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Post by skytroll on Jun 21, 2008 13:22:18 GMT -5
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Post by skytroll on Jun 21, 2008 13:30:11 GMT -5
......."Some algae can be harmful to humans. A few species produce toxins that may be concentrated in shellfish and finfish, which are thereby rendered unsafe or poisonous for human consumption. The dinoflagellates (class Dinophyceae) are the most notorious producers of toxins. Paralytic shellfish poisoning is caused by saxitoxin or any of at least 12 related compounds. Saxitoxin is probably the most toxic compound known; it is 100,000 times more toxic than cocaine. Saxitoxin and saxitoxin-like compounds are nerve toxins that interfere with neuromuscular function. Alexandrium tamarense and Gymnodinium catenatum are the two species most often associated with paralytic shellfish poisoning. Diarrheic shellfish poisoning is caused by okadaic acids that are produced by several kinds of algae, especially species of Dinophysis. Neurotoxic shellfish poisoning, caused by toxins produced in Gymnodinium breve, an organism associated with red tides, is notorious for fish kills and shellfish poisoning along the coast of Florida in the United States. When the red tide blooms are blown to shore, wind-sprayed toxic cells can cause health problems for humans and other animals that breathe the air." www.britannica.com/EBchecked/topic/14828/algae/31715/ToxicitySkytroll
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Post by godsgrace on Jun 21, 2008 22:45:49 GMT -5
ciguatera ia also a toxin from fish.
It causes seafood poisioning.
A Professor at the University of Hawaii studied many people with chronic fatigue syndrome(Myalgic Encephalomyelitis) and found that people with CFS/ME have higher levels of ciguatera than people with acute seafood poisioning.
No wonder when I was ill I had this strange feeling I was being poisioned.....slowly.
godsgrace
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Post by skytroll on Jun 21, 2008 23:22:17 GMT -5
Saxitoxin Saxitoxin From Wikipedia, the free encyclopedia Jump to: navigation, search Chemical structure of saxitoxin Saxitoxin Systematic (IUPAC) name (3aS-(3a-α,4-α,10aR*))2,6-diamino- 4-(((amino-carbonyl)oxy)methyl)-3a,4,8,9-tetrahydro- 1H,10H-pyrrolo(1,2-c)purine-10,10-diol Identifiers CAS number 35523-89-8 PubChem 37165 Chemical data Formula C10H17N7O4 Molar mass 299.29 SMILES N=C1N[C@ h](COC(N)=O)[C@H]3[C@] 2(N=C(N)N3)N1CCC2(O)O Complete data Saxitoxin (STX) is a cyanotoxin found in marine dinoflagellates (algae). It is a neurotoxin that is a selective sodium channel blocker. The United States military isolated saxitoxin and assigned it the chemical weapon designation TZ. It is almost unique among toxins in that it acts very quickly, in a matter of minutes. The median lethal concentration (LCt50) of TZ is 5 mg·min/m³. The medical importance of saxitoxin is in relation to red tide in shellfish and causes the paralytic shellfish poisoning (PSP) food poisoning. The blocking of the sodium channel produces a flaccid paralysis that leaves its victim calm and conscious through the progression of symptoms. Death is from respiratory failure. It is listed in schedule 1 of the Chemical Weapons Convention. According to the book Spycraft, U2 spyplane pilots were provided with needles containing Saxitoxin to be used in the event escape was impossible. Though its early isolation and characterization were related to military efforts, saxitoxin has been more important to cellular research in delineating the function of the sodium channel.".... en.wikipedia.org/wiki/Saxitoxinthe sodium channel (salt) mmmmm so the military as well. cellular research looking for function of the sodium channel. Would that be to utilize it to introduce fungus into body plans? Skytroll
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Post by Jill on Jun 22, 2008 6:29:40 GMT -5
A woman I know had a trip to emergency last fall. She said it was due to food poisoning- a sea food meal. Shrimp, etc.
2 weeks ago, she was at the doctor again. Had a 'cyst' removed. She said it's been there a while- it's on her stomach- she thought it was a boil- but it didn't go away.
