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Post by fritolay66 on Nov 30, 2009 15:09:12 GMT -5
MMP-9 is a Zn+2 dependent endopeptidase, synthesized and secreted in monomeric form as zymogen. The structure is almost similar to MMP2, another member of matrixmetalloproteinase family. The nascent form of the protein shows an N-terminal signal sequence ("pre" domain) that directs the protein to the endoplasmic reticulum. The pre domain is followed by a propeptide-"pro" domain that maintains enzyme-latency until cleaved or disrupted, and a catalytic domain that contains the conserved zinc-binding region. A hemopexin/vitronectin-like domain is also seen, that is connected to the catalytic domain by a hinge or linker region. The hemopexin domain is involved in TIMP (Tissue Inhibitors of Metallo-Proteinases) binding e.g. TIMP-1 & TIMP-3, the binding of certain substrates, membrane activation, and some proteolytic activities. It also shows a series of three head-to-tail cysteine-rich repeats within its catalytic domain. These inserts resemble the collagen-binding type II repeats of fibronectin and are required to bind and cleave collagen and elastin. Like other proteolytic enzymes, MMP-9 is first synthesized as inactive proenzyme or zymogens. Activation of proMMP-9 is mediated by plasminogen activator/plasmin (PA/plasmin) system. The regulation of MMP-9 activity is also controlled through TIMP-3. Expression MMP-9 expression is regulated by several cytokines and growth factors, including interleukins, interferons, EGF (Epidermal growth factor), NGF (Nerve growth factor), basic FGF (Fibroblast growth factor), VEGF (Vascular endothelial growth factor), PDGF (Platelet derived growth), TNF-a (Tumor necrosis factor), TGF-b (Tranforming growth factor), the extracellular matrix metalloproteinase inducer EMMPRIN and also osteopontin. Many of these stimuli induce the expression and/or activation of c-fos and c-jun proto-oncogene products, which heterodimerize and bind activator protein-1 (AP-1) sites within of MMP9 gene promoters. Localisation Peri/extracellular Function Primary function is degradation of proteins in the extracellular matrix. It proteolytically digests decorin, elastin, fibrillin, laminin, gelatin (denatured collagen), and types IV, V, XI and XVI collagen and also activates growth factors like proTGFb and proTNFa. Physiologically, MMP-9 in coordination with other MMPs, play a role in normal tissue remodeling events such as neurite gowth, embryonic development, angiogenesis, ovulation, mammary gland involution and wound healing. MMP-9 with other MMPs is also involved in osteoblastic bone formation and/or inhibits osteoclastic bone resorption. Homology Homology in amino acid sequence is seen with the other members of Metalloproteinase family especially with MMP-2. Mutations Germinal Not yet reported. Implicated in Entity Invasive and highly tumorigenic cancers Disease Elevated expression of MMP-9, along with MMP-2 is usually seen in invasive and highly tumorigenic cancers such as colorectal tumors, gastric carcinoma, pancreatic carcinoma, breast cancer, oral cancer, melanoma, malignant gliomas, chondrosarcoma, gastrointestinal adenocarcinoma. Levels are also increased in malignant astrocytomas, carcinomatous meningitis, and brain metastases. Oncogenesis MMPs promote tumor progression and metastasis in invasive cancers by degradation of the ECM (ExtraCellular Matrix), which consists of two main components: Basement membranes and interstitial connective tissue. Though ECM comprises of many proteins (laminin-5, proteoglycans, entactin, osteonectin) collagen IV is the major element. MMP-2 & MMP-9 efficiently degrade collagen IV and laminin-5 thereby, assisting the metastatic cancerous cells to pass through the basement membrane. The degradation of ECM not only assists migration of metastatic cancerous cells, but also allows enhanced tumor growth by providing necessary space. Further, it is noteworthy that the ratio of active to latent form of MMP-9 increased with tumor progression in invasive cancers. MMP-9, with its family members also promotes angiogenesis (a critical process required for tumor cell survival) by degrading the vascular basement membrane interstitium and also by releasing sequestered VEGF, which is a well know angiogenic molecule. Localization of MMP9 to the cell surface is required to promote tumor invasion and angiogenesis. Entity Arthritis, autosomal recessive osteolysis disorder, coronary artery disease, pulmonary-emphysema and diabetic retinopathy. en.wikipedia.org/wiki/MMP9 |
