LONDON IS LATE FOR WORK
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Post by LONDON IS LATE FOR WORK on Mar 1, 2006 8:06:35 GMT -5
Thank you! Just about a month ago my left forearm began breaking out in these little patches of blisters.....I think it is DH, which is:
Background: Dermatitis herpetiformis (DH) is an autoimmune blistering disorder associated with a gluten-sensitive enteropathy (GSE). It is characterized by grouped excoriations; erythematous, urticarial plaques; and papules with vesicles. These are located on the extensor surfaces of the elbows, knees, buttocks, and back. It is exquisitely pruritic, and the vesicles are often excoriated to erosions by the time of physical examination. Diagnosis requires direct immunofluorescence of a skin biopsy specimen showing deposition of immunoglobulin A (IgA) in a granular pattern in the upper papillary dermis. Although most patients are asymptomatic, more than 90% have an associated GSE upon endoscopic examination. DH is treated with dapsone and with maintenance of a gluten-free diet.
Pathophysiology: DH is a disease of the skin. It is caused by deposition of IgA in the papillary dermis. This triggers an immune cascade, including recruitment of neutrophils and complement into the area.
An underlying genetic predisposition to the development of DH has been demonstrated. Both DH and celiac disease (CD) show an increased expression of HLA-A1, HLA-B8, HLA-DR3, and HLA-DQ2 haplotypes. Environmental factors are also important; monozygotic twins may have DH, CD, and/or GSE with variable symptomatology.
Evidence is mounting that the autoantigen in DH is transglutaminase (TGase), specifically epidermal TGase3, and the skin lesions are caused by dermal deposition of circulating immune complexes that contain both IgA and TGase3. This is supported by the finding that skin-bound IgA precipitates in DH lesions contain TGase3. Also, serum from DH patients has been shown to contain high-affinity IgA antibodies directed against TGase3. Both skin and gut TGases are highly homologous, and serum from patients with GSE, with or without skin disease, has been shown to contain IgA antibodies to both skin and gut types. The target autoantigen, TGase3, has not been demonstrated in normal papillary dermis, suggesting it is already bound in circulating complexes and deposited in the papillary dermis, rather than originating from the papillary dermis.
The leading theory for DH is that genetic predisposition to gluten sensitivity, coupled with a diet high in gluten, leads to the formation of IgA antibodies to gluten-TGase complexes. These antibodies cross-react with TGase3, and IgA/TGase3 complexes deposit within the papillary dermis to cause the lesions of DH.
IgA deposits in DH skin have been shown to function in vitro as a ligand for neutrophil migration and binding in skin. Neutrophil accumulation in the skin of patients with DH is the characteristic histologic finding. Collagenase and stromelysin 1 may be induced in basal keratinocytes by cytokines released from neutrophils or by contact with keratin from damaged basement membrane matrix. Stromelysin 1 may contribute to blister formation.
Deposits of C3 also may be present in a similar pattern at the dermoepidermal junction. The membrane attack complex, C5-C9, also has been identified in perilesional skin, although it may be inactive and not contribute to cell lysis.
IgA deposits disappear after long-term (up to 10 y) avoidance of dietary gluten.
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Now isn't this special.....gluten from barley and malts! THIS IS WHAT THEY USED WHEN CONING THIS S_ _ _! i'M SO DAMN MAD . i HOPE TO GOD THEY READ THIS FORUM BECAUSE I'M GOING TO GET THOSE SOB'S IF IT'S THE LAST FRIGGIN THING i DO ON THIS EARTH.
IM MISERABLE AND i'M GOING TO DO EVERYTHING IN MY POWER TO MAKE THEM MISERABLE!!!
i SAID A LONG TIME AGO TO WATCH THE DERMATOLOGY FIELD JUST EXPLODE WITH BUSINESS.