The 'cyst' was removed and a plug installed for the drainage. Several days later, she was back at the doctors- he removed the plug- and the packing and stiched it up.
She had been visiting the tanning booth just prior to the 'cyst' presenting itself.
I think there was something in her lower tract- intestines- that was drawn out by the tanning. Something that was caused by the sea food fiasco.
Somehow- I think it's related to the shellfish and some sort of parasite. Could have to do with the algae as well.
Jill
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Post by skytroll on Jun 22, 2008 8:06:52 GMT -5
It is the algae, there are HABs, this is where the critter is formed, I believe. HAB: A proliferation of unicellular phytoplankton that can cause massive fish or shellfish kills, contaminate seafood with toxins and alter aquatic ecosystems in ways that humans perceive as harmful. There are two phenomena, the so called red tide and toxin-producing plankton. Red Tide: Water discoloration by vastly increased unicellular phytoplankton that induces deterioration of aquatic ecosystems and occasional fishery damage. Toxin-producing Plankton: Phytoplankton species that produce toxins within their cells and contaminate fish and shellfish throughout the food chain. Countermeasure: Measures that are implemented to prevent or minimize damage from HABs. Some red-tide species have multiple scientific names due to past taxonomic amendments (e.g. the synonym of Karenia mikimotoi: Gymnodinium nagasakiense; basynonym of K. mikimotoi: Gymnodinium mikimotoi). This booklet mostly uses the same scientific names as in the Integrated Report, but in some cases scientific names from the source reference are used. www.cearac-project.org/wg3/publications/HAB_Booklet.pdfThis seems to be a problem everywhere, seems whatever put in wild to change species, may have back fired. Here I mean the fisheries, not only was land food (gm) manipulated, but, the fisheries as well. GM modified fish. .............. "Table 1 summarizes the status of red-tide events in the NOWPAP region. To date, 75 red-tide species have been recorded in the NOWPAP region. Three flagellate species (Heterosigma akashiwo, Noctiluca scintillans, Prorocentrum minimum) and one diatom species (Skeletonema costatum) have been frequently recorded in the coastal waters of all NOWPAP members. All three flagellate species have caused extensive damage to local fisheries. Other common and damage-causing dinoflagellate (Dinophyceae) species include Karenia mikimotoi, Gymnodinium sanguineum and P. micans. In recent years, Cochlodinium polykrikoides has caused serious damage to fisheries in Japan and Korea." Cochlodinium polykrikoides: Click on plate link. Look at #6 please and would appreciate comments. Some of that look familiar? Also look at the morphology of it. Who it morphs. Skytroll botany.si.edu/references/dinoflag/Taxa/Cpolykrikoides.htm I believe the toxin produced is Saxitoxin. Skytroll
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Post by skytroll on Jun 22, 2008 8:17:07 GMT -5
Others identified int he project: Karenia mikimotoi, Gymnodinium sanguineum and P. micans. others mentioned: Heterosigma akashiwo, Thalassiosira subtilis and Skeletonema costatum. for cleanup this has been used, and not why it is not good. The polymer does not dissolve. Synthetic polymers Synthetic polymers remove plankton through flocculation and sinking. To date, 15 types of synthetic polymers have been tested, which are listed below. Tested synthetic polymers: Petrosize J, Petrosize U, Polyethyleneimice, Polyoxyethylene Laurylamine, Polyoxyethylene Lauryl Alchohol Ether, Tween20, Tween40, Tween60, Tween80, Aminoethyl Amylose Acetate, FLONAC N1, sodium alginate, KAYAFLOC C-533-1P2, KAYAFLOC C-533-1O2 and giant kelp 1 product of KYOWA TECNOS CO., LTD (http://www.kyowatecnos.com/) 2 product of KAYAFLOC CO., LTD (http://www.kayafloc.co.jp/) According to laboratory experiments, some synthetic polymers caused cell lysis or deformation of Chattonella marina cells, even at low concentrations (< 10 ppm). However, synthetic polymers are currently not used, because they are toxic to other aquatic organisms and do not decompose in seawater (Kagoshima Pref., 1986, 1987). Proof that it remains. So use of this may have perpetuated the problem and done more damage. www.cearac-project.org/wg3/publications/HAB_Booklet.pdf
