neurosteriod as well. en.wikipedia.org/wiki/DehydroepiandrosteroneGGTP: A liver enzyme that converts glutathione. Relationship to Bile ducts!!! Gliadin: Is a protein found in gluten and is considered the toxic element in gluten. Some evidence suggests that gliadin can be incorporated into the intestinal wall acting as a “self” molecule. This has basis in autoimmunity diseases and can result from mold exposure or biotoxin illnesses. HLA-DR: These are groups of immune response genes that control the attachment of HLA to antigens in dendrite cells. The dendrite cells are antigen presenting cells that engulfs the antigen before processing it and presenting the antigen for antibody formation. This is tied into the inate immune response and the acquired immune response. Interestingly enough, you will find upon more research, that dendrite cells are also in our skin. IgA, Immunoglobulin A: is an antibody which plays a critical role in mucosal immunity. More IgA is produced in mucosal linings than all other types of antibody combined. In its secretory form, IgA is the main immunoglobulin found in mucous secretions, including tears, saliva, colostrum and secretions from the genito-urinary tract, gastrointestinal tract, prostate and respiratory epithelium. It is also found in small amounts in blood. en.wikipedia.org/wiki/IgATTG IgA, Tissue transglutaminase Immunoglobulin A: It is particularly notable for being the autoantigen and is also known to play a role in apoptosis, cellulardifferentiation, and matrix stabilisation. The antibodies to tissue transglutaminase follow a complex pathway of generation. For most antigens, T-cells specific to those antigens develop, for autoimmunity autoreactive T-cells are not suppressed or antigens escape the protective process. T-cells are stimulated by antigen, presented by MHC molecules (HLA in humans) and surface IgM on antigen reactive B-cells. These T-helper cells then stimulate B-cells to multiply and mature into plasma cells that make IgG to that protein. en.wikipedia.org/wiki/Tissue_transglutaminaseen.wikipedia.org/wiki/Anti-transglutaminase_antibodiesIgE, Immunoglobulin E: Capable of elicting one of the mostpowerful immune responses within the human body as well as its relation to allergy. This is part of the adaptive immune response. IL-1B, Interluekin-1-beta: Is one of the cytokienes that causes increased inflammation and is commonly found in biotoxin illness patients. IL-6, Interluekin 6: Is a pro-inflammatory cytokiene released as part of the recognition of foreign antigens and after activated by TNF and IL-1B. Leptin: This is a hormone made by fat cells. It is also a cytokiene that participates in regulating body mass and storing fatty acids efficiently. It also stimulates the production of MSH, which in turn regulates much of the immune response from the innate immune system. High Leptin and low MSH are the markers for obesity caused by toxins!!! With high Leptin in a biotoxin patient, weight loss in incredibly difficult. It is also associated with weight gain in short periods of time, and is a direct result of the bodys response to biotoxin illness. MMP-9, Matrix Metalloproteinases, including #9, and are enzymes that effect tissues underneath blood vessel walls, the sub-intimal space. It is particularly adept at delivering inflammatory elements out of the blood vessels and putting them where they shouldn’t be such as the brain, lung, muscle, and joints. If you see a high MMP-9, look for toxins and cytokienes. MSH, Melanocyte Stimulating Hormone: Is a small peptide hormone that regulates all aspects of the innate immune-response. Made in the hypothalamus, it controls nerve, hormone, cytokiene function, skin cells, and mucous membrane defenses, as well as controlling production of endorphins and melatonin!!!! Lil’sissy, this is where I believe Karen’s mucous membrane production of mucous lies. I have not forgot about you two, and I am still working hard to find what is going on with us and cancer occurrence. MBP, Myelin Basic Protein: Antibody to myelin basic protein. MBP is related to the myelin coating on nerves and commonly in biotoxin patients, the development of this antibody, then causes a autoimmunity to the coating on the nerve cells, the myelin, and attacks the proteins because they are viewed by the immune system as forgeing, when in fact they are not. en.wikipedia.org/wiki/Myelin_basic_proteinNFkB: Is a nuclear factor that rapidly activates the cytokiene nuclear receptor and is normally sheltered in a protecting protein called IkA. NFkB is released from IkA by a second messenger sent from a cell surface membrane after a toxin binds to the surface of a Toll Receptor. A Toll Receptor is a part of the innate immune response that provides rapid response after they are activated y bacterial, viral, and toxin components circulating in the blood. TNF, Tumor Necrosis Factor: One of the cytokienes that also increases inflammation. It is rarely elevated in patients of mold toxin illness but is associated with Post or Chronic Lyme Disease!!! There are many interconnections that are based on the HLA genetic types. Consequently, Chronic Lyme is also typed in the HLA types, the