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Post by kammy on Mar 19, 2010 17:32:40 GMT -5
This thread is to discuss the relationship of the various species that are becoming ill with known and unknown diseases and their relationship to Morgellons Disease. Is Morgellons appearing to be a zoonotic disease? Zoonosis en.wikipedia.org/wiki/Zoonosis"A zoonosis (pronounced /ˌzoʊ.əˈnoʊsɨs/) or zoonose[1] is any infectious disease that can be transmitted (in some instances, by a vector) from non-human animals, both wild and domestic, to humans or from humans to non-human animals (the latter is sometimes called reverse zoonosis). Many serious diseases fall under this category. The simplest definition of a zoonosis is a disease that can be transmitted from other vertebrate animals to humans. A slightly more technical definition is a disease that normally infects other animals, but can also infect humans. The reverse situation (transmission from human to animal) is known as anthroponosis."
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Post by kammy on Mar 19, 2010 17:38:57 GMT -5
Frog / Koala Disease and Morgellons The first years in my home in Atlanta, I had so many frogs around it in the summertime, it was so loud and eerie sounding, like a bad Alfred Hitchcock frog movie. Then the past 2 summers not a single frog, it was very quiet - I have to say, this is even more eerie! There had always been frogs around my house - where did all the frogs go? As a matter of a fact, I don't recall there being as many cricket or "caddiedids" sounds, either. jcm.asm.org/cgi/reprint/37/7/2378.pdf"Chlamydia pneumoniae in a Free-Ranging Giant Barred Frog (Mixophyes iteratus) from Australia The koala biovar of Chlamydia pneumoniae was identified in lung tissue from a sick, free-ranging giant barred frog (Mixophyes iteratus) by using electron microscopy, C. pneumoniae-specific fluorescent-antibody staining, cell culture, and sequencing of the ompA, ompB and 16S rRNA genes. This is the first report of a chlamydial strain infecting both a homeotherm and a poikilotherm and only the fourth host (in addition to humans, koalas, and horses) to be naturally infected with this species of Chlamydia. The frog had severe, chronic, mononuclear pneumonia and nonregenerative anemia and pancytopenia. Chlamydia infections have been reported previously in captive amphibians, causing moderate to high mortality rates in various species... There have also been a few case reports of Chlamydia infections in captive and wild reptiles. In all cases, however, the chlamydial species was either unknown or assumed to be C. psittaci. Here we report the first isolation of Chlamydia from a frog in Australia and demonstrate that it is identical to the C. pneumoniae strain that infects koalas." I cannot link the photos in the .pdf file but look at the photograph in Figure 4 taken at 700nm, what they are calling example "I", "condensed elementary bodies" is very similar to a photograph that I took that I called "Mr. Brown Eye" cultured from my ear, 28 days growth, at 100x: [/img][/center] "The chlamydial particles included dense elementary bodies (314 6 39 nm in diameter [mean 6 standard deviation; n 5 12]), intermediate bodies (371 6 44 nm [n 5 12]), and numerous dividing reticulate bodies (537 6 130 nm [n 5 12]) (Fig. 4). The round elementary bodies had eccentric nuclei and a narrow or nonexistent periplasmic space. Mitochondria were not associated with the inclusion membrane. These ultrastructural characteristics are consistent with those of C. pneumoniae (14). The human strains of C. pneumoniae have also recently been shown to be able to infect and grow in both peripheral blood and alveolar macrophages (15) as well as vascular endothelium and arterial smooth muscle cells (1). At this stage, however, we are unsure of the source of infection, although the Orara East State forest where the frog was found contains a significant population of koalas and reports indicate that C. pneumoniae infection is common in most Australian koala populations. Several studies have reported the very high genetic similarity of human C. pneumoniae strains (5) and also the clonality of koala C. pneumoniae strains (24), suggesting a possible recent divergence of these biovars. It is possible that the infection of a wild frog described in this report was an isolated incident, or alternatively, increased testing may show that amphibians are commonly infected with C. pneumoniae and that they are a natural reservoir for this species." We need to look at the various animals that are becoming sick in nature at this time to see if there is a correlation between their disease and ours, one inclusive of the bats, bees, frogs, and we need to especially look at what has happened to the koala and frogs with this Chlamydia aspect. It looks highly probable that those of us with evident Morgellons have a disease similar to what is eliminating the frog and koala populations, this needs looked into closer. I can easily see the correlation between the fungus gnat/baculorviral vector with the koala, frog, horse, reptile, cat, dog and man. Once again, we have GIANT cells involved in our disease, look at "Mr. Brown Eye" in the photo below at 450x, he's bulging out of the frame! [/img][/center] The cell infecting the koala and frog are also GIANT C. pneumonia cells called "large inclusions". " Koala Biovar of Chlamydia pneumoniae Infects Human and Koala Monocytes and Induces Increased Uptake of Lipids In Vitro iai.asm.org/cgi/content/abstract/69/12/7894We examined the ability of the koala biovar of Chlamydia pneumoniae to infect both Hep-2 cells and human monocytes and the effect of infection on the formation of foam cells. The koala biovar produced large inclusions in both human and koala monocytes and in Hep-2 cells." Reference: www.sciencedaily.com/releases/2008/08/080812135654.htm"Extreme cases of this are called mass extinction events, and there have been only five in our planet's history, until now."