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Post by jeany on Nov 30, 2009 15:47:28 GMT -5
Kammy and Jeany, I believe what you are specifically looking for is MMP9. When looking at MMP9, you may also want to look more closely at the hypothalmus and its role. Frito Hi Frito, yes, thank you...MMP is an enzyme that is needed for tissue reproduction and Angiogenesis which is a process in building new blood vessels and wound healing. In fact, I was looking at this last night too while researching cysteine..could it be that you sent me your 'waves' over? ;D..I will also look in to the Hypothalamus/Thyroid connection. A dysfunctional endocrine/hormone system is IMO another aspect of susceptibility. Thanks for mentioning that, Frito! This is also interesting: Matrix metalloproteinases (MMPs) are zinc-dependent endopeptidases;
They are known to be involved in the cleavage of cell surface receptors, the release of apoptotic ligands (such as the FAS ligand), and chemokine/cytokine in/activation. MMPs are also thought to play a major role on cell behaviors such as cell proliferation, migration (adhesion/dispersion), differentiation, angiogenesis, apoptosis and host defense. The MMPs are initially synthesized as inactive zymogens with a pro-peptide domain that must be removed before the enzyme is active. The pro-peptide domain is part of the “cysteine switch.” This contains a conserved cysteine residue which interacts with the zinc in the active site and prevents binding and cleavage of the substrate keeping the enzyme in an inactive form
A zymogen (or proenzyme) is an inactive enzyme precursor. A zymogen requires a biochemical change (such as a hydrolysis reaction revealing the active site, or changing the configuration to reveal the active site) for it to become an active enzyme. The biochemical change usually occurs in a lysosome where a specific part of the precursor enzyme is cleaved in order to activate it.
The amino acid chain that is released upon activation is called the activation peptide. The pancreas secretes zymogens partly to prevent the enzymes from digesting proteins in the cells in which they are synthesised. Fungi also secrete digestive enzymes into the environment as zymogens.
The external environment has a different pH than inside the fungal cell and this changes the zymogen's structure into an active enzyme.
Doxycycline, at subantimicrobial doses, inhibits MMP activity, and has been used in various experimental systems for this purpose; such as for recalcitrant recurrent corneal erosions. It is used clinically for the treatment of periodontal disease and is the only MMP inhibitor which is widely available clinically. **I believe antibiotic residues in our water/food are also contributing to a malfunction of this specific enzyme. I've been taking high doses of Zinc and Magnesium and I am convinced, after I know how many beneficial effects it has on the metabolism and how important it is to keep our minerals well balanced, to be the KEY! IMO zinc plays a main role in fighting this disease! Zinc is part of my 'recovery plan' and I take it on a daily basis. After a relapse last summer I leveled my zinc intake up and noticed a decrease of symptoms (fibers, black specks, 'twitching' hair) in a short period of time. It also strengthens the immune system which is another factor we need to keep an eye on! Jeany |
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Post by fritolay66 on Nov 30, 2009 16:03:01 GMT -5
Jeany,
Look at the glycosylation aspect in respect to the MMPs and specifically MMP2 and especially MMP9.
Frito
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Post by fritolay66 on Nov 30, 2009 16:07:55 GMT -5
And then look at the interluekin 1, TNF alpha, and RAGE. It is here you will find the connection to amyloids.