same as mold sensitivity and gluten allergies!!! They are finding correlations of certain HLA types and the likelihood of the occurrence or predispostion in a patient to develop refractory or Chronic Lyme Disease, mold sensitivities, and gluten antibody formation!!!!TSH, Thyroid Stimulating Hormone or also known as thyrotropin: Is a peptide hormone secreted by the Anterior Pituitary Gland which regulates the endocrine function of the thyroid gland. In the case of biotoxin illness, these levels are found to be low. in my own opinion, many find upon testing that levels may be considerd normal when in fact T3 is low. My opinion is based on what I have found many Lyme patients experiencing and are having their T3 compounded by a local pharmacy and has been quite helpful in the fatigue area of their disease. VEGF, Vascular Endothelial Growth Factor: One of many suspected growth factors. And exerts extraordinary control of blood flow in capillaries. High levels of VEGF stimulate the making of new blood vasculature called angiogenesis. The role of VEGF in cancer and heart disease is being researched currently. Low VEGF is common in biotoxin patients and affects the NFkB and cytokienes. VEGF Resistence: High levels of VEGF in the face of low VO2 max and low anerobic threshold. In this case, the VEGF receptor is not responding to VEGF despite high levels found in the blood. One must distinguish this condition from high VEGF without resistence by performing a pulmonary function test. High levels of VEGF are associated with the Lyme co-infection of Bartonella. Reference to Dr. Schaller publications. Reference to Dr. Ritchie Shoemaker publications. Sources to be added as time permits. I will get them here for those of you whom would like to follow up on this information.
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Post by fritolay66 on Feb 5, 2010 14:01:45 GMT -5
Complex Terminology relevant to these tests and again, in alphabetical order:
Acquired Immune Response: Is the antibody formation by special B lymphocytes after an HLA-tagged antigen is presented to the B lymphocytes by Dendrite T cells.
Alternative Pathway of Complement Immune System: Is a series of proteins activated by antigen’s only, without antibodies, particularly found to be important in illnesses caused by biotoxins in the HLA-susceptible genotypes.
Antibody: Are compounds made by the acquired immune response designed to eliminate a specific molecule, recognized at “not self” by lymphocytes.
Antigen: Is a specific molecule that stimulates an immune response.
Autoantibodies: produced by the acquired immune response to our own tissues.
Classical Pathway of Complement Immune System: Is a series of proteins activated by a combination of antigen and antibody joined to make an “immune complex”. Classical pathways activation is commonly seen in diseases of autoimmunity, but is also highly important in eliminating infectious organisms.
Complement Immune System: It is the innate immune first responder composed of thirty differing proteins, receptors, andinhibitorss. I don’t know what those thirty are as of yet. It is also the third most common protein found in the blood. Highly associated with immune responses in acute exposures where as chronic elevation of the alternative pathway increases C3a, a major factor found in chronic illness symptomologies.
GeneticSusceptibilityy: Predetermined group of immune-response genes represented by HLA-DR. Particular toxin illnesses are highly linked to presence of particular genotypes. While the genes do not make the patient ill, exposure to toxins will result in illness. essentially in those with thesusceptiblee genotypes. TheHLAA link to lack of antibody formation is a bridge from the innate immune response and the acquired immune response immune system.
Genotype: Is a person’s genetic makeup or their DNA sequences. Innate Immune Response: Is considered first responders originating from germ cell lineages and includes dendrite cells, macrophages, toll receptors, complement, and cytokienes.
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Post by fritolay66 on Feb 8, 2010 15:27:50 GMT -5
Well, after I had done all this work to post the above info, I came across this on the internet during my Lyme searches. This is the actual order sheet for a mold or biotoxin workup. Resource by Dr. Ritchie Shoemaker www.moldwarriors.com/PDFs/NTordersheet.pdfwww.moldwarriors.com/PDFs/NTinchartdrawsheet.pdfDid anyone happen to actually read the glossary? I believe that glossary I worked so hard on explains many of the things we have been identifying on our own here at Lymebusters. How the mitochondria are involved, the hormones, the cytokienes, the immune responses, Toll receptors, immune and detox pathways, and identified abnormalities in labs posted by the MRF. Frito
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Post by Sidney on Feb 8, 2010 16:13:09 GMT -5
Sidney, You mentioned CD 57. Can I ask, have you taken it and have those results, or is that on your to do list? Anybody here have any immune function tests they would like to post? Frito No, Frito, have not had this test, but it is definitely on the list. Thanks!