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Post by kammy on Mar 19, 2010 17:47:17 GMT -5
www.chlamydiae.com/restricted/docs/infections/vet_koalainfections.asp"Chlamydial infections in animals - Infections in koalas C. pecorum Free-living populations of a marsupial, the koala, are seriously affected by chlamydial infections that cause blindness and infertility. Conjunctivitis and chemosis had been noted for many decades in koala (Studdert, 1976). "Wet tail" or "dirty bottom" is also a characteristic sign [see external link: koala threats]. Major mortality among koala populations has also been observed as far back as 1887. These were apparently caused by severe eye infections which lead to impaired vision, reduced food intake and death (Cockram and Jackson, 1981). Respiratory tract infections have also been observed in koala populations. The chlamydial strains infecting koala proved to be genetically diverse (Timms et al., 1996), explaining some of the clinical and immunological diversity observed. Diversity was particularly found in the C. pecorum strains from koala conjunctivitis, urinary tract infection and infertility. This suggests that koala may become infected from various animal sources, such as cattle and sheep, possibly pigs, but not birds. C. pecorum may thus cross host barriers easily, perhaps transmitted though faecal contamination of pastures and leaves. Indeed, C. pecorum infection may have been introduced in the last 200 years when sheep and cattle were imported into Australia. Reproductive tract infection is probably sexually transmitted. Tetracycline is effective in eradicating the infection and has been used to save koala populations that otherwise were threatened with extinction. C. pneumoniae It is now clear that koala are also infected with C. pneumoniae. In free-range koala populations C. pneumoniae infection is rarely associated with respiratory, ocular or urogenital tract disease. However, in one captive koala population C. pneumoniae infection caused a severe and extended respiratory episode. Wardrop et al. (1999) demonstrated that koalas could be infected with C. pecorum and C. pneumoniae, both of which would have been formerly designated Chlamydia psittaci. In humans, C. pneumoniae has been linked to coronary artery disease and is thought to disseminate to atherosclerotic plaque from an initial respiratory focus of infection via peripheral blood monocytes [see: C. pneumoniae and CAD]."
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Post by kammy on Mar 19, 2010 18:14:31 GMT -5
Feb 2000 CDC Report: www.cdc.gov/ncidod/EID/vol6no2/reed.htm"Chlamydia pneumoniae Infection in a Breeding Colony of African Clawed Frogs (Xenopus tropicalis) More than 90% of a breeding colony of clawed frogs (Xenopus tropicalis) imported to the United States from western Africa died in an epizootic of chlamydiosis. Chlamydial inclusions were observed by light and electron microscopy in the liver of an infected frog. Chlamydia pneumoniae was isolated in cell cultures from four frogs. A cutaneous infection by a chytridiomycete fungus observed in two frogs could have been a cofactor in the die-off. Chlamydia infections cause disease in humans, birds, and mammals. Of the four currently recognized Chlamydia species, C. psittaci is the most important animal pathogen. Psittacosis, which can manifest as severe enteric and respiratory illness in many avian species, is highly contagious and can be transmitted to humans and many other mammals (1)." **I was under the impression that the chytridiomycete fungus was killing the frogs, not chlamydiosis.