Frito
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Post by jeany on Nov 30, 2009 16:13:03 GMT -5
And then look at the interluekin 1, TNF alpha, and RAGE. It is here you will find the connection to amyloids. Frito You're great, Frito!  Jeany |
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Post by kammy on Dec 1, 2009 19:04:28 GMT -5
Scale Insects I found this interesting photo above in the USDA archives and was reading it and it caused me to look... what are they talking about? www.sel.barc.usda.gov/coccoidea/scale2004.htm"Loss of a great coccidologist Jan Koteja was an innovative scientist and added numerous interesting and exciting bodies of information to the study of scale insects. According to ScaleNet he wrote 114 research papers on scale insects (and there likely are several others in press) and described at least 7 new families, 20 new genera, and 42 new species. He frequently had ideas that didn’t “fit in the box” of tradition; but in many instances his hypotheses have gained wide acceptance in coccidology. His findings in systematics, morphology, phylogeny, and fossils will remain important reading for scientist well into the future." **I wondered what a coccidologist did, and see that they specialize in "scale insects", so I looked at that: The scale insects are small insects of the order Hemiptera, generally classified as the superfamily Coccoidea. There are about 8,000 species of scale insects. "Most scale insects are parasites of plants, feeding on sap drawn directly from the plant's vascular system. A few species feed on fungal mats and fungi, e.g., some species in the genus Newsteadia in the family Ortheziidae. They secrete a waxy coating for defense; this coating causes them to resemble reptilian scales or fish scales, hence the name.Scale insects feed on a wide variety of plants, and many scale species are considered pests. Some types are economically valuable, such as the cochineal, Polish cochineal and lac scales. Scale insects' waxy covering makes them quite resistant to pesticides, which are only effective against the first-instar nymph crawler stage. However, scales are often controlled with horticultural oils, which suffocate them, or through biological control. en.wikipedia.org/wiki/Scale_insectScale insects are divided into three groups: (1) armored scales, (2) soft scales, and (3) mealybugs. The armored and soft scales are one of the most destructive groups of insects that attack ornamental crops. Mealybugs are not generally considered a problem on most woody ornamentals. The armored scales secrete a waxy covering over their bodies. This covering is not an integral part of the insect's body. The scale lives and feeds under this covering which resembles a plate of armor, hence the name. They vary in size from 1/16 inch to 1/8 inch in diameter and can be almost any color, depending on the species. Armored scales may be circular, oval, oblong, thread-like, or even pear-shaped. The female's armor is larger than that of the male, while the shape and color may be similar or distinctly different, depending upon the particular species. Soft scales also secrete a waxy covering, but it is an integral part of their body. Soft scales vary widely in color, size, and shape. They range from 1/8 inch to 1/2 inch in diameter and may be nearly flat to almost spherical in shape. BiologyThe armored scale life cycle is generalized as follows. The eggs are laid underneath the waxy covering and hatch over a period of one to three weeks. The newly hatched scales (called crawlers) move about over the plant until they locate succulent new growth. They insert their piercing-sucking mouthparts into the plant and begin feeding. Female scales lose their legs and antennae during the first molt. They molt a second time before reaching maturity and do not pupate. The cast skins (exuviae) are incorporated in the scale cover. Male scales go through two additional molts and pupate underneath the wax. Adult males are tiny two-winged, gnat-like insects without mouthparts. In some armored scales the adult stage is reached in six weeks, and there are several generations per year. In the females of the soft scales the antennae and legs are not lost, but are reduced to such an extent that though the adults can move about somewhat they seldom do. The wax when secreted, usually forms a sac at the rear end of the body enclosing the eggs, and the scale on the back of the insect becomes much thickened, forming a thick fluffy mass. The life cycle is similar to the armored scales except some soft scales require one year to reach maturity. entnemdept.ufl.edu/fasulo/woodypest/scales.htmarmored scale (Diaspididae) Scale Insects (Coccidae and Diaspididae) There are two types of scale insects, unarmored and armored. The females of the unarmored or soft scales (Coccidae) are oval to nearly circular and flat to nearly globular, and are sometimes partially covered with wax. Common examples are the brown soft scale, Coccus hesperidum L. (figure 348), and the hemispherical scale, Saissetia coffeae (Walker). Many species become stationary when fully developed. The males usually pupate under a small, thin, transparent, flat scale, and emerge as tiny winged adults. The females of the armored scales (Diaspididae) are flat, elongate to circular, and have a thick, protective shell or armor above and a very thin layer beneath the body. Common examples are the oleander scale, Aspidiotus nerii Bouch , and the greedy scale, Hemiberlesia rapax (Comstock) (figure 349). Once the motile first-instar nymphs settle down on a plant, the females never move again. The males transform to winged adults as with the unarmored scales. Scale insects suck sap from the plant, resulting in retarded growth and even defoliation. The unarmored scales, but not armored scales, excrete honeydew, which results in the growth of "sooty-mold" fungus and attracts ants. Pests of House Plants * Earthworms (Chaetopoda) * Slugs and Snails (Gastropoda) * Sowbugs and Pillbugs (Isopoda) * Mites (Acarina) o Spider Mites o False Spider Mites o Cyclamen Mite * Springtails (Collembola) * Psocids (Psocoptera) * Thrips (Thysanoptera) * Aphids (Aphididae) * Mealybugs (Pseudococcidae) * Scale Insects (Coccidae) and Diaspididae) * Whiteflies (Aleyrodidae) * Cutworms and Other Caterpillars * Fungus Gnats (Mycetophilidae) * Ants (Formicidae) Fungus Gnats (Mycetophilidae)Fungus gnats are small, slender, delicate, gray or dark-gray mosquito like insects, most species being 3 to 6 mm long, with a few as long as 12 mm or more. They are attracted to light, tend to gather at windows, and are nuisances in the home. The larvae are slender and whitish, with dark heads, and live in damp soil. Large quantities of decaying vegetable matter in damp soil are conducive to heavy infestations. The larvae often feed on the roots and crowns of plants, causing them to be stunted, discolored, and possibly defoliated (see chapter 10). They can be controlled by drenching the soil surface with a dilute chlordane emulsion prepared with the emulsifiable concentrate. Gnats flying about in a greenhouse can be killed with common household aerosols or dichlorvos resin strips." entomology.ucr.edu/ebeling/ebel11.html#scale%20insects**I then realized that just about EVERY insect that we have said we believe is involved in our Morgellons is considered to be a scale insect. Scale refers to size - small. There have been some parasitic wasps reported, which are also small in size.
