sci.tech-archive.net/Archive/sci.med.diseases.lyme/2006-10/msg00978.htmlFoley Hearts Chuck wrote:
Please distribute for your health safety, due to the lack of competent
"law enforcement":
==============================
A result of the deliberate medical neglect of the immune suppression
result of Borrelia infection by Yale (especially Eugene Shapiro) and
the Infectious Disease Society of America (IDSA) is this weird and
horrible skin disease:
www.fox11az.com/news/topstories/stories/kmsb-20061005-wfaajc-morgellons.b6607d0.html(It's probably a lichen or a fungi- and it is not surprising that the
CDC has not decoded the DNA of the growths, or at least have not
reported their findings, since it is clearly a real disease and not
psychogenic, ie., the CDC is perpetually full of crap, ie., Google MMR
and SSPE and see that brain damage is a known outcome of these
childhood immunizations, ie., CDC dropped the ball on all infectious
diseases and on all factual matter relating to health, ie., see my
website for the links to the European patent database and the patents
owned by CDC staff, ie., CDC staff are all more dangerous scum than
these fungi due to their chronic lies and biotech profiteering.)
The reason for these new phenomena, is that it is known that HLA
tolerization (no antibodies are produced after a time) to mycoplasmal
lipoproteins (the opposite of what is seen in people who have an
inflammatory response to the borrelial-mycoplasmal lipoproteins, which
are the same thing, as shown in the Aug 2003 Lyme RICO complaint
graphic on my website),...
...results in the inability of the mammal to defend against new fungi
or new lipoprotein bacerial toxins, which are the same kind that are
shed by borrelia (Lyme spirochetes), as explained with the following
published scientific journal articles (that means VERIFY my claims on
the National Library of Medicine Database):
www.actionlyme.org/BIOWEAPONEERS_CORIXA_YALE_TLRS.htm[You will see more of these weird infections related to immune
incompetence to tropical infections and new fungi due to global
warming, and this is the reason it is so ridiculous to fund any CDC
biowarfare labs. You are all at great risk due to the fact that Yale
refuses to let us use their only VALID test for Lyme, which is patented
under US patent 5,.618,533 and related Borrelia specific antiflagellin
antibody tests and all they and IDSA do, is *LIE* about Lyme
borreliosis.]
Signaling through TLR-2 by lipoproteins may represent a double-edged
sword for host responses to chronic intracellular pathogens such as M.
tuberculosis. Short-term signaling through TLR-2 activates macrophages
and initiates acute inflammation that may help control initial
infection. In contrast, prolonged TLR-2 signaling in macrophages
results in down-regulation of certain critical immune functions, such
as MHC-II Ag processing. M. tuberculosis infects, survives, and
persists in macrophages. The ability of M. tuberculosis to survive
acute inflammation positions the bacilli to take advantage, through
secretion of lipoproteins such as LprG and LpqH, of this
down-regulation of macrophage immune function."
www.jimmunol.org/cgi/content/full/173/4/2660(No antibodies are produced, after a time. Borrelia do the same
thing through blebbing.)