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Post by skytroll on Jun 22, 2008 8:23:02 GMT -5
Other species to investigate: Karenia mikimotoi Gymnodinium sanguineum P. micans. Heterosigma akashiwo, Thalassiosira subtilis Skeletonema costatum. SCOTLAND: The potentially harmful dinoflagellate Karenia mikimotoi has regularly been identified in Scottish waters, but with few major environmental consequences. However, a red tide in a number of sea lochs of the Firth of Clyde in 1980 was associated with fish deaths in Loch Fyne. Karenia cell Subsequently K. mikimotoi assumed reduced significance in Scottish waters for nearly two decades, with the next recorded bloom of red tide proportions being in 1999 in Orkney and in 2003 in the Orkney and Shetland Islands. In 2006, phytoplankton monitoring programmes indicated the presence of a dense bloom of K. mikimotoi around the Scottish coast. The bloom was first evident on the west coast and travelled from Mull up the west coast and then to the Northern Isles of Orkney and Shetland as well as down the east coast as far as Stonehaven. Satellite data strongly suggest that the K. mikimotoi bloom developed offshore and was advected towards the coast. It was then carried on the Scottish coastal current in a northerly direction. Karinia mikimotoi has the potential to case the death of farmed fish through the generation of anoxia and/or the production of toxins. Fortunately, few if any farmed fish deaths occurred during the 2006 bloom. However, a major impact were mortalities of benthic organisms with many faunal mortalities being reported to SEPA and FRS during August 2006 and thought to be related to the K. mikimotoi bloom. A research project at SAMS has been funded by the Crown Estate to investigate the 2006 K.mikimotoi bloom in more detail. This project will involve mathematical modelling of bloom transport, and through collaboration with Plymouth Marine Laboratory the development of algorithms to allow early warning of K.mikimotoi bloom development fro satellite remote sensing observations. Link to the recent Harmful Algae News article on the K.mikimotoi bloom by Davidson et al. tinyurl.com/3ptqruwww.sams.ac.uk/research/departments/microbial-molecular/mmb-project-themes/harmful-algal-blooms/researchproject.2007- 05-01.1004870833 Ireland: www.marine.ie/NR/rdonlyres/1821AB9C-676C-40F5-9BCD-4D3C803EEA1D/0/MEHS21.pdf tinyurl.com/4nq7fywww.habes.net/about/harmful_algal_blooms.htmSkytroll
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Post by skytroll on Jun 22, 2008 8:40:06 GMT -5
Gymnodinium sanguineum One with Luciferase connection: www.biologie.uni-hamburg.de/b-online/e44/44f.htm"Gymnodinium sanguineum is a dinoflagellate (microscopic plant). Dinoflagellates occasionally multiply rapidly to create blooms when conditions are favourable. These blooms include the 'red tides' that are capable of killing fish and other marine life. Red tides are so named because of the high density of cells loaded with the red photosynthetic pigment beta-carotene. Red tides have been recorded in Lake Illawarra in the past, causing the death of some fish and eels. Excessive nutrients may be to blame for the blooms, as well as the input of humic material to the lake during storms." www.lia.nsw.gov.au/lakelife/algae.htmlOther dinos; note the one above mentioned: www.serc.si.edu/labs/phytoplankton/guide/dinoflagellates/dinos.jspSkytroll
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Post by skytroll on Jun 22, 2008 9:02:03 GMT -5
