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Post by kammy on Apr 11, 2009 6:41:28 GMT -5
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Post by kammy on Apr 11, 2009 7:20:16 GMT -5
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Post by kammy on Apr 11, 2009 7:23:32 GMT -5
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Post by kammy on Apr 11, 2009 11:54:43 GMT -5
Pleospora Papaveracea I wasn't aware that Morgellons and "The Silent Superbug" are synonyms for each other, that they are appearing to be the same thing? There's only 2 YouTube videos and very limited information available. There's a website called silentsuperbug.com that I must have joined at one time. I tried to log in but they said I had been made inactive, so I wrote the webmaster to see if I can get back in. I might have to go into this site above to get some information. I notice that they have really good microscopic shots of various pathogens to compare what I've been seeing with what they are claiming the Super Bug is. Somehow, I ended up on a Silentsuperbug page, scrolling down the page I see our friend, the "Comets", the blue ones with the heads... @ silentsuperbug-reference.blogspot.com/This fungus is called: pleospora papaveracea www.dogpile.com/dogpile/ws/results/Web/pleospora%20papaveracea/1/417/TopNavigation/Relevance/iq=true/zoom=off/_iceUrlFlag=7?_IceUrl=trueen.wikipedia.org/wiki/Pleosporawww.sunshine-project.org/agentgreen/newpleosporapubs.html"The fungus Pleospora papaveracea and Nep1, a phytotoxic protein from Fusarium oxysporum, were evaluated for their biocontrol potential on opium poppy (Papaver somniferum). Four treatments consisting of a control, P. papaveracea conidia, Nep1 (5 µg/ml), and P. papaveracea conidia plus Nep1 (5 µg/ml) were used in detached-leaf and whole-plant studies. Conidia of P. papaveracea remained viable for 38 days when stored at 20 or 4¡C. Nep1 was stable in the presence of conidia for 38 days when stored at 4¡C or for 28 days at 20¡C. The presence of Nep1 did not affect conidia germination or appressoria formation. Nep1 was recovered from drops applied to opium poppy leaves in greenhouse and field studies 24 h after treatment. Opium poppy treated with the combination of Nep1 and P. papaveracea had higher necrosis ratings than the other treatments. There were changes in the intercellular protein profiles, determined by sodium dodecyl sulfate gel electrophoresis and silver staining, due to application of treatments; the most intense occurred in response to the combination of Nep1 and P. papaveracea. The combination of Nep1 and P. papaveracea enhanced the damage caused to opium poppy. Dendryphion penicillatum and Pleospora papaveracea were isolated from blighted Papaver somniferum and Papaver bracteatum plants grown in growth chambers and the field in Beltsville, MD. The etiology of the diseases was determined, and the fungi are being investigated as potential mycoherbicides to control the narcotic opium poppy plant. P. papaveracea is known to be a highly destructive seedborne pathogen of Papaver somniferum, causing seedling blight, leaf blight, crown rot, and capsule rot. Single conidia and ascospores were isolated and cultures established from naturally infested seed and diseased foliage and pods of opium poppy from Iran, Colombia, Venezuela, Sweden, India, and the United States (Maryland and Washington). Mycelia and conidia of P. papaveracea and D. penicillatum produced on necrotic leaf tissues appear morphologically similar, and the fungi were previously considered to be anamorph and teleomorph. However, no anamorph/teleomorph connection could be established, and the fungi appear to be distinct taxa. P. papaveracea produced conidia, mature pseudothecia, and chlamydospores in vitro and on infected stems. D. penicillatum produced conidia, microsclerotia, and macronematous conidiophores. Although both fungi were pathogenic to three poppy cultivars, conidial inoculum from P. papaveracea cultures was more virulent than conidial inoculum from D. penicillatum. Eight-week-old plants became necrotic and died 8 days after inoculation with a conidial suspension of P. papaveracea at 2 ¥ 10(^5) spores per ml. Disease severity was significantly enhanced by inoculum formulations that contained corn oil, by higher conidial inoculum concentrations, and by increased wetness periods. Symptoms on plants inoculated with either pathogen included leaf and stem necrosis, stem girdling, stunting, necrotic leaf spots, and foliar and pod blight. Inoculated seedlings exhibited wire stem, damping-off, and root rot. Conidia, and less frequently pseudothecia, of P. papaveracea and conidia of D. penicillatum were produced abundantly on inoculated, necrotic foliage, pods, and seedlings. Cultures from conidia or ascospores reisolated from these tissues consistently produced fungi whose morphologies were typical of the fungus from which the inoculum was derived. Two pathogenic fungi of opium poppy, Pleospora papaveracea and Dendryphion penicillatum, were isolated from field material in Beltsville, MD. The processes of infection by these two fungi were studied to determine the optimal environmental conditions for infection. Both fungi formed appressoria capable of penetrating directly through the plant epidermal layer. Of the two fungi, P. papaveracea was more aggressive, causing more rapid necrosis. " --------------------------- That figures, our Government in order to save us with their "War on Drugs" has sprayed the Opium Poppy with a fungus that is now running through our bodies! I think it's also known as "Agent Green"? *I need some of the fungus experts to look at this one. I believe this is the one I'm seeing? I'll get some more photos for comparisons.