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Post by fritolay66 on Feb 8, 2010 16:26:35 GMT -5
I would like to recover the connection between mold in the home and bacteria. These two occur together. Also, some of these environmental molds are documented to cause infectious disease. And I suspect more of them would also be connected to infectious disease, if the exposure was high enough or long enough.
First though let me explain what I have learned about indoor mold growth and the difference between it and outdoor or the ubiquitous mold. In the outdoors, there are literally gazillions of mold spores, but these gazillions are of many differing species. Not only do mold spores abound in the outside environment, but also bacteria, and others. Each of these functions as a control for the other. So no one bacteria or mold is dominant in the air. What is found outside of building structures is basically fighting for survival, and this is survivial of the fittest, those that can reproduce in numbers, or how fast, etc.
Now, for the indoors molds. These do not have the competition that exists in the outside environment. Commonly, there are about 7-12 of these molds found on the indoors. With the lack of competition, these molds grow rapidly, and instead of just fighting to survive, like in the outdoors, they don't have to. So in this sense their growth is amplified. Amplified mold growth is what is the difference between the outdoors and the indoors. This amplified growth, since there is no competition, they can then spend their energy making spores and toxins. Consequently, about 1000 molecules of toxins can fit in the size of a pin head. There growth is not limited by competition. Not only do mold do this in this type of scenario, but so do bacteria.
One of those bacteria is actinobacteria!!! Others include gram positive bacteria including Staphylococcus, Streptococcus, Enterococcus, Bacillus, Corynebacterium, Nocardia, Clostridium, Mycobacteria, and Listeria. Does any of this sound familar to you? These are all things we have come up with here!!!
So infectious disease is documented in the moldy environment, but it is not limited to systemic mycoses, but should be expanded to include bacterial infections and resistent ones at that. Because, just like no competition of the indoors environment and the mold growth, the same occurs for bacterial growth. With all the household cleaners and such with anitbacterial agents in them, and in an environment in which is laden with amplified growth, you will be breeding the resistent forms.
Frito
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Post by fritolay66 on Feb 8, 2010 16:41:00 GMT -5
I would like to go back to the sinus issues. One of the very first symptoms besides weight gain, diarhhea, and extreme fatigue, were my sinuses and the base of my ears itched like no man yana. It was shortley after this that my symptoms exploded into some thrity to fourty symptoms of which no one could identify. My God, I should of gotten out then. This was also about the time I started manifesting hundreds of lesions.
Lets back up a minute. Going to the derm, they diagnosed me with Tinea versicolor and they thought I had resistent bacteria in my nose. Since the bulk of my first lesions were located under my nose and around my mouth. They did not however do a culture. God, I hate derms.
When they do an Api Staph culture, this culture is not one in which they sample your nostrils. They do a culture of your deep sinuses, and to give you an idea of just what deep means, that means about three inches into your nasal cavity!!!! Thats a lot of real estate inside ones head. This also correlates with my suspicions of the nasopharyngeal connection to infections in myself and then hence my posting to you all for consideration.
And thinking back, this all correlates with the water intrusions I had in my home at the time.
Frito
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Post by Sidney on Feb 8, 2010 21:48:07 GMT -5
Frito, I hope you don't consider all your hours of information gathering, assimilating and sharing, wasted effort. While much of this is way over my head I do recognize that there's much to be learned from your work.
I also recognized much that was very familiar, including these: "One of those bacteria is actinobacteria!!! Others include gram positive bacteria including Staphylococcus, Streptococcus, Enterococcus, Bacillus, Corynebacterium, Nocardia, Clostridium, Mycobacteria, and Listeria. Does any of this sound familar to you? These are all things we have come up with here!!!"
I wonder if any LB posters have been tested for Nocardiosis. It's another one to add to my list, although cutaneous lesions associated with Nocardiosis are extremely disfiguring.
Anyway, thank you so much. I copied, pasted and emailed much of this to myself for printing.
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