YEAH, THAT AND THE CANCER FIELD.
i THINK THIS IS GOING TO HAVE (ALSO) SOMETHING TO DO WITH uv LIGHT......i'M SEARCHING
AS HARD AS i CAN TO FIND INFO OUT ABOUT THIS.
tHANK YOU FOR THE INFO CATHOLIC 4 LIFE!
ps THE MEDS THEY SAY USE FOR dh IS: DAPSONE
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Post by LONDON as a guest on Mar 1, 2006 8:25:16 GMT -5
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Post by questionhair1 on Mar 1, 2006 10:11:27 GMT -5
A cat flea can give you a tapeworm. There are some 200 kinds of botfly type of maggots that can infest humans. A maggot can get into your nasal passage (or anywhere) as a result of a tick bite. The fly transmits its eggs to humans by way of a tick. It deposits its eggs on the tick. Humans are infested when bitten by a tick. Apparently the folks at Davis and CDC are not especially interested in original research on vector borne disease. Also MRSA tends to colonize the nose. New hybrid strains can also invade deep tissue. Can be fatal. Hi: This is interesting. I hate to see it buried in this thread. If you have any more thoughts on this, I hope you will start a new thread. The bug-vector idea is something I still like to look at. Please see the Lyme section of this board - I last left off looking at this back then - I think the subject heading is Biting Fly . . . something something Lyme - hopefully it is still on page one. If you have any more links or ideas or references on this, I'd be interested in reading more. QH
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Post by London at work on Mar 1, 2006 10:22:34 GMT -5
Carrie ( i hope you see this; it's from the Netherlands; I havethe entire packet-another 80 + or so poster abstracts that go along with these)
London at stupid work with morgellons, swollen feet, herpes on my arm........and I'm hasving a bad had day too; gotta gun anyone?
Selection of potential fungal agents of bioterrorism using evolutionary criteria
G.S. de Hoog Centraalbureau voor Schimmelcultures, Utrecht, the Netherlands
The list of potential agents of bioterrorism issued by the USA Department of Occupational Safety and Environmental Health (OSEH) presently contains a single fungus, Coccidioides immitis. This is the agent of Valley Fever, a disseminating disease which commonly infects humans but exceptionally takes a fatal course, then mainly in patients with impaired acquired immunity. C. immitis is not the only fungal species known to cause fatal disease; the question then arises whether other fungi should be put on the list. Agents should combine host-specific pathogenicity with a high degree of virulence. The probability that species display these criteria optimally in terms of bioterrorism is determined by the evolutionary history of the group at hand. The fungal kingdom is reviewed in search of clades with (1) shared virulence factors including species with (2) mammal hostdependence with dual life cycles and (3) production of zoodemes while (4) fitness is increased. In addition (5) the degree of adaptation is discussed. About 400 fungal species have been reported from humans; this is less than 0.5% of the fungi known to date. Suitable criteria are encountered in only a small fraction, the remaining species being occasional opportunists or superficial pathogens. Required properties are combined in only two species. Among these is not C. immitis. A03 Experimental coevolution A. Buckling A04 Genome reduction in bacterial pathogens and symbionts N.A. Moran Department of Ecology and Evolutionary Biology, University of Arizona, Tucson, Arizona, USA When bacterial lineages make the transition from free-living or facultatively parasitic life cycles to permanent associations with hosts, they undergo a major loss of genes and DNA. Complete genome sequences are providing a detailed view of which genes are lost, and it is now apparent that relatively few genes are universally preserved. Analysis of genome sequences indicate that gene loss occurs largely as the result of mutations fixed through genetic drift, resulting in deletion, inactivation and erosion of genes. High levels of genetic drift are expected to result from the reduction in genetic effective population size that accompanies obligate dependence on hosts. These high levels of genetic drift underlie some other characteristic features of small bacterial genomes, including rapid sequence evolution, biases in nucleotide composition and amino acid content of encoded polypeptides, and thermal instability of secondary structures of gene products. Some or all of these features have been documented for phylogenetically diverse groups of hostdependent bacteria, including mycoplasmas, rickettsiae, chlamydeae, and gammaprotoebacterial endosymbionts of insects. The latter, which include Buchnera and Wigglesworthia, are relatively closely related to well-known bacteria with larger genomes, such as Escherichia coli and Yersinia pestis; comparative analyses allow more exact reconstruction of the process of genome reduction in these groups. Continued completion of more genome sequencing projects will allow more detailed understanding of the evolutionary processes that underlie the radical genome changes observed in pathogenic and symbiotic bacteria.