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Post by kammy on Apr 11, 2009 15:17:59 GMT -5
TY, K... I think you understand my motivation for being here better than most? I haven't been able to get much treatment and I am sick. My experiences with the Medical Professionals - has been in my opinion, negligent.
I've been on the sites and studied the various treatments people are trying at home and tried a variety of things and nothing much has worked for me and I know there must be other people in my same situation? I think most people have had some treatment to 'knock back' a few of these pathogens and hopefully only have a few pathogens left to where they can function pretty well? That there's some pathogens that we all have in common that could or could not be addressed yet in some people.
I'm trying to identify as many pathogens as possible for people to look in that direction to see if that's possibly something that they or their doctor has missed. Without seeing a lot of samples yet, all I have to work with is a small handful to see what the variables in common are. As time goes by and more samples come in... then, we will get a better picture.
As I attempt to identify each pathogen, with help... I am attempting to make suggestions as to what might be helpful, such as this drug you've mentioned.
These are my goals - to identify as accurately as possible what's happened/happening. And, look and see what might help us have a better quality of health while waiting on our scientists/doctors to devise a protocol for us. I may miss the boat on some, however, hopefully, I'll get a few right. And, I need help in verifying that what I think might be a Morgellons pathogen - IS a Morgellons pathogen. That is where everyone else comes in - it's going to take a village to figure this out!
And, please folks, don't be afraid to stand up and say - 'I don't think you're on the right track there...', that's how we learn!
TY very much for you input, K.
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Post by kammy on Apr 11, 2009 15:20:24 GMT -5
Our Motto - "Bring No Harm" - we pretty much know that some fungal agent is involved! Itraconazole www.medicinenet.com/itraconazole/article.htmGENERIC NAME: itraconazole BRAND NAME: Sporanox DRUG CLASS AND MECHANISM: Itraconazole is an anti-fungal drug in the same class of drugs as fluconazole (Diflucan), ketoconazole (Nizoral), and miconazole (Micatin, Monistat). It prevents growth of several types of fungi by preventing the fungi from producing the membranes that surround the fungal cells. The FDA approved itraconazole in September 1992. PRESCRIPTION: Yes GENERIC AVAILABLE: Yes PREPARATIONS: Capsule: 100 mg; Oral Solution: 10 mg/ml STORAGE: Capsules should be stored at room temperature, 15-25 C (59-77 F) and protected from light and moisture. Solution should be stored below 25 C (77 C) but not frozen. PRESCRIBED FOR: Itraconazole is used for the treatment of fungal infections in both HIV- and non-HIV-infected individuals. It is active against fungal infections such as aspergillosis, blastomycosis, histoplasmosis, and candidiasis, as well as fungal infection localized to the toenails and fingernails (onychomycosis). It also is used for treating patients with fever and low white blood cell counts who are likely to develop a fungal infection.