ALSO... I have heard from others and reported myself about how my ear was covered with a thick 'biofilm' and it resembled fish scales, we see that certain ones produce this characteristic.
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Post by kammy on Dec 2, 2009 11:35:11 GMT -5
I believe the above post is very important to what is happening with some of us that have/are experiencing the insect part of this disease? That by understanding the biology of the insects we will be better to understand our disease. I PM'ed Jo to let her look at this post, hopefully, she has the time to look into this further and report back to us or incorporate this information into her research reports, (if she feels it's important) and is already aware of the possible role of scale insects and the science of coccidology in Morgellons?
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Post by kammy on Dec 2, 2009 18:50:27 GMT -5
I'm trying some Coconut Oil right now on my ears and eating some too, this article touts its benefits. It says we should eat 3 or 4 tablespoons a day, everyday... Has anyone else had good results with it? www.mnwelldir.org/docs/nutrition/coconut.htmI'm having a little bit of success with the coconut oil on my lesions, it's too early to tell but my ear lesion is breaking up into thousands of debris pieces inside of it, and breaking up some of the 'cones' - it's bringing the stuff out... I've been eating 4 tablespoons (or more) a day, also. I'm reading about how to lyse (kill) our spheres and wondered if anyone has tried this method below on a lesion, it's one of the ways to lyse cells?: "Freeze/Thaw The freeze/thaw method is commonly used to lyse bacterial and mammalian cells. The technique involves freezing a cell suspension in a dry ice/ethanol bath or freezer and then thawing the material at room temperature or 37°C. This method of lysis causes cells to swell and ultimately break as ice crystals form during the freezing process and then contract during thawing. Multiple cycles are necessary for efficient lysis, and the process can be quite lengthy. However, freeze/thaw has been shown to effectively release recombinant proteins located in the cytoplasm of bacteria and is recommended for the lysis of mammalian cells in some protocols." |
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Post by jeany on Dec 2, 2009 19:42:40 GMT -5
Good site about the benefits of Coconut Oil: www.organicfacts.net/organic-oils/organic-coconut-oil/health-benefits-of-coconut-oil.htmlHere a few excerpts: These benefits of coconut oil can be attributed to the presence of lauric acid, capric acid and caprylic acid, and its properties such as antimicrobial, antioxidant, antifungal, antibacterial, soothing, etc. The human body converts lauric acid into monolaurin which is claimed to help in dealing with viruses and bacteria causing diseases such as herpes, influenza, cytomegalovirus, and even HIV. It helps in fighting harmful bacteria such as listeria monocytogenes and heliobacter pylori, and harmful protozoa such as giardia lamblia. Healing: When applied on infections, it forms a chemical layer which protects the infected body part from external dust, air, fungi, bacteria and virus. Coconut oil is most effective on bruises as it speeds up the healing process by repairing damaged tissues. Infections: Coconut oil is very effective against a variety of infections due to its antifungal, antiviral, and antibacterial properties. According to the Coconut Research Center, coconut oil kills viruses that cause influenza, measles, hepatitis, herpes, SARS, etc. It also kills bacteria that cause ulcers, throat infections, urinary tract infections, pneumonia, and gonorrhea, etc. Coconut oil is also effective on fungi and yeast that cause candidiasis, ringworm, athlete's foot, thrush, diaper rash, etc. Jeany |
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Post by kammy on Dec 4, 2009 10:20:03 GMT -5
It makes sense that if we are seeing our "white fuzzy" fungus in all the petri dishes, and if you could see microscopically - you would also see the same spheres that I've been showing you all along, and I have showed you that they are in certain red wines, paper and cotton products, water, on the eggs of fungus gnats, within the bodies of fungus gnats, etc. That these spheres may all look alike initially but take on different characteristics and end up producing different types of fungi, and other 'things', they have different functions, reproduce differently and all seem to be a part of a system.