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Post by jeany on Mar 19, 2010 18:20:20 GMT -5
Here's an interesting article according CPN and how it's linked to atherosclerosis. Chlamydia pneumonae infected cells turn in to 'foam cells' by 'feeding off' on Cholesterol and lipids. The foam cells are also called lipid macrophage cells. www.graduateschool.uwm.edu/research/spectrum/snapshots/anthony-azenabor/When Chlamydia pneumoniae—a respiratory pathogen that causes pneumonia and bronchitis—encounters macrophages, it assumes a chronic form, changing itself into a stubborn variant that infects the infection-fighters. C. pneumoniae is of particular concern because of its connection to atherosclerosis, a clogging, narrowing, and hardening of the body's arteries that can lead to stroke or heart attack, as well as eye and kidney problems. The pathogen releases signals that render macrophages' toxic-chemical-producing enzymes ineffective as "killing molecules." It hijacks their intracellular cholesterol, which macrophages need to stretch and move effectively. And C. pneumoniae causes macrophages to take on extracellular cholesterol and lipids that they can't use. As these Chlamydia-infected cells take on more and more cholesterol, they become immobile foam cells. They multiply and accumulate in blood vessel walls, forming lesions that are the precursors of atherosclerosis. C. pneumoniae attaches to macrophages, it initiates a pattern of calcium ion movement into macrophages that disturbs the production of toxic radicals that would normally kill microbes. But recently, Azenabor and his team discovered a drug that kills C. pneumoniae outright. He can't discuss details of this brand-new technology, disclosing only that he applied for a patent in March, and has submitted his findings for publication to the journal Medical Microbiology and Immunology. *this article is from 2006. Perhaps we should look if we can find his 'CPN killer drug'? Jeany
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Post by lilsissy on Mar 19, 2010 18:43:35 GMT -5
I need to take time to read this but yes morgellons is zoonotic ,I believe that the C.D.C. releases re: Morgellons leave one to conclude that, at least they ( C.D.C.) seem to think so.
I was shocked to read that insects are contaminated with Chlamydia , the monarch butterfly was almost wiped out from this if I remember right. The 1 q 42 gene sequence has something to do with the Chlamydia gene, seems we have a natural partial code for Chlamydia , at least that's what the article I read seemed to be saying.
Over -all Chlamydia is necessary for light encoded control of the brain, Stanford University has a great video on this.
They made a viral chlamydia gene vector system for future technology some time ago.
Yes , zoonoctic.... yes, chlamydia
but more think X.M.R.V. has probably the largest genome of all = many pieces of gene sequences to add to our natural. X.M.R.V. also causes amyloid fibrils = prions. X.M.R.V. with amyloid fibrils were found in connection with 1q42 gene related prostate cancer.
These partial gene sequences do not hurt you unless you get the entire code for an organism, then your own body can make them, at least that is how I understand it.
So they add snippets of foreign D.N.A. to us by something like X.M.R.V. to pair up with our partial snippets and wellla...... now your D.N.A. contains full instructions for the assembly of lower life organisms .
Example, You have a partial D.N.A. code for a fruit fly from birth then then get the rest of the code by gene transfer, a virus... , now you have the entire code of a fruit fly in your own genome , this is when it causes illness.
I would like to have more knowledge of this , does this mean our own body can display the embryonic development of fruitflys under the right circumstances . A D.N.A. code is only a D.N.A. code to create an organism when there exists the right circumstances such as ... when a egg and sperm meet. Can a self genesis of lower life forms occur in the right circumstances within the human tissue? I think so.
Often I see samples posted of small specimens that seem to be forming in small embryos here on the board. Cancer itself is part of natural embryonic processes gone a stray.
This would explain why we see intracellular birth of foreign insects and the like.