Same type of antigen as LYMErix is found in mycoplasmas:
"Lipoproteins and lipopeptides have been identified in a large number
of microorganisms, the most prominent ones being mycobacteria,
mycoplasms, and spirochetes. They have been found to exhibit both a
strong innate inflammatory response in the host and an enduring
adaptive immune response in mammalian hosts (16). The strong
proinflammatory capacities of lipoproteins were first described for
outer surface proteins A and B of Borrelia burgdorferi, which are also
highly immunogenic (17) and have lately been the basis for a Lyme
disease vaccine development (18). These compounds exhibit an
triacylated lipid anchor structure comprising an
N-palmitoyl-S-[2,3-bis(palmitoyloxy)-(2RS)-propyl]-(R)-cysteinyl
(Pam3Cys) moiety at the N terminus (19), a feature that was previously
described for the Braun lipoprotein from Escherichia coli (20). Because
the N-terminal Pam3Cys modification is essential for immunoactivation
caused by lipoproteins of B. burgdorferi as well as of another
spirochete, Treponema pallidum (21), subsequent studies investigating
immune responses to spirochetes used synthetic lipopeptides (22). The
Pam3Cys moiety was also reported to be present in cytokine-inducing
lipoproteins of Mycobacterium and Mycoplasma spp. (23, 24); thus, it
can be regarded as a highly conserved molecular motif among different
classes of bacteria. In Mycoplasma fermentans, the presence of a
macrophage stimulating lipopeptide, termed 2-kDa macrophage-activating
lipopeptide (MALP-2), was observed, being stimulatory active at
picomolar concentrations (25). This compound, in contrast to the
predominant lipopeptide structures present in lipoproteins of E. coli,
B. burgdorferi, and mycobacteria, lacks the N-palmitoyl group, thus
containing a diacylated (Pam2Cys) lipid anchor structure at the N
terminus. Following studies revealed the presence of closely related
compounds in other Mycoplasma spp. (26).." from:
www.jimmunol.org/cgi/content/full/173/4/2683More on this type of immune suppression from MedLine
Must read:
www.jbc.org/cgi/content/full/274/47/33419 (UCONN's
Justin Radolf)
"Toll-like receptors (TLRs) 2 and 4 are signal transducers for
lipopolysaccharide, the major proinflammatory constituent in the outer
membrane of Gram-negative bacteria. We observed that membrane
lipoproteins/lipopeptides from Borrelia burgdorferi, Treponema
pallidum, and Mycoplasma fermentans activated cells heterologously
expressing TLR2 but not those expressing TLR1 or TLR4. These
TLR2-expressing cells were also stimulated by living motile B.
burgdorferi, suggesting that TLR2 recognition of lipoproteins is
relevant to natural Borrelia infection. Importantly, a TLR2 antibody
inhibited bacterial lipoprotein/lipopeptide-induced tumor necrosis
factor release from human peripheral blood mononuclear cells, and
TLR2-null Chinese hamster macrophages were insensitive to
lipoprotein/lipopeptide challenge. The data suggest a role for the
native protein in cellular activation by these ligands. In addition,
TLR2-dependent responses were seen using whole Mycobacterium avium and
Staphylococcus aureus, demonstrating that this receptor can function as
a signal transducer for a wide spectrum of bacterial products. We
conclude that diverse pathogens activate cells through TLR2 and propose
that this molecule is a central pattern recognition receptor in host
immune responses to microbial invasion."
www.jimmunol.org/cgi/content/full/173/4/2683How Borreliosis causes chronic illness- shedding lipoproteins and
antigen variation from multiple strains:
"2.1 Methods of Treatment
"An important aspect of the invention is the recognition that Borrelia
VMP-like sequences recombine at the vls site, with the result that
antigenic variation is virtually limitless. Multiclonal populations
therefore can exist in an infected patient so that immunological
defenses are severely tested if not totally overwhelmed."
(Due to this antigenic variation, the current CDC testing schema for
Lyme is totally bogus, all you can use for detection is Borrelia
specific flagellar antibodies, and it is impossible to ever have a Lyme
vaccine..)
"Mycoplasma fermentans is capable of fusing with lymphocytes and
changing their immunological characteristics."
www.immuno-sci-lab.com/html/mycoplasma.htmlSpiroplasma in ticks Mycoplasma fermentans and immortalized cells
"Spiral membranous inclusions resembling spiroplasmas have been seen in
brain biopsies taken from patients with Creutzfeldt-Jakob disease."
More
The Multiple Sclerosis- Mycoplasma link
www.fas.org/irp/congress/2002_cr/s092002.html Congressional
Record- Bioweapons sold by USA to Iraq to use against Iran: Page
S8987-S8998
"...Over the protest of some Pentagon skeptics,
the Reagan administration began allowing the Iraqis to buy a
wide variety of "dual use" equipment and materials from
American suppliers. According to confidential Commerce
Department export-control documents obtained by NEWSWEEK, the
shopping list included a computerized database for Saddam's
Interior Ministry (presumably to help keep track of political
opponents); helicopters to transport Iraqi officials;
television cameras for "video surveillance applications";
chemical-analysis equipment for the Iraq Atomic Energy
Commission (IAEC), and, most unsettling, numerous shipments
of "bacteria/fungi/protozoa" to the IAEC. According to
former officials, the bacterial cultures could be used to
make biological weapons, including anthrax...."