We might think that we're solely seeing fungi/mold because that is what is visibly evident in our petri dishes at first. If Morgellons was as simple as a fungal infection - don't you think we would have figured that out by now, or the CDC would have said something earlier? I'm hoping and praying that since the fungal aspect seems to be central around which the system operates that by getting the fungal aspect under control or stopping it with medicines - that, we can stop the entire system. In the event that this isn't true or if we do take medicines and then we become sick again because of contact with the product that is affecting our water, wine, food, environment, the insects, etc., that's why we're still here plugging along.
This sphere IS the Morgellons mystery, we believe we're seeing the actual patented product - this whole 3 ring circus system is a product that is intended to do this job with our crops or insect control, except it has somehow mistakenly got into our bodies. From the nature of it being in the water, it has naturally affected agriculture and because we're sick and poisoned - from its properties and intended uses, it appears to be a synthetic pesticide or insecticide.
We keep trying to define what it is we're seeing microscopically and looking for the scientific technical terms that fit what we're seeing and looking at the patents that contain all of the science that we are listing as we go along and comment on what we believe we're seeing. Eventually, we will have Morgellons pegged, if it is not described in the patents then we will know that it is from another source.
We're finding methods of operation that the agriculture/forestry/and other government sectors are using that could possibly be causing or adding to our disease. Our goal is to pinpoint what the product is and what is most likely causing this product to get into our systems so that we can bring attention to it. Do you think the USDA/EPA/NIH, or whatever branch is responsible to bring this to our attention, is going to do this on their own? Possibly, the CDC is reluctant to point a finger at whatever 'sister' branch is unaware or responsible and that's why they are so slow in acting on our disease, that Morgellons is a very 'political' issue. So, if you feel inspired to look by something we might say out here... then by all means, help us look!?
The more of us that are looking microscopically and reporting what we're seeing, the better.
It is easy to do a patent search, you just go to freepatents.com and type in the string of variables, such as, "pesticide baculovirus crystals sphere" and the patents that contain those words will come up.
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Post by kammy on Dec 4, 2009 11:16:45 GMT -5
Nevermind! We believe Jeany has found it!
She's preparing a report... stay tuned...
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Post by kammy on Dec 6, 2009 10:25:25 GMT -5
I was writing an email to someone who was concerned about the parasite part of our disease and this is what I wrote her and asked permission to make part of it public. I thought it might clear it up for some?
"Our parasites are not the 'normal' kind - they can't even be seen yet by traditional medicine. They are GM lab-created - they start out as granules, crystals, aeresol, powder, etc., there is a science where 'life' can be created from 'particles', it is being debated in the science world like the theory of evolution has been... is this possible? - well, molecular biology has made it come to life and Morgellons might prove that it is? I just wrote an article about it on my blog site at morgellons1.wordpress.com (and Jeany is morgellons2)...
Why they can't be 'seen' by traditional medicine is because the fields of science that have created them are not in sync with the medical field... in other words, entomologists, coccidiologist, molecular biology, nano science, forestry scientists, material science, food science, etc., are at work behind our disease and medicine hasn't caught up... the tools that molecular entomologists use to 'see' what's happening are not in use in the medical field, the tests they preform to analyze what's happening with plants and insects are not preformed on humans... yet.
We're in a 'crack zone' of science where one branch (medicine) hasn't caught up with the others.
We're not sure - other than... we have seen thru my experiments that the fungus gnat is capable of being inside a baculovirus capsule (the carbon polyhedron) and is possibly inside of us - however, 'they' (entomologists) claim that baculovirus are not capable of being inside of humans. Our Government owns part (or all?) of the patents on the baculovirus or had a hand in their invention... and they are being used in vaccines (not the insect version, though, I don't believe?) so... it's their 'baby' and the politics behind all of this... is immense! That's why they are dragging their heels... Morgellons crosses every branch of science and government."
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Post by kammy on Dec 6, 2009 11:54:11 GMT -5
This is something that I'm noticing and want to note, I'm not sure how it affects us?
In my childhood, and early adulthood - me and my brother both would have times when our hands and feet would sweat - sometimes profusely. (I think my mother was the same way - something we inherited?) It seemed to not be as great when I got into my mid-30's. It's not near as bad now in my 50's but sometimes... my hands or feet will perspire to where it's still very noticeable.