Jen
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Post by kammy on Mar 19, 2010 20:16:41 GMT -5
Thanks J & J - Jen and Jeany - this puzzle should start coming together very easily now that we're finding some of the pieces. I wasn't aware that it was Chlamydia that was affecting the butterflies?... let's add them to the list... here's Mr. Salamander, too... txtwriter.com/onscience/Articles/deadfrogs.html"After Bradford reported this puzzling disappearance to other biologists, an alarming pattern soon became evident. Throughout the world, local population of amphibians (frogs, toads, and salamanders) were becoming extinct. Amphibians have been around for 350 million years, since long before the dinosaurs. Their sudden disappearance from so many of their natural homes sounded an alarm among biologists. What are we doing to our world? If amphibians cannot survive the world we are making, can we? (3) chemical pollutants that are toxic to amphibians, and (4) fatal infections by parasites. The infectious agent proved to be a virus common in fish called a ranavirus. Usually a harmless soil fungus that decomposes plant material, this particular chytrid (with the Latin name of Batrachochytrium dendrobatidis) is far from harmless to amphibians. This killer chytrid was introduced to Australia near Melbourne in the early 1980s. Now almost all of Australia is affected. How did the disease spread so rapidly? Apparently it travelled by truck. Infected frogs move all across Australia in wooden boxes with bunches of bananas."
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Post by kammy on Mar 19, 2010 21:07:37 GMT -5
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vasue
Junior Member
Posts: 63
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Post by vasue on Mar 19, 2010 21:38:26 GMT -5
Lilsissy, find your question & answer very interesting: "Can a self genesis of lower life forms occur in the right circumstances within the human tissue? I think so." Doctors Kolb & Staninger mention this within a radio interview from March 8, 2008, recorded here: tinyurl.com/yfu8ehmBegin listening at 31 minutes into the discussion. The entire interview is well worth the time.
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Post by kammy on Mar 19, 2010 22:25:34 GMT -5
Thanks Vasue!,www.sosanfibios.org/fungus.htm"The chytrid fungus Batrachochytrium dendrobatidis is a recently described species that has been responsible for the total extinction of populations and species of amphibians worldwide. The group of chytridiomycete fungi is well known through genera that parasitize invertebrates and plants. They are found all over the world, in all environments including areas well conserved and very sensitive to contamination. The recently described amphibian chytrid species is unique in being the first case of a chytrid fungus that affects a vertebrate. The amphibian populations affected by this species of fungus (known as the "assassinator fungus") generally disappear in a few consecutive months, following a pattern of dispersion that is typical of infectious." Amphibians Come Under Study www.flatrock.org.nz/topics/animals/frogs.htm""This is now a front-burner issue," says James Hanken, herpetology curator at Harvard University's Museum of Comparative Zoology. "This is not just a plan; things are happening." Hanken was among a group of scientists who met early last month in West Virginia to design ARMI. Officials from various federal agencies, including the National Science Foundation, the National Institutes of Health and the Environmental Protection Agency, also took part." "The national Amphibian Research and Monitoring Initiative (ARMI) has been set up by the US Department of the Interior with $5 million to study why frogs, salamanders and toads are in such trouble. An additional $2 million is being sought for next year's budget." **WHAT? They are spending $5 - $7,000,000 annually on the frogs and only $300,000 total on a one-time human study?
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Post by kammy on Mar 19, 2010 23:10:58 GMT -5
It's time for a photo of another fun frog... anybody see one you can post in here? Or, a salamander, toad, bee, bat, butterfly, or koala bear... any of these will do? We're creating Noah's Ark here before they are all gone... ;D A photo of the Ark would do, too...
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Post by lilsissy on Mar 19, 2010 23:14:38 GMT -5
Chlamydia was only one gene that they needed to add watch this and you will know what i mean, ChR2 is the other . very short www.youtube.com/watch?v=v7uRFVR9BPUB.T.W. our hallow hair is the fiber optic, this is delivered by a virus much work has been done on this.