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Post by kammy on Apr 11, 2009 16:02:38 GMT -5
THINKING... I'm thinking out loud here... using a metaphor - we know Morgellons is a jigsaw puzzle, with a lot of pieces... and we don't have the picture on the box to help us. And some of the pieces that should be the 'end pieces'... aren't even flat to let us know that they are 'end pieces'... I thinking to self - "How in the hail did you get all this? What did you do or who were you in contact with to get this?"... as I'm sure this is a universal thought, that we've all pondered at one time or another? So far, if anything is correct... I have Crypto, I. belli, Lung Flukes, strep, Agent Green, bacillus something or another... and the mystery one with the cotton fuzz carbon balls that makes fibers. And, I'm suspecting a couple of more... quite a bizarre cocktail for someone like myself who's lead a fairly normal and healthy life - not a risk taker, exercised, was in good physical condition, no health issues, long-term relationships, nutrition and supplement conscious, clean environments, etc., just the average, patriotic but skeptical - American. Obviously, I came into contact with these pathogens - I believe something happened around 1997, as I look back on my symptoms. Maybe, with each passing year another pathogen was added - as I went along? Or several at once? What do I instinctively feel? I feel that some major body burden was added within the year prior to the evident signs of Morgellons visibly manifesting itself. Opinions, anyone else, what do you think happened? And, then at the end, we can look at the pieces and try to put together a picture that is part or most of this puzzle? --------------------------- What is this Petri Dish experiment telling me? I flicked debris from my ear into the dish to reveal all this. hmmm... I'm under the impression that I only have 4 or 5 different kinds of specks coming out of my ear. Of course, I could be wrong, I haven't studied the debris as individual pieces, yet. I'm doing this experiment backwards, but that's ok, too - most scientists would start with the debris pieces, isolate each speck, culture them individually and see what they turned into in the dish. And, then possibly add two specks together to see what they did, etc. Well, if it's true that I only have 5 varieties of specks... then how come I'm seeing 16 different things? And, I knowingly only flicked a small number of specks into the dish... when I first looked - I had a big population of multi-colored 'quantum' dots... where did they come from? I already know that the particles coming out of my ear are small or nano in size, maybe that accounts for some it? I need to start collecting the specks for identification and isolation purposes and documenting that information here.
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Post by kammy on Apr 12, 2009 10:41:17 GMT -5
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Post by kammy on Apr 12, 2009 15:31:06 GMT -5
What a collective group of us that are involved in research have figured out is that there is a new generation of antibiotics coming out, and some are already available, that these should 'scramble' the quorum sensing abilities of most everything involved. That this disease is communicating between the parts. Jeany lead us to here, she's smart: Ok, it seems that there might be some available, that we need to stress to our doctors that we want a quorum sensing antibiotic. Most are in the development phase: quorum sensing antibiotics - Dogpile Web SearchThis article is interesting: Einstein researchers develop novel antibiotics that don't trigger resistance"Rather than killing Vibrio cholerae and E. coli 0157:H7, the researchers aimed to disrupt their ability to communicate via quorum sensing. Their target: A bacterial enzyme, MTAN, that is directly involved in synthesizing the autoinducers crucial to quorum sensing. Their plan: Design a substrate to which MTAN would bind much more tightly than to its natural substrate—so tightly, in fact, that the substrate analog permanently "locks up" MTAN and inhibits it from fueling quorum sensing."
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Post by jeany on Apr 12, 2009 15:47:50 GMT -5
Bonnie Bassler: Discovering bacteria's amazing communication system Bonnie Bassler on how bacteria communicate | Video on TED.com Bonne Bassler explains Quorum sensing. Bonnie Bassler discovered that bacteria "talk" to each other, using a chemical language that lets them coordinate defense and mount attacks. The find has stunning implications for medicine, industry -- and our understanding of ourselves. Another site with Bonnie Bassler: Princeton University - Department of Molecular Biology - Bonnie Bassler www.molbio.princeton.edu/index.php?option=content&task=view&id=27Watch the video -- interesting! Jeany
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Post by jeany on Apr 12, 2009 15:48:47 GMT -5
Hey Kammy, ....The scientists studying Morgellons have been looking at the by-products that our bodies are emitting... the fibers and other unusual anomolies...