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Post by London at work on Mar 1, 2006 10:24:34 GMT -5
Carrie ( i hope you see this; it's from the Netherlands; I havethe entire packet-another 80 + or so poster abstracts that go along with these)
London at stupid work with morgellons, swollen feet, herpes on my arm........and I'm hasving a bad had day too; gotta gun anyone? ****************
Selection of potential fungal agents of bioterrorism using evolutionary criteria
G.S. de Hoog Centraalbureau voor Schimmelcultures, Utrecht, the Netherlands
The list of potential agents of bioterrorism issued by the USA Department of Occupational Safety and Environmental Health (OSEH) presently contains a single fungus, Coccidioides immitis. This is the agent of Valley Fever, a disseminating disease which commonly infects humans but exceptionally takes a fatal course, then mainly in patients withimpaired acquired immunity. C. immitis is not the only fungal species known to cause fatal disease; the question then arises whether other fungi should be put on the list. Agents should combine host-specific pathogenicity with a high degree of virulence. The probability that species display these criteria optimally in terms of bioterrorism is determined by the evolutionary history of the group at hand. The fungal kingdom is reviewed in search of clades with (1) shared virulence factors including species with (2) mammal hostdependence with dual life cycles and (3) production of zoodemes while (4) fitness is increased. In addition (5) the degree of adaptation is discussed. About 400 fungal species have been reported from humans; this is less than 0.5% of the fungi known to date. Suitable criteria are encountered in only a small fraction, the remaining species being occasional opportunists or superficial pathogens. Required properties are combined in only two species. Among these is not C. immitis. A03 Experimental coevolution A. Buckling A04 Genome reduction in bacterial pathogens and symbionts N.A. Moran Department of Ecology and Evolutionary Biology, University of Arizona, Tucson, Arizona, USA When bacterial lineages make the transition from free-living or facultatively parasitic life cycles to permanent associations with hosts, they undergo a major loss of genes and DNA. Complete genome sequences are providing a detailed view of which genes are lost, and it is now apparent that relatively few genes are universally preserved. Analysis of genome sequences indicate that gene loss occurs largely as the result of mutations fixed through genetic drift, resulting in deletion, inactivation and erosion of genes. High levels of genetic drift are expected to result from the reduction in genetic effective population size that accompanies obligate dependence on hosts. These high levels of genetic drift underlie some other characteristic features of small bacterial genomes, including rapid sequence evolution, biases in nucleotide composition and amino acid content of encoded polypeptides, and thermal instability of secondary structures of gene products. Some or all of these features have been documented for phylogenetically diverse groups of hostdependent bacteria, including mycoplasmas, rickettsiae, chlamydeae, and gammaprotoebacterial endosymbionts of insects. The latter, which include Buchnera and Wigglesworthia, are relatively closely related to well-known bacteria with larger genomes, such as Escherichia coli and Yersinia pestis; comparative analyses allow more exact reconstruction of the process of genome reduction in these groups. Continued completion of more genome sequencing projects will allow more detailed understanding of the evolutionary processes that underlie the radical genome changes observed in pathogenic and symbiotic bacteria.
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Londons bad day from hell
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Post by Londons bad day from hell on Mar 1, 2006 10:25:55 GMT -5
oops, sorry, I did not know that went on two times> I have no way of removing it. So sorry, I suck!
London
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Post by skytroll on Mar 1, 2006 10:54:56 GMT -5
London,
Kick the he.l outta the devil while you are down there. That sucka thinks he's got power.
Lives in too many of these creaps, heh?
I wonder if that stuff I had, was similar to DH. Hey, it could the that olecranon bursitis Tam Tam talked of? mmmmmmmmm I miss Tam Tam don't you?
London some good articles there. Something about that fungus still lingers, say fungus and algae mix with other things. They are having a good time altering our lives aren't they?
Take care,
Skytroll
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Post by London to skytroll on Mar 1, 2006 11:06:07 GMT -5
They have got one for every thing. From folliculitis tio super folliculitis to burrows compresses.......Sky, no one read this but I urge you to! Find my post ( prob. about page 2 somewhere, titiled "now this is weird" and when it opens up, just scroll down until you see skin infections and the stuff they say to use on them!
