Post by southcity on Oct 12, 2005 0:08:17 GMT -5
someone on a different forum posted this information last week. the entire thread can be found here... www.biology-online.org/biology-forum/viewtopic.php?p=17929#17929
There is quite a few areas of research and investigational direction given in this persons post. This is also the first explanation, as far as I can tell so far, that I am inclined to believe.. Im looking forward to reading the information that all of you may come up with after reading this below....
Southcity.
********************************************
The fiber disease constitutes an infection with a genetically modified
quorum sensing stem cell related micro organism.
Basically the organism represents a cyano bacterium that knows a stage as a giant cell and as a mold.
Diversification resulting in morphologically different acting micro organisms takes place by shaft, giant cell but also by fission.
Hence a coccidiod cyano bacterium like(single, diploid and chain forming)micro organism that lives on the skin and in wounds is the mayor responsible agent.
No association is made by micro biologist or histopathologist in connection to this pathogen.
Nota bene,
Intermediate stages resemble a (quorum sensing) transparant gel
that constitutes a protoplasm. This makes detection of the agent in biopsy impossible during standard clinical research.
Single cell micro organisms function on a multi cellular level,
communication between species of the same kind as well as different kind is possible.
The micro organism has been fused with a mayor parasitic protozoa, a butterfly(lepidoptera) and a mammal specie.
Multi lineage differentiation is fact.
The fibers constitute protein.
Specks are chitin like polymers.
Fibers represent cell that are most connected to sensing parts like antennea, tongue, feet and wing.
The colors of the fibers are connected to the colored wing pattern of lepidoptera. Actually they constitute variants with a semi parasitic nature.
Keratin erosion is evident.
The breeding ground for the organism in skin is almost exclusively the follicle(stem cell per definition)
The phenomenon is directly linked to proteome and genome research and it concerns commensal bacteria without the status of known pathogen, but some members of the tree are known as nosocomial (like the pseudomona)
Infection resembles most protothecosis.
Amphoteracine B and amikacine will be the most logic choice for therapy but no protocol exist for this type condition.
Sulfa Methoxazole/ Trimethoprim and Tetracycline and derivatives are suppressive but not curative.
Also azoles like itraconazole, econazole, micanozole are effective.
Infection may directly or indirectly relate to IBS, fibromyalgia, chronic fatigue syndrome, therapy resistant skin lesions, onycholisis, neurogenic bladder and chorioretinitis.
Sincerely,
Gaiapacha
(by the way, does anyone recognize this persons name or meaning of it. perhaps its a place(location)? or is it simply a name?)
There is quite a few areas of research and investigational direction given in this persons post. This is also the first explanation, as far as I can tell so far, that I am inclined to believe.. Im looking forward to reading the information that all of you may come up with after reading this below....
Southcity.
********************************************
The fiber disease constitutes an infection with a genetically modified
quorum sensing stem cell related micro organism.
Basically the organism represents a cyano bacterium that knows a stage as a giant cell and as a mold.
Diversification resulting in morphologically different acting micro organisms takes place by shaft, giant cell but also by fission.
Hence a coccidiod cyano bacterium like(single, diploid and chain forming)micro organism that lives on the skin and in wounds is the mayor responsible agent.
No association is made by micro biologist or histopathologist in connection to this pathogen.
Nota bene,
Intermediate stages resemble a (quorum sensing) transparant gel
that constitutes a protoplasm. This makes detection of the agent in biopsy impossible during standard clinical research.
Single cell micro organisms function on a multi cellular level,
communication between species of the same kind as well as different kind is possible.
The micro organism has been fused with a mayor parasitic protozoa, a butterfly(lepidoptera) and a mammal specie.
Multi lineage differentiation is fact.
The fibers constitute protein.
Specks are chitin like polymers.
Fibers represent cell that are most connected to sensing parts like antennea, tongue, feet and wing.
The colors of the fibers are connected to the colored wing pattern of lepidoptera. Actually they constitute variants with a semi parasitic nature.
Keratin erosion is evident.
The breeding ground for the organism in skin is almost exclusively the follicle(stem cell per definition)
The phenomenon is directly linked to proteome and genome research and it concerns commensal bacteria without the status of known pathogen, but some members of the tree are known as nosocomial (like the pseudomona)
Infection resembles most protothecosis.
Amphoteracine B and amikacine will be the most logic choice for therapy but no protocol exist for this type condition.
Sulfa Methoxazole/ Trimethoprim and Tetracycline and derivatives are suppressive but not curative.
Also azoles like itraconazole, econazole, micanozole are effective.
Infection may directly or indirectly relate to IBS, fibromyalgia, chronic fatigue syndrome, therapy resistant skin lesions, onycholisis, neurogenic bladder and chorioretinitis.
Sincerely,
Gaiapacha
(by the way, does anyone recognize this persons name or meaning of it. perhaps its a place(location)? or is it simply a name?)