That's what I always assumed. The fibers are only a byproduct of these organisms (bacterial-fungi) in our bodies and the body trying to get rid of it. Observing the fibers in the FIRST point IMO is irrelevant. I believe the scientists should focus on blood samples. Morgellons Disease is not a skin disease for my opinion...I believe it's a multi systemic disease with different bacteria and fungi. The different types of Morgellons are due to different locations and environmental circumstances and the way we got infected for I think there are several ways to become infected. But only ONE and ONLY true original vector. Surely it depends also in what physical condition one is due to an upload on toxins and /or other illnesses. That's why I think some people are more infected than others..in different stages..which would explain different species of this disease. There could be a similarity in our genes...the way our body fights pathogens too.
In order to kill these organisms it will take long term treatments on antibiotics and antifungal treatment TOGETHER. Bacteria feed on fungi and vice versa...kill one..you kill the other!
There could also be a parasite involved here but I think it's opportunistic and not the original vector..an enhancer maybe.
This is all speculation ofcourse..just my thoughts about this.
Jeany
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Post by jeany on Apr 12, 2009 15:49:46 GMT -5
Kammy, there is this german wikipedia site which is always a little different in information than the english one. Damn! I need to translate. ... Die ersten Organismen, in denen Quorum sensing beobachtet wurde, sind die komplexen Myxobakterien und Spezies aus der Gattung der Streptomyceten. Am bekanntesten ist jedoch die Biolumineszenz von Vibrio fischeri, eine Bakterienart, die als Symbiont in den leuchtenden Organen einer Sepiaart lebt. Freilebende Bakterien dieser Art erreichen nicht dieselbe Konzentration wie innerhalb dieser Organe, weshalb sie dort nicht leuchten. Streptococcus pneumoniae nutzt Quorum sensing, um Kompetenz zu erreichen. Beim dimorphen Pilz Candida albicans wirkt Farnesol als Quorum-sensing-Molekül. Es hemmt bei hoher Populationsdichte das Hyphenwachstum. It says...well don't expect too much..it's not easy for me, but I'll try. Here we go: The first organisms which has shown Quorum sensing are the complex Myxobacteria and species from Streptomyces. The most known bacteria is Vibrio fischeri (a grammnegative bacteria which lives in the OCEAN!) which has bioluminescence..a bacteria which lives as a symbiont in the glowing organs of a sepia. Free living bacteria cannot reach this high concentration like in the organs that's why they don't glow. Streptococcus pneumonae uses Quorum sensing to reach capacity. With the dimorph fungus Candida albicans Farnesol works as a quorum sensing molecule. It blocks the hyphae growth by high population. As you can see there are bacteria and marine organisms involved here together with fungi. The whole bunch! Biotechdaily - New Generation of Antibiotics Disrupts Quorum Sensing - Online bioresearch news biotechdaily.com/?option=com_article&Itemid=294722535&cat=Drug%20Discovery...cells are forming a biofilm – a colony of bacteria that contains resistant organisms and is involved in many antibiotic-resistant infections – they perform a function that enables them to leave a unique imprint on the world: their DNA. A small percentage of cells explode in a process called “lysis,” leaving behind a sticky residue that contains DNA and other cellular bioproducts which are then incorporated into the larger cell community to build a stronger biofilm. New Hope For Fighting Antibiotic Resistance www.sciencedaily.com/releases/2007/04/070426135314.htmThis disease has also something to do with anitibiotic overuse and antibiotic resistance I think. Antibiotic Compounds That Bacteria Can't Resist www.scientificblogging.com/alternate_allele/antibiotic_compounds_bacteria_cant_resistMethylthioadenosine nucleosidase Methylthioadenosine nucleosidase - Wikipedia, the free encyclopedia en.wikipedia.org/wiki/Methylthioadenosine_nucleosidaseEffects of antibiotics on quorum sensing in Pseudomonas aeruginosa. www.ncbi.nlm.nih.gov/pubmed/18644954Effects of antibiotics on quorum sensing in Pseudo...[Antimicrob Agents Chemother. 2008] - PubMed Result ....The effects of the three antibiotics administered at subinhibitory concentrations were investigated by use of DNA microarrays. Consistent results from the virulence factor assays, reverse transcription-PCR, and the DNA microarrays support the finding that AZM, CFT, and CPR decrease the expression of a range of QS-regulated virulence factors. ....Three of the antibiotics tested, AZM, ceftazidime (CFT), and ciprofloxacin (CPR), were very active in the assay and were further examined for their effects on QS-regulated virulence factor production in P. aeruginosa. Jeany