One of the Plum Island strains mycoplasmas (fungi) of stealth
disablers; PubMed:
: J Hyg (Lond). 1983 Jun;90(3):441-9. Links
Immunogenic variation among the so-called LC strains of Mycoplasma
mycoides subspecies mycoides.
bullet Smith GR, Oliphant JC.
Much evidence of immunogenic heterogeneity among the LC strains of
Mycoplasma mycoides ssp. mycoides emerged from cross-immunization and
-hyper-immunization experiments in mice in which three LC strains
(Vom/Plum Island, 74/2488, and Mankefar 2833) were used for challenge
purposes. All heterologous LC-strain vaccines cross-immunized against
the three challenge strains, but protection was usually only 'partial',
i.e. significantly less than that given by homologous vaccine.
Cross-hyperimmunization with all heterologous LC but not SC strains
produced protection against challenge with Vom/Plum Island that was
virtually 'complete', i.e. similar to that produced by homologous
vaccine. Challenge with 74/2488 gave generally similar results; but
against Mankefar 2833 six heterologous LC vaccines gave complete
protection and six did not. Vaccines prepared from the Smith (1423)
strain of M. mycoides ssp. capri gave some protection against Vom/Plum
Island but none against 74/2488 or Mankefar 2833. The cross-immunizing
ability of three further M. mycoides ssp. capri strains appeared to
resemble that of Smith (1423). In a cross-hyperimmunization experiment,
vaccines prepared from SC strains of M. mycoides ssp. mycoides varied
greatly in their ability to protect against challenge with strains
74/2488 and Mankefar 2833. PMID: 6190898 [PubMed - indexed for
MEDLINE]
Mycoplasmas cause lameness and inhibit milk production in
livestock:
1: J Clin Microbiol. 1975 Sep;2(3):165-8.Click here to read Links
Naturally occurring and experimentally induced mycoplasmal
arthritis of cattle.
bullet Stalheim OH, Page LA.
Mycoplasma agalactiae subsp. bovis strain Iowa 1136 was isolated
from synovial fluids of a clinical case of arthritis in cattle on
pasture in Iowa. When given to calves and cows by intra-articular or
intravenous injection, it caused severe and persistent joint infections
with fever, lameness, and swelling of the affected joints, plus
synovitis, tendonitis, and fibrinous-purulent synovial fluids of high
protein content. Intramammary administration of the organism caused
severe mastitis. Calves nursing the cows developed severe mycoplasmal
arthritis. PMID: 1176623 [PubMed - indexed for MEDLINE]
Squalene and Gulf War Illness. Good one.
www.sftt.org/cgi-bin/csNews/csNews.cgi?database=Unlisted.db&command=viewone&id=19What's the Stealth Pathogen?
What's the New Great Imitator of the Stealth Pathogen?
Lyme Scam 401- The Overall Picture of "Interests" - Who is doing
what with research that you and I paid for, but are not allowed to
have? Why/How are the Walker-Welds and the CIA-Bank Bushies involved?
www.john-loftus.com/bushnazi_1951.asp"The basic answer is that military and political leaders did not
want anything to mar the notion of a clean and virtuous victory in the
Gulf. And they certainly didn't want to raise the possibility that
there might be tens of thousands of "friendly fire" casualties who were
wounded not by the enemy but by our own actions. It is clear that the
military's first reaction to Gulf War syndrome was to treat the sick as
malingerers and weaklings. This colored the entire debate for two
years. In that time Gulf War syndrome simply didn't exist. If you said
you were sick you were either a coward or you were trying to hustle the
government for medical care or a pension." ...See the Lyme Scam 401
link above or "Against All Enemies," below.
If they worked so hard to suppress and discredit Gulf War Illness
veterans, then the "Plum Island accidental release of Lyme disease"
scenario sure fits the US Government's spin on Lyme borreliosis.
However, the State of Connecticut hires people far more insane that
people who who have Lyme -this permanent brain infection. Jessica
Gauvin is a raving lunatic.
Is Lyme a brain infection? Absolutely.
How soon after tick bite does it infect the brain?
Gee, maybe we should ask Ray Dattwyler
Why keep checking for Mail? The all-new Yahoo! Mail shows you when
there are new messages.
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