Of course, no one read it! I feel like that it is written in the form of secret coding or tagging by them. I made a copy of it and last night when I was googling some of the medicines that it says to use, well i got blogs being pul;ed up and one was a poem about our diease! weird!!! check it out/ Plus, the medicine it recomends to use is right on> even a little a head of us!
London
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Post by informative guest on Mar 2, 2006 21:51:09 GMT -5
List of Animal Pathogens for Export Control 1 - Chemical Weapons Precursors - Dual-use chemical manufacturing facilities and equipment and related technology - Dual-use biological equipment - Biological agents - Plant pathogens - Animal pathogens April 2005
Viruses
AV1. African swine fever virus
AV2. Avian influenza virus 2
AV3. Bluetongue virus
AV4. Foot and mouth disease virus AV5. Goat pox virus AV6. Herpes virus (Aujeszky's disease) AV7. Hog cholera virus (synonym: swine fever virus) AV8. Lyssa virus AV9. Newcastle disease virus AV10. Peste des petits ruminants virus AV11. Porcine enterovirus type 9 (synonym: swine vesicular disease virus) AV12. Rinderpest virus AV13. Sheep pox virus AV14. Teschen disease virus AV15. Vesicular stomatitis virus AV16. Lumpy skin disease virus AV17. African horse sickness virus
Except where the agent is in the form of a vaccine.
This includes only those Avian influenza viruses of high pathogenicity as defined in EC Directive 92/40/EC:
“Type A viruses with an IVPI (intravenous pathogenicity index) in 6 week old chickens of greater than 1.2: or
Type A viruses H5 or H7 subtype for which nucleotide sequencing has demonstrated multiple basic amino acids at the cleavage site of haemagglutinin”
Bacteria
AB3. Mycoplasma mycoides
Genetic Elements and Genetically-modified Organisms
AG1 Genetic elements that contain nucleic acid sequences associated with the pathogenicity of any of the microorganisms in the list.
AG2 Genetically-modified organisms that contain nucleic acid sequences associated with the pathogenicity of any of the microorganisms in the list.
Technical note : Genetic elements include inter alia chromosomes, genomes, plasmids, transposons, and vectors whether genetically modified or unmodified.
Nucleic acid sequences associated with the pathogenicity of any of the micro-organisms in the list means any sequence specific to the relevant listed micro-organism:
- that in itself or through its transcribed or translated products represents a significant hazard to human, animal or plant health; or
- that is known to enhance the ability of a listed micro-organism, or any other organism into which it may be inserted or otherwise integrated, to cause serious harm to human, animal or plant health.
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Post by skytroll on Mar 2, 2006 21:54:11 GMT -5
What is the resource informative?
Come on, now
Skytroll
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Post by INFORMATIVE GUEST on Mar 2, 2006 23:50:06 GMT -5
DEAR MS. SKYTROLL,
I WILL BE HAPPY TO GIVE THAT TO YOU ON A PM TOMORROW. PLUS MANY OTHERS AS WELL.
DON'T THINK TOO MANY HERE ON FORUM CARE FOR IT ANYWAY, BUT I KNEW LONDON
AND PEOPLE LIKE YOURSELF MIGHT LIKE TO VIEW IT, SO THAT'S WHY I POSTED THE PARTIAL ARTICLE WHERE I DID.
HERE IS SOME MORE FROM THAT ARTICLE;
Some information from your local federal government……Thought you All might want to see the truth and veracity of their nomenclature…… I just love our government. And no way, conspiracy theories Exist, not with our government system. No way!