P. micans Toxicity: Although P. micans is capable of forming extensive blooms, it is usually considered harmless (see Taylor & Seliger 1979; Anderson et al. 1985; Graneli et al. 1990). It may excrete substances that inhibit diatom growth, but apparently these substances do not enter the food chain or affect organisms at higher trophic levels (Uchida 1977). There are only a few reports of P. micans having caused problems: shellfish kills in Portugal (Pinto & Silva 1956) and South Africa (Horstman 1981). Toxicity of this species needs confirmation. Early reports on P. micans being a paralytic shellfish poison (PSP) producer (Pinto & Silva 1956) are unconfirmed, and recent incidents involving shellfish mortality have been attributed to oxygen depletion (Lassus & Berthome 1988). botany.si.edu/references/dinoflag/Taxa/Pmicans.htmNote the main phosphorus link in oceans: orthophosphate wasP Wang Zheng-fang1, Zhang Qing1 and Gong Min1 (1) Second Institute of Oceanography, SOA, 310012 Hangzhou www.seriestemporales-ieo.net/en/galeria/galeria4/galeria4.htmSkytroll Received: 8 December 1994 Accepted: 16 June 1995 Abstract Laboratory culture experiments showed that <100μ mol/L nitrate, amonium or mixture of amino acids promote the growth of the red tide organismProrocentrum micans Ehrenb, but that >100μmol/L of ammonium, or mixture of glycine and glutamate was harmful to growth, and that orthophosphate wasP. micans’ main phosphorous source in the ocean. Presence of 80μ mol/L EDTA, 0.5 to 1 μmol/L Fe3+, 1.0 to 20.0 μ mol/L Mn2+ 0.1 to 0.4 μmol/L Co2+ in the culture medium could improve the growth ofP. micans. Vitamin B1 promoted growth, but vitamin B12 and biotin did not. The estimated minimum cell quotas (q o) for nitrogen and phosphorus being 0.74 pmole/cell and 0.045 pmole/cell show that phosphorus (more than nitrogen) limits the growth ofP. micans in the study area. Key words Prorocentrum micans - chemical environment culture experiment This project was supported by the Natural Science Foundation of Zhejiang Province.
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Post by lilsissy on Jun 22, 2008 11:27:09 GMT -5
Yes this is the direction, Ospins exist in humans alga, fungus, across the kingdoms. Humans normally have three that help us see light and colors. There are giving us more so to change our ability to utilize the energy produced from light. Our cells and neurons will be able to harvest and store this energy . Making new technologies never imaginable before involving human's possible. en.wikipedia.org/wiki/OpsinPlease all read up about opsin, Jennifer
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Post by skytroll on Jun 22, 2008 12:55:39 GMT -5
Lilsissy, this comes around full circle to the bacteriorhodopsins, and then the halorhodopsins.
The b first than the halo. which involves the red algae and the blooms.
But, there is bioluminescence and there is light from sun. Biolumines can light in dark,
photosynthesis throught sun. and it is the Retinal, involved.
I believe one of these is the what caused the original later gene transfer, with the photons, involving protons and electrons.
It was a perturbation event, most likely the very bioturbation event Darwin hoped for.
Two genes of interest in H. akashiwo, not previously reported in any chloroplast genome, include tyrC, a tyrosine recombinase, which we hypothesize may be a result of a lateral gene transfer event, and an unidentified 456 amino acid protein, which we hypothesize serves as a G-protein-coupled receptor. The H. akashiwo chloroplast genomes share little synteny with other algal chloroplast genomes sequenced to date.
The List again, and then will see where dunaliella opsins and bacteriorhodopsins and halodopsins fit in.
Karenia mikimotoi Gymnodinium sanguineum P. micans. Heterosigma akashiwo, Thalassiosira subtilis Skeletonema costatum. Two more to go from list of HABS....that cause human disease, etc.
but this as an Interlude: brings us full circle around to the self assembing, and recombinant....of Silent Superbug.picasaweb.google.com/SILENTSUPERBUG/CBL00103/photo#5026379995537513778Skytroll
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Post by skytroll on Jun 22, 2008 12:58:54 GMT -5
And the circle won't be broken.............by and by.........