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Post by kammy on Mar 19, 2010 23:20:00 GMT -5
TY, Jen! It's getting late for me here, I'll come back and watch it tomorrow. I've been looking at the microscopic photos of the Batrachochytrium dendrobatidis fungus, it looks like it's in our mix too... I'll look closer at this tomorrow... Here's a map of where Batrachochytrium dendrobatidis is in the world, how does this map relate to the Morg Map? [/img][/center] www.spatialepidemiology.net/bd/
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Post by kammy on Mar 20, 2010 6:55:29 GMT -5
Morgellons Is Partially the Frog Fungus Batrachochytrium dendrobatidis or Chytrid I'm looking for Stock Photos of Batrachochytrium dendrobatidis on the Web to see if I can match them with my microscopic photos of cultured human Morgellons samples. www.esf.edu/efb/brunner/research.htmDevelopment of Batrachochytrium dendrobatidis zoospores. The name of the photograph above is "ChytridDevelopment.png", this suggests that this is how the Chytrid species develops overall? Below is a comparison from my ear lesion, cultured in nutrient agar at 30 days growth at 100x: I'm noticing that there aren't too many good microscopic photos, but this one above is a good example to give us an indication that Batrachochytrium dendrobatidis or a Chytrid fungus species is most likely involved as one of the mystery Morgellons fungi.Literature says, "Chytrids will not infect human skin since they do not multiply above 31° C." something is definitely happening in my ear lesion photo above and it's above 31 degrees C (87.8 F)! A hybrid?
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Post by kammy on Mar 20, 2010 8:52:57 GMT -5
This is a very good blog article to give us an indication of what the issues behind zoonotic diseases are: whyfiles.org/180zoonotic_disease/I found this statement at the bottom of the page most interesting: "(Zoonoses are distinct from vector-borne diseases, like malaria, that are spread from person to person by arthropods like ticks or mosquitoes.)" I'm saying that Morgellons is zoonotic and has an gnat/nematode vector - what does this mean, that Morgellons is the first zoonotic/vector disease?
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Post by bannanny on Mar 20, 2010 15:32:53 GMT -5
I know I gave morgs to my dogs... so it's definitely a human to animal disease. I got it first, then my environment and my dogs became contaminated. That's why I won't be around another animal until this mess is over with... which has really fu*ked up my life. Big empty hole in my heart ya know?
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Post by kammy on Mar 20, 2010 16:35:13 GMT -5
Yes, Banny, I understand... it's sad the predicament we've been put in. Here's an article discussing whether or not global warming is responsible for the frog fungus to get out of control. In Jan 2006, LiveScience reported that it was... it is now being disputed in this article: news.mongabay.com/2008/0325-frogs.html"Writing in the March 25 issue of PLoS Biology, a team of researchers led by Karen Lips of Southern Illinois University-Carbondale report finding "no evidence to support the hypothesis that climate change has been driving outbreaks of amphibian chytridiomycosis" — a disease blamed for large-scale die-offs of amphibians. Other researchers have argued that climate shifts are worsening the outbreak of the fungal disease. Lips says that since the fungus can be spread via the ornamental plant and aquarium wildlife trade, governments and environmental agencies should test on imports for the presence of the pathogen. Once discovered, anti-fungal drugs can destroy the fungus before it can contaminate an area."
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Post by kammy on Mar 20, 2010 16:52:24 GMT -5
"World’s Frogs Dying From Heart Failure" insciences.org/article.php?article_id=729523 October 2009 "“Our team of pathophysiologists, veterinary pathologists and disease ecologists found that the skin’s functions are severely impaired by the fungus. We think this leads to loss of electrolytes and cardiac arrest in frogs infected with the pathogen,” she said." “Resolving the cause of death in infected frogs is an important step in understanding how this disease is causing catastrophic amphibian declines around the world.”
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Post by kammy on Mar 20, 2010 17:14:41 GMT -5
news.mongabay.com/2009/0813-pesticides.htmlPesticide use linked to dying frogs in California August 13, 2009 news.mongabay.com/2009/0813-pesticides.html"In January 2007 a study led by Frank Wania of the University of Toronto found that pesticides used in lowland areas in Central America are carried by air currents to higher elevations where they are they precipitated out as rain when the air cools. The chemicals -- especially the insecticide endosulfan and fungicide chlorothalonil -- then accumulate in the montane ecosystems, which have experienced particularly severe declines in amphibian populations over the past thirty years. Meanwhile other research has linked Atrazine, one of the most widely used pesticides in the United States, to dying salamanders."
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Post by kammy on Mar 20, 2010 17:20:07 GMT -5
Gatorade Could Be The Cure For Dying Frogs? www.huffingtonpost.com/2009/10/25/gatorade-could-be-the-cur_n_332173.html"New Scientist: The fungus now decimating frog populations around the world does its damage by impairing the animals' ability to absorb electrolytes through their skin. This discovery may eventually lead to treatments that make the disease less lethal."
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