a. Agents of concern. A thematic paper and presentation could usefully set out the approach that could be followed nationally by States Parties in developing a national control list of "agents of concern" to prevent the misuse of such agents by individuals within the State. This thematic paper could start by recognizing that the term "pathogenic microorganisms and toxins" is very broad and includes many microorganisms and toxins which pose little hazard. The principles of risk assessment applicable to human, animal and plant pathogenic microorganisms and toxins in order to draw up the national list of "agents of concern" should be set out and should be broader than just the inherent properties of the agent. b. Legislation addressing the security of "agents of concern." This thematic presentation could start by recognising that in most States Parties there will already be existing legislation to protect public, animal and plant health and the environment. This could set out the required physical containment and access control for the various categories of pathogenic microorganisms and toxins. Although these are primarily intended to ensure that the agents do not escape and are not inadvertently released, they should already include many of the requirements to provide physical security and access control to prevent unauthorized access to or acquisition of the agents of concern. This thematic presentation could then continue to address the importance of good legislation, coordination between the various government agencies involved -- which are frequently different for human, animal and plant pathogens -- enforcement,and the requirement for such legislation to have penal clauses. b. Facilities and Equipment. The standards and guidelines for containment equipment, facility design and security arrangements are already well established with various internationally used standards and guidelines (such as those by the WHO (Laboratory Biosafety Manual, Second Edition, 1993), OIE and the US CDC/NIH guidelines (BMBL, 4th Edition, 1999)). This is an area that was discussed in detail by the Ad Hoc Group during its deliberations and is a topic that needs little debate at the experts meeting in August 2003. The common understanding that emerges should be the use of existing standards which a country can nationally upgrade if it feels such upgrading to be necessary -- for example to handle specific pathogenic microorganisms under a higher biosafety level.
************************** AND MY FAVORITE FROM PART OF THE ENTIRE ARTICLE SAYS:
77. Terminology. For effective use of the limited time available for the experts meeting in Geneva, it will be important to avoid sidetracks and confusion resulting from the different meanings of some of the terms used in the discussions. At the ARW it became clear that there are different meanings and understandings for the following terms:a. Biosafety. This is frequently used to mean the policies, practices and equipment to prevent biological agents harming humans, animal or plants or the environment. However, with the advent of the Cartagena Protocol on Biosafety, the term biosafety is sometimes used to refer to the procedures relating to living modified organisms. b. Biosecurity. It was pointed out that in some languages, such as French, the same word is used for biosafety and biosecurity. The term biosecurity also has a particularly wide meaning in respect of FAO activities.
c. Deliberate release. In the BTWC arena, this term is usually used to relate to a deliberate release by terrorists or by State Action. There is another meaning -- a planned deliberate release -- in the context of the European wide directives relating to the deliberate release of genetically modified organisms or microorganisms.There could be benefit in a common understanding being agreed prior to the start of the experts meeting in August as to what the terms "biosafety", "biosecurity" and "deliberate release" will mean during the experts meeting. Alternatively, each State Party should be asked to make it clear what is meant by the terms in their papers or presentations.
PS: the chemicle agent they are using as a fumigator ( plus other areas this agent is in) is Called METHYL BROMIDE. IT WAS SUPPOSED TO BE PHASED OUT BY JANUARY 2005.
INFORMATIVE GUEST
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Post by curry on Mar 3, 2006 2:11:39 GMT -5
London...you are funny...no guns on bad hair day...that's a no-no. It's already bad enough to throw the brush and break the mirror...a gun...nope...go to the corner.
I haven't looked over everything but I did look at some...there is a lot of information here. I think I have a good idea about your concerns with this. I've been puttin' my two cents in on so many threads...I can't remember where but I asked you a couple questions...I don't think your responded or I've not noticed...could have missed it.
I'm only asking because I would like to get a better idea of your outlook on the disease and/or process.
What is the point of the immune depleting, altering, suppress, destroying, toxic chemicals. Where do your concerns lie with those issues? Along with the bio-genetic, man made or altered bacteria/fungus/viral organism...what is your basic bottom line thought at this particular time? I'm only asking so I can get an idea of where you are with it in connection with the symptoms of the disease we share. Drug resistance, disease process, intended for disease or ignorant to ramifications of toxins by our lovely government!?