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Post by jeany on Apr 12, 2009 15:50:22 GMT -5
Kam, body temperature? That's why we should do exercise or go to a sauna...to heat up our temperature. Could be. Do we all get more or easier cold than other people? Oh, ya, now I know why some people accuse me to be "cold blooded"! No, just kidding! The "balls" and the fibers are IMO fungus..coated maybe with heavy metals which are "caught" in our tissues. This could explain the movements some people have seen due to body electricity. The fibers are loaded. Fungus and bacteria both feed on heavy metals. That's why I think DETOX is essential too..but BEFORE use of antibiotics and antifungals. I believe there is a highly resistent type of fungus such as Fusarium and/Or Candida species involved. Together with bacteria..such as Lyme or Chlamydia pneumone already existent in the body..it's DISNEY LAND!!
Jeany
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Post by jeany on Apr 12, 2009 16:54:23 GMT -5
....The first organisms which has shown Quorum sensing are the complex Myxobacteria and species from Streptomyces.... Streptomyces de.wikipedia.org/w/index.php?title=Datei:Streptomyces_sp_01.png&filetimestamp=20060418012831The Streptomycetes are members of the bacterial order Actinomycetales, bacteria which resemble fungi in their branching filamentous structure. However, they are true bacteria - prokaryotic cells - unlike eukaryotic fungal cells. As Actinomycetes grow, they form branching filaments of cells which become a network of strands called a mycelium, similar in appearance to the mycelium of some fungi. Actinomycetes are also unique in the way they form spores and in the production of numerous antibiotics. By far the most successful genus in this group is Streptomyces with over 500 species Actinomycetes are non-motile, filamentous, Gram-positive bacteria. Streptomyces species produce spores from aerial filaments called sporophores. These rise above the colony and form spores called conidia by simple cross-wall divisions of the filament. You can see the refractile spores on the video. Note that this process is distinct from the formation of bacterial endospores. Streptomyces species are found worldwide in soil and are important in soil ecology. Much of the characteristic earthy smell of soils arises from chemicals called geosmens given off by Streptomyces species. Streptomycetes are metabolically diverse and can "eat" almost anything, including sugars, alcohols, amino acids, organic acids, and aromatic compounds. This is achieved by producing extracellular hydrolytic enzymes. There is considerable interest in these organisms as agents for bioremediation. Streptomycetes are also of medical and industrial importance because they synthesize antibiotics. There are several theories to may explain antibiotic production, the most widely accepted one being that antibiotics help the organism compete with other organisms in the relatively nutrient-depleted environment of the soil by reducing competition. Over 50 different antibiotics have been isolated from Streptomycetes species, including streptomycin, neomycin, chloramphenicol and tetracyclines. There IS the relationship to Actinomycetes. Remember my picture? Many antibiotics are "made" out of natural Streptomycetes. No wonder some antibiotics don't help! In fact they're being fed and grow more rapidly. Jeany
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Post by jeany on Apr 12, 2009 16:56:49 GMT -5
Myxobacteria The myxobacteria are an interesting family of gliding bacteria that produce fruiting bodies in starvation conditions. They are common in animal dung and organic-rich soils of neutral or alkaline pH. Some of them grow by utilising cellulose, but many of them feed by secreting antibiotics to kill other bacteria and then produce enzymes to lyse the cells of their prey. The vegetative cells of all myxobacteria are aerobic, Gram-negative, elongated rods with either rounded or tapered ends They glide in water films across solid surfaces, secreting slime (polysaccharide) tracks in which many cells migrate to produce feathery extensions at the colony margin At the onset of nutrient depletion the cells migrate back along the slime tracks, aggregating by chemotaxis, to form large concentrations of cells. These aggregates then develop into fruiting bodies As the vegetative cells migrate upwards into the fruiting body they undergo a progressive