Skytroll
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Post by skytroll on Jun 23, 2008 2:32:16 GMT -5
Melan opsins Melanopsin is a photopigment found in specialized photosensitive ganglion cells of the retina that are involved in the regulation of circadian rhythms, pupillary reflex, and other non-visual responses to light. In structure, melanopsin is an opsin, a retinylidene protein variety of G-protein-coupled receptor. Melanopsin, atypical in vertebrates, functionally resembles invertebrate opsins, including an apparent intrinsic photoisomerase activity.[1] It is presumed that melanopsin signals through a G-protein of the Gq family, as invertebrate opsins are known to do, but this is not firmly established. en.wikipedia.org/wiki/Melanopsinelectrophysiology: Electrophysiology is the study of the electrical properties of biological cells and tissues. It involves measurements of voltage change or electrical current flow on a wide variety of scales from single ion channel proteins to whole tissues like the heart. In neuroscience, it includes measurements of the electrical activity of neurons, and particularly action potential activity. en.wikipedia.org/wiki/ElectrophysiologicalTalking "free energy"....... how many ways, only Tesla knew. Skytroll
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Post by skytroll on Jun 23, 2008 2:38:44 GMT -5
Biological clocks,,,,,,,,,,,,,,,'from: Biologists Close In on the 'Tick-Tock' Genes ,,,,"But a bigger mystery remains for humans: What are all these time-keeping genes and proteins doing in skin and muscle cells? ''We are ticking all over our bodies,'' Dr. Takahashi said. ''In my view, the suprachiasmatic nucleus is still the master pacemaker. All these peripheral clocks may work in a hierarchy similar to the heart. ''Each heart cell can beat intrinsically but the heart functions as a synchronous whole with a single pacemaker to drive the system. The same may be true for the circadian system. Besides, there is no good evidence of feedback from the periphery to the master clock.'' Nevertheless, scientists at Cornell University had their shining success last spring when they showed that human biological clocks could be reset at the back of the knee. If this experiment can be replicated by other scientists, researchers will need to explain how the light signal is carried from skin cells to brain cells. The pigments in blood are among the candidates. In the meantime, the new findings may have practical value. ''If we know the genes that make up the clock, there should be drugs that target the clock alone and not the rest of the body,'' Dr. Takahashi said. ''This may be useful in treating insomnia caused by defects in the clock system.'' Dr. Sancar has another suggestion. Opsins use vitamin A as a cofactor, which is why people are told to eat carrots for good vision. But cryptochromes use vitamin B2 as a cofactor in absorbing light, he said. Thus vitamin B2 might be helpful to some people in treating jet lag, depression and other problems linked to biological clocks that are out of whack. " tinyurl.com/6xcttxquery.nytimes.com/gst/fullpage.html?res=9F0CE1DE143DF936A25751C1A96E958260&sec=&spon=&pagewanted=all Opsins related to biological clocks. ON SKIN AND MUSCLE CELLS. Skytroll
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Post by skytroll on Jun 23, 2008 2:43:56 GMT -5
This will help explain some of the clock, oral mucosa and skin: "Circadian Expression of Clock Genes in Human Oral Mucosa and Skin Association with Specific Cell-Cycle Phases Georg A. Bjarnason,*† Richard C. K. Jordan,*‡§ Patricia A. Wood,¶ Qi Li,∥ David W. Lincoln,** Robert B. Sothern,†† William J. M. Hrushesky,¶ and Yaacov Ben-David∥ From the Toronto-Sunnybrook Regional Cancer Centre,* Toronto, Ontario, Canada; the Department of Medicine† and the Faculty of Dentistry,‡ University of Toronto, Toronto, Ontario, Canada; the Departments of Pathology§ and Medical Biophysics and Division of Cancer Biology Research,∥ Sunnybrook and Women’s College Health Sciences Centre, Toronto, Ontario, Canada; the WJB Dorn VA Medical Center,¶ Columbia, South Carolina; the Research Service,** Stratton Veterans Affairs Medical Center, Albany, New York; and the College of Biological Sciences,†† University of Minnesota, St. Paul, Minnesota Accepted February 5, 2001. Small right arrow pointing to: This article has been cited by other articles in PMC. T Abstract We studied the relative RNA expression of clock genes