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Post by curry on Mar 3, 2006 4:16:55 GMT -5
London...I just posted this on the Gulf War thread...it may seem like I'm joking around...in which I am a bit. However...I am also very serious...could the SLF vector your man made weapon? Could they do that...hell yeah. I've been reading that Veterans have been saying the government has been testing on them for years. I can get an image and identify with that in my head. How though...I guess easy enough for the scientist. But wouldn't that kill the fluke? Something has got to be vectoring the suppose to be tick-borne disease's other than the tick...but how could that be with out damaging the vector. As a matter of fact...why doesn't the bacteria harm the vector anyway? I've not thought of that either. It's not like the vector for the filarial worms that get vectored in varies stages right. It's not like the bacterium is in the intestine of the vector...doesn't it have to be in the gut to get vectored?Okay...I'm getting too carried away...I'm going to bed. I am cut and pasting that reply from there here because I would like for you to read it when you have time and I'd like to see what you think of that and if it is possible...hope that's okay:
With all the talk of manipulation of bio warfare...the SLF is the most tested upon and played with trematode...hum...because of it's ability to conform and wealth of knowledge of the parasite because it is so common here...hum again...and I could imagine...maybe lack of identification. I wonder if this is partially why I am having such a difficult time finding good...identification benchmark aids on this parasite. I should probably consider that.
Thinking about that...whew...I can can get with that...I can understand and feel that. What if they are playing with London's bactofungivira...creating an even deadlier method of mass destruction!?
Something to consider...especially for my naive, stubborn mind...
Can I post this over on London's thread in case she doesn't come here? I would like her to be "proud" of me...I'm trying to come out of my box....consider all options.
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rem
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Post by rem on Mar 3, 2006 4:20:04 GMT -5
like to join in on the subject « Message sent on Today at 4:08am »
-------------------------------------------------------------------------------- ive been reading alot of your posts, im not as smart as you all but i admire your consistant research on this nightmare were suffreing. Me and my huusband, son, and daughter are suffering from this for a little over a year now. we have been treated by dr. schwartz as we live in new mexico but since november i have not heard a word from the man. at first we seemed to be gettin better but as always these BASTAAAAAAAAArds came back. Ive only posted a few times but we have mold in our home, 4 linds, moved out, still itched. also aresnic was found 45 miles away from where i live big investigation on news I sent link to london and skytroll, i beleive this is something created b our government, and is in my opinion some type of bacterial, fungas, mutated worm, fluke, or parasite. From my own experiences with lots of crazy ideas from antifungal creams to baking soda. it reacts to this stuff. what you yhink...I only wish i had the answer as we all do. but lets chat, by the way your posts along with london, skytroll,and spec and fris have helped us so much in understanding. bless all you guys
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Post by curry on Mar 3, 2006 4:43:08 GMT -5
I spend so much time on here some times lately...I get what I get from it...which is good right!?
By that kind of post remygarcia makes it worth it too...thanks.
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rem
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Post by rem on Mar 3, 2006 4:55:47 GMT -5
carrie, i will email you in the a.m. opps i forgot it is a.m. haha..got to have a sense of humor with this sh@t, and talk more about the investigation just read todays local newspaper and there is an article i will forward. Lets kick this thing in the ass and beat it, with all of us together i know weve gotten further and more answers from each other than what our so called doc's are giving us.. night
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rem