Anyway, since I'm moving - I have been uncovering objects around the house that I haven't touched in a while and I'm seeing a dry, white mold on a few things. Like for instance, a black leather jacket I had hanging on the wall on my 'coat rack hanger' near the front door to grab on my way out... and it was mostly on the left side of the jacket (it's my left ear that's producing)... naturally, I threw the jacket away. (And, two other black, leather jackets had some light white mold looking spots on them, also.)
What really got my attention yesterday was - I have a lot of percussion musical instruments that I collect from around the world and I have this 'bean' shaker from the Amazon, it's one big long brown bean that when you shake it - it makes a neat sound. Anyway, I pick it up and it has the white fungus smeared all over it, like... my hands were probably sweating when I played with it last and I think the fungus came out through my sweat?
Also, in a black leather office chair that I was sitting in when I was having my head outbreak, I was putting various liquids on my head and ears then... I can see where the liquid of whatever I used is mixed in with the white fungus and has left some vague, visible marks of the fungus/liquid mixture on the chair now. I know I wiped the chair down when I was having problems but haven't been using the chair in almost a year now... and haven't cleaned it thoroughly in some time.
The fungus in all these instances, the 'fungus' didn't appear to be "fuzzy" (to the naked eye) nor necessarily to even be growing... it is milky/watery-looking in color and in a 'dry' form.
We've heard Banny talk about how hers comes out of her hands in a clear gel form?
I believe from what I'm seeing that if we are infected enough, the fungal aspect can come out through our sweat, whether or not it is a pathogen to others is unknown. Jeany doesn't think it can be transmitted this way, air born spores - most likely, but not from someone touching something that we've touched and especially those that doesn't seem to be 'allergic' to the "M" pathogens probably wouldn't be affected? We don't know - just something to be aware of... (it seems to like leather).
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Post by kammy on Dec 6, 2009 12:09:11 GMT -5
Frito... how are you doing with your flu? Will you check in, please?
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Post by jeany on Dec 6, 2009 12:53:33 GMT -5
Jeany doesn't think it can be transmitted this way, air born spores, most likely, but not from someone touching something that we've touched and especially those that doesn't seem to be 'allergic' to the "M" pathogens probably wouldn't be affected? We don't know - just something to be aware of... What I was trying to say, is that I think it takes a specific combination in the body, such as a dysfunctional metabolism, mineral imbalance, hormone/insulin imbalance, and I still think, a genetic disorder we might all share, what makes a person susceptible to become infected by the Morg pathogens and at the end showing visible signs and symptoms. Of course inhaling fungal/mold spores isn't healthy for anybody, but this special combination, which also leads to a dysfunctional immune system, serotonin and dopamine might play a role too,... is IMO what causes an outbreak. I had a very rare form of ovary cancer a few years back..It was 000,1% out of all known ovary tumor species. Of course I researched all about this rare type and it was at the end caused by low serotinin and imbalanced estrogen/progesteron/testosterone levels. I'm following a theory and have done some studying on this subject what could cause susceptibility. I think that could be the reason why not everybody who gets in contact with us or in our enviroment, or by inhaling or ingesting gets infected. It would also explain why some family members do not become infected due to this missing combination and genetic similarity. Jeany |
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Post by fritolay66 on Dec 6, 2009 13:44:43 GMT -5
Checking in. Miserable, going back to bed......  |
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Post by jeany on Dec 6, 2009 13:59:51 GMT -5
Checking in. Miserable, going back to bed...... Hey Fritolini...sorry you're not feeling well..hope you get better soon, we need you here! Sending you a beary angel...might help a bit..??   |
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Post by fritolay66 on Dec 6, 2009 23:28:28 GMT -5
Ah thanks you guys. Jeany, I think your angeling is working. Feeling a little bit better. Not the someone beat me with a hammer feeling, now its just the steamroller feeling. Totally flattened.
Frito
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Post by kammy on Dec 19, 2009 16:41:47 GMT -5
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Post by kammy on Dec 27, 2009 13:16:29 GMT -5
Ok, it's time for us to get back to work.
I am already getting a little better with my ear lesion, I don't know why, exactly. The water is definitely better here and I don't have a film on my skin, hair and teeth now...
What are our objectives?
To be able to take information based on our collective research conclusions and information sharing to the German Lyme/Morgellons doctors/scientists - pointing them in directions as to where to look as the cause or source of Morgellons disease. Hopefully, based on this information - they will know more specific tests to run and which specialists to send us to? Of course, we will be sharing our findings as we go along.
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Jeany