throughout one 24-hour period in biopsies obtained from the oral mucosa and skin from eight healthy diurnally active male study participants. We found that the human clock genes hClock, hTim, hPer1, hCry1, and hBmal1 are expressed in oral mucosa and skin, with a circadian profile consistent with that found in the suprachiasmatic nuclei and the peripheral tissues of rodents. hPer1, hCry1, and hBmal1 have a rhythmic expression, peaking early in the morning, in late afternoon, and at night, respectively, whereas hClock and hTim are not rhythmic. This is the first human study to show a circadian profile of expression for all five clock genes as documented in rodents, suggesting their functional importance in man. In concurrent oral mucosa biopsies, thymidylate synthase enzyme activity, a marker for DNA synthesis, had a circadian variation with peak activity in early afternoon, coinciding with the timing of S phase in our previous study on cell-cycle timing in human oral mucosa. The major peak in hPer1 expression occurs at the same time of day as the peak in G1 phase in oral mucosa, suggesting a possible link between the circadian clock and the mammalian cell www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=11337377skytroll
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Post by skytroll on Jun 23, 2008 2:55:52 GMT -5
Well, this seems to tell us more: "Circadian rhythms in Neurospora crassa: Clock gene homologues in fungi Laura M. Lombardi and Stuart BrodyCorresponding Author Contact Information, E-mail The Corresponding Author Division of Biological Sciences, Molecular Biology, UCSD, La Jolla, CA 92093-0116, USA Received 23 February 2005; accepted 23 June 2005. Available online 9 September 2005. Abstract Computer-based analysis of a total of 17 filamentous fungal and yeasts genomes has shown: (1) homologues of frq, wc-1, wc-2, and vvd, key gene components of the Neurospora crassa clock, are present in Magnaporthe grisea, Gibberella zeae, and Podospora anserina, suggesting an frq-based oscillator in these organisms; (2) some fungal species that are more distantly related to Neurospora, such as Rhizopus oryzae do not appear to have frq homologues; (3) many fungal species that do not appear to contain frq, such as Aspergillus nidulans, do contain wc homologues; (4) of 11 well-described genes classified as clock-controlled genes (ccgs), in Neurospora, all of them were found to have homologues in other fungi; (5) the ccg-8 gene of N. crassa has homologies to opi1p, a transcriptional regulatory gene in Saccharomyces cerevisiae involved in inositol regulation. This suggests the possibilities of rhythmic inositol regulation, and/or a cascade of rhythmic activation of other genes in N. crassa. Keywords: Neurospora crassa; Neurospora crassa clock homologues; Clock homologues; Clock-controlled genes; Computer-based analysis of fungal genomes; Rhythmic inositol regulation; Inositol regulation; frq homologues; vvd homologues Article Outline 1. Introduction 2. Results and discussion 2.1. Homologues of clock genes and clock-controlled genes 2.2. The homology of ccg-8 to Saccharomyces cerevisiae’s opi1p Acknowledgements References Table 1. Neurospora clock components in selected fungia View table in article a Only the above organisms (with the exception of Aspergillus nidulans, as a point of reference) contained frq homologues out of the 17 fungal genomes and all non-redundant GenBank CDS translations + RefSeq Proteins + PDB + SwissProt + PIR + PRF searched, although some other fungi contain homologues for the one or both of the WC proteins. Fungal genomes searched include the complete genomes of Candida glabrata, Debaryomyces hansenii, Encephalitozoon cuniculi, Eremothecium gossypii, Kluyveromyces lactis, Podospora anserina, Saccharomyces cerevisiae, Schizosaccharomyces pombe, and Yarrowia lipolytica, while unfinished genomes include Aspergillus nidulans, Candida albicans, Chaetomium globosum, Cryptococcus neoformans, Gibberella zeae, Magnaporthe grisea, Neurospora crassa, Rhizopus oryzae, and Ustilago maydis. b Shading of the column indicates that the protein sequences used for the BLAST SEARCH belonged to that organism. c Merrow and Dunlap (1994). d Lewis et al. (1997). e Homology to frq also noted in Dunlap and Loros (2005). f The Podospora anserina sequence was obtained via the Fungal Genetics Stock Center website (www.fgsc.net) and homologues were found using protein–nucleotide BLASTS (tblastn). g All