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Post by rem on Mar 3, 2006 5:09:49 GMT -5
carrie, i just changed my user name to rem so my posts will no longer view as remygarcia, bye
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Post by London toCARRIE on Mar 3, 2006 5:29:18 GMT -5
Dear remygarcia, this: the chemicle agent they are using as a fumigator ( plus other areas this agent is in) is Called METHYL BROMIDE. IT WAS SUPPOSED TO BE PHASED OUT BY JANUARY 2005. BUT NOW THEY ARE ASKING FOR EXTENSIONS. AND JUST THIS MORNING, BUSH GIVES INDIA NUCLEAR POWER. CARIE, YOUR MIND RACES, HELL I CANT REMEMBER WHAT ALL YOUR QUESTIONS WERE.......AND NO, i LOVE TO READ YOUR POST AND YOUR QUESTIONS.....I DON'T KNOW WHICH ONE YOU WERE REFERRING TO WHEN YOU ASKED IF I HAD SEEN YOUR POST OR NOT.....WHICH ONE, THE ONE ON THE FLUKE? OH YES, i SAW IT YESTERDAY> THE ONE WHERE SPEC WRITES FOR EVERYONE TO REALLY LOOK AT IT. YES, THAT COULD BE IT.. COW/ FLUKE/ NOT ONLY DID THEY GIVE THE BOVINE GROWTH HORMONES,,,,,, SAY THEY DID NOT AND YOU HAVE A NICE CLEAN LITTLE 30 ACRES WITH 25 CATTLE STOCK OR SO. AND YOU WERE A GOOD PERSON THAT JUST LIKED TO LIVE OUT IN COUNTRY AND HAVE A " PLAY -RANCH ...WHATEVER-SAY THAT WAS JUST A HOBBY OF YOURS AND YOU WERE NEVER GOING TO DO ANYTHING CHEMICLE WITH THE ANIMAL-NO GROWTH HORMONES, ETC., WELL, WHAT COULD HAPPEN FOR THE COW TO GET SICK? HE COULD HAVE EATEN INFECTED INSECT OR IT COULD HAVE BITTEN HIM OR.....(NEVERMIND, IT'S ALL ANTHRAX, WHETHER HE GOT IT FROM INSECT VECTOR OR SPORE (bT.) ........ sAY THAT DID NOT EVEN HAPPEN. wELL GUESS WHAT? SEE THAT LITTLE WORD IN THE FIRST SENTENCE THAT I WROTE TO REMY IN THIS POST (AT TOP) WELL, THEY PUT IT IN THE FEED WITHOUT ANY OF THE FARMERS KNOWING IT. aNTHRAX/ Bt POISONING. FOOT AND MOUTH DISEASE. IT'S BIG RIGHT NOW. TO ANSWER YOUR Q. RE: HOW DOES IT SURVIVE? HELL IF I KNOW! IF IT WAS GENETICALLY MODIFIED THEN IT CAN W/STAND 90% OF ANYTHING -PROBABLY. HEY CARRIE SINCE I HAVE BEEN SEARCHING NON-STOP THE LAST 4-5 MONGHTS , i CAN HONESTLY SAY i HAVE BEEN PULLING UP ARTICLES AND ILNESSES FROM FLUE TO STREP TO DAIBETES, TO HERPES-TO HERPES 16 TO ALZHEIMERS, HIV TO LEAUKEMIA. AND YOU KNOW WHAT? THAT IS RIGHT , DAMN RIGHT! (SOME RUDE-ARSE PERSON "A GUEST" , OF COURSE WROTE ABOUT 1 month ago and said " WELL LAST WEEK YOU THOUGHT IT WA S THIS AND NOW THIS....." and now I'm finding out all these carry are fitting in... throw in the synthetic ploiomyvirus, Legionaires disease, leprosy....... there is fibers from the textiles from cotton from fiberoptics from protein........ ( sorry about my mispelled words and typos) I got to go back to bed asap! hopefully tomorrow evening, I can finish writing to you maybe the vector is something that is genetically modified as well and can withstand fire, heat, you name it.......I honestly do not know. but I sure as hell do not know how you could be eating a bowl of tuna pata or something and out of nowhere, worms just show up... This aint right carrie I heard that ( okay, I just fell asleep and woke up as I was typing this to you) oh, I must go, but will be in touch. I'm glad you azre part of this forum! How old are your kids? London
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Post by specuelatin on Mar 3, 2006 5:47:18 GMT -5
yeah, I have a worm frozen in my freezer that was embedded in the fake crab., uuueeww or how ever you speell it.
remygarcia,
Always good to know when people are benefitting from these posts, really good to know.
Hey I heard some of Scwartz patients are from Socorro.. really want to find out if the arsenic dump is anywhere near Socorro..
Please leave the link or let me know!! I have a relative in Socorro.
Thanks, spec
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rem
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Post by rem on Mar 3, 2006 6:09:42 GMT -5
spec, glad to know you saw my post, the sight is in springer, its quite a ways from soccoro but just think about the roswell poop. I know that there is some toxic crap there (aliens they claim) maybe, maybe this stuff is alienated. dont know but i think our govt knows and i also think bush is part of it. hmm makes me wonder all most as long as he's been president this stuff has surfaced more and more and even more of an issue know with the war in iraq what do you think...
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