Post by Lisa on Aug 2, 2005 13:15:51 GMT -5
Found this interesting and thought I'd pass it along.
This person (soficrow) is giving their take on where our disease may originate from.
www.abovetopsecret.com/forum/thread150959/pg2
At this link, they give a lot of information on the subject.
www.abovetopsecret.com/forum/thread112220/pg1
(taken from the very bottom of the page)
Fibromuscular dysplasia (FMD) is caused by a misfolded protein called a-smooth muscle actin (a-SMA) - obviously an infectious prion protein. a-SMA is likely "Mad Cow" in its early stages, before it affects the brain directly. ...More important, a-SMA is able to access/infect/take over virtually any cell in the human body - and any other lifeform - via actin proteins present in every cells' cytoskeleton, membrane and nucleus. ...Misfolded actin proteins are implicated in most new diseases in most species and kingdoms - from Dutch Elm disease and gall cancers to the new pseudorabies, airborne E. coli and super bugs (C. difficile, MRSA, VRE, flesh eating disease (necrotizing fasciitis), and ESBL-producing bacteria) to most human cancers.
FMD is particularly adaptable and able to evolve because it is an actin protein - there are actin proteins in virtually every cell in the body, and in every other lifeform on earth. ...So FMD's a-SMA is like a master key that can get into any cell, anywhere. For example, if an infectious microbe comes into contact with FMD lesions inside the body, the a-SMA can get into the microbe's cells through actins in the microbe's cytoskeleton or membrane, and take it over and make it mutate - without killing it. ...This process creates new diseases too, and a-SMA can infect people, animals, plants, insects, bacteria, viruses, spirochetes, molds, protozoa - anything.
Eventually the lesions break through the vessel walls and then, infect new cells on contact - this is how FMD spreads from the blood and lymph vessels to glands, bone, joints and other parts of the body. Prions adapt to new cells by changing their "conformation" - meaning they mutate so they can infect other cells more easily. So as the disease moves through the body infecting new cells, it literally morphs into new and different diseases as it travels.
As more and more cells are infected, the (misfolded) a-SMA builds up to form "fibrosis" and create "vacuoles" or holes in tissue (where cells are missing), creating cold-sore-like lesions. In blood vessels for example, the fibrosis creates blockages called "stenosis" and the vacuoles become aneurysms.
This person (soficrow) is giving their take on where our disease may originate from.
www.abovetopsecret.com/forum/thread150959/pg2
At this link, they give a lot of information on the subject.
www.abovetopsecret.com/forum/thread112220/pg1
(taken from the very bottom of the page)
Fibromuscular dysplasia (FMD) is caused by a misfolded protein called a-smooth muscle actin (a-SMA) - obviously an infectious prion protein. a-SMA is likely "Mad Cow" in its early stages, before it affects the brain directly. ...More important, a-SMA is able to access/infect/take over virtually any cell in the human body - and any other lifeform - via actin proteins present in every cells' cytoskeleton, membrane and nucleus. ...Misfolded actin proteins are implicated in most new diseases in most species and kingdoms - from Dutch Elm disease and gall cancers to the new pseudorabies, airborne E. coli and super bugs (C. difficile, MRSA, VRE, flesh eating disease (necrotizing fasciitis), and ESBL-producing bacteria) to most human cancers.
FMD is particularly adaptable and able to evolve because it is an actin protein - there are actin proteins in virtually every cell in the body, and in every other lifeform on earth. ...So FMD's a-SMA is like a master key that can get into any cell, anywhere. For example, if an infectious microbe comes into contact with FMD lesions inside the body, the a-SMA can get into the microbe's cells through actins in the microbe's cytoskeleton or membrane, and take it over and make it mutate - without killing it. ...This process creates new diseases too, and a-SMA can infect people, animals, plants, insects, bacteria, viruses, spirochetes, molds, protozoa - anything.
Eventually the lesions break through the vessel walls and then, infect new cells on contact - this is how FMD spreads from the blood and lymph vessels to glands, bone, joints and other parts of the body. Prions adapt to new cells by changing their "conformation" - meaning they mutate so they can infect other cells more easily. So as the disease moves through the body infecting new cells, it literally morphs into new and different diseases as it travels.
As more and more cells are infected, the (misfolded) a-SMA builds up to form "fibrosis" and create "vacuoles" or holes in tissue (where cells are missing), creating cold-sore-like lesions. In blood vessels for example, the fibrosis creates blockages called "stenosis" and the vacuoles become aneurysms.