protein–protein BLASTS (blastp) were run using the default setting of the www.ncbi.nlm.ni.gov software (expect: 10, Filter:default). h The question mark signifies that genomes for these organisms are not yet available, and, therefore, the presence of homologues for clock components other than the FRQ protein cannot presently be ascertained. i Homologies also noted in Dunlap and Loros (2005). j Greene et al. (2003). Table 2. Neurospora clock-controlled gene homologues in selected fungia View table in article a Only fungal species with frq homologues and whose sequences were available were included in this table. Sequences that were not available included S. fimicola, L. australiensis, and C. spinulosa. b Shading of the column indicates that the protein sequences used for the BLAST SEARCH belonged to that organism. c All protein–protein BLASTS (blastp) were run using the default setting of the www.ncbi.nlm.ni.gov software (Expect: 10, Filter:default). d Although no significant E values were obtained, these three species contain hydrophobin genes (Fuchs et al., 2004 and Talbot et al., 1993). e The P. anserina sequence was obtained via the Fungal Genetics Stock Center website (www.fgsc.net) and homologues were found using protein–nucleotide BLASTS (tblastn). f ccg-12 length is 26 amino acids, so search parameters were lessened to Filter:none. g Zhu et al. (2001). Table 3. Homologues to the S. cerevisiae opi1p over production of inositol) genea View table in article a All protein–protein BLASTS (blastp) were run using the default settings of the www.ncbi.nlm.nih.govsoftware (Expect: 10, Filter:default). b Shading of the column indicates that the protein sequences used for the BLAST SEARCH belonged to that organism. c The actual size of the protein is unknown, as the sequence was obtained via a protein–nucleotide BLAST (tblastn) of the not-yet-translated P. anserina sequence on the Fungal Genetics Stock Center website (www.fgsc.net). Fungal Genetics and Biology Volume 42, Issue 11, November 2005, Pages 887-892"........ tinyurl.com/6eratmwww.sciencedirect.com/science?_ob=ArticleURL&_udi=B6WFV-4H2PJWK-3&_user=10&_ rdoc=1&_fmt=&_orig=search&_sort=d&view=c&_acct= C000050221&_version=1&_urlVersion=0&_userid= 10&md5=75f4301af72bc70517da3802d406d749 Seems the giant Neospora Crassa study from years ago by a multitude of Universities, businesses, and govt. is now quite useful. Skytroll
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Post by lilsissy on Jun 23, 2008 18:16:48 GMT -5
All makes you wonder about ancient alchemy , the eye of toad.
Would be ospin from toad's.
Wonder how long Alchemists have been on to this?
I knew a big link to this Morgellons would tell us why we cannot eat carrots any longer. Karen , my boyfriend and I all of a sudden within 1 year get bloody stools if we eat carrots. Too much beta caroteen already from build up. Don't you think?
I had a dream sometime back around the time I first read about Luciferase .
I walked out on my mothers back porch in the dream and there were beautiful glowing butterflies on all the power lines. As beautiful as it appeared there was an air of foreboding. Ford factory appeared dark and uninhabited in the background. She lives across the park from Ford's.
I ran across an article last year that said they had found Chlamidia in white butterfly's. It was an old article. Do you remember how most of the male butterflys died off a few years back? They are making a comeback now but lost the greater part of the Males. Isn't one of the ospins related to the testies? Wonder if it started with the butterfly's?
Forgive me my spell check is off and I do not spell well.
The origin of the word spell, HMMMMMMMMMMMM.
I began writing a children book after that dream about the white butterfly in connection with the Indian Prophecy about the White Buffalo that was how much the dream effected me.
Great work Skytrool , Great, now how do we do sputum cultures on this? Lilsissy
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Post by lilsissy on Jun 23, 2008 18:33:28 GMT -5
godsgrace, I wonder how they test for ciguatera. Will see if I can find something on itt. My Doctor has been letting me have test that I request , so far anyway. That is very informative infromation. I was diagnosed with FMS. CIGUATERA HMMMMMMMMMMMM Jennifer
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Post by godsgrace on Jun 23, 2008 22:50:56 GMT -5
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