differentiation into rounded myxospores Myxococcus species are easily cultivated on media containing a mixture of amino acids, which they use as the carbon and energy source; they do not utilise carbohydrates. Unlike some other myxobacteria, Myxococcus species produce simple fruiting bodies in which the stalk is composed of vegetative cells and slime. They also show a clear differentiation of the myxospores, which are spherical, refractile bodies. The myxospores are slightly heat-resistant, but not nearly as much as the endospores of Bacillus species. They resist UV irradiation and desiccation, surviving several years in dry conditions. de.wikipedia.org/w/index.php?title=Datei:Myxococcus_xanthus.png&filetimestamp=20061204191649Jeany
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Post by bannanny on Apr 12, 2009 22:44:17 GMT -5
About the chloracne... how would that relate to those of us who don't have lesions? I often wonder if there are 2 different strains of morgellons. Those with lesions and those without... those without seem to have more hair problems and electrical sensations. I dunno, but I do know whenever something comes up that has to do with lesions, I'm not able to relate to it at all.
What do I think about all of this? Well, I can't help but think it's the gel that's responsible for producing alot of what we experience. I think it's the crux of morgellons. I believe the "bubbles" we see are made up of the actual gel itself. A larger gel bubble will explode (so to speak) into many smaller bubbles which then begin to connect, resulting in a connect the dots type of networking. I think this networking then has the capability to permeate anything. I believe the gel can take on many different forms too. It can crystallize into pod forms and it can replicate and self assemble. I also believe the gel itself produces "energy particles." As the particles begin to group together via quorum sensing into their individual groups, the gel will seal them into a segmented tube form. I think the different colored segments incubate in this tube as they develop into the different colored fibers we see. Once developed, the gel releases the tube and each segment of fibers begins to unwind. I also think this gel will elongate, resulting in long transparent web-like strands which hold incredible adhesive properties... which is another form of networking that takes place outside of the body to continue its journey in the environment.
Anyway, I figure the gel probably uses whatever we have in our own DNA and goes from there... it could very well be the "magnet" that's pulling everything into our bodies from our environments too. If so, the fact that we're all in our own individual and different types of environments would give cause as to why we show certain differences from one another too.
I've been thinking the fungal aspect of this could very well be fusarium. Why? Becuz it comes up all too often, plus I had my house tested for mold and it was one of the more toxic of molds found. I think fusarium is probably a common toxic mold in most peoples homes as well. I've also given chaetomium alot of time on research and still think it could be involved (in my case anyway.) It was also one of the more toxic molds found in my home along with stachybotrys and asperguillis. I think morgs reacts with whatever a person comes into contact with once they've become infected by it. People live their lives in many similar ways, which would account for some of our common symptoms... it's reacting with the things we're all exposed to in everyday life. The differences we have from one another come into play becuz our environments are different. Does that make any sense at all?
love ya's ~~ bannanny
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Post by kammy on Apr 13, 2009 2:55:09 GMT -5
Bannanny -
I don't know. lol
Here's what I'm thinking without checking it out. We know that Morgellons is a 'cocktail' or 'stew'... with a variety of things in it? I believe it was conceived around the "Rainbow Agents" - I didn't look at any of the other colors except for the Orange and the Green, but, even these - not in any detail. I got enough information about the Orange to understand that dioxin is a 'base' - I don't know if dioxin is the base for the Green, or not? If it isn't, I can guess, that it's a 'cousin' or 'sister'... And, we saw how dioxin is in a lot of things that we unknowingly come into contact with? I find it interesting that they call them the "Rainbow Agents" and what color are the 'fibers', makes me wonder? But, like I said, I didn't research that in detail. And, we don't know for positively sure that dioxin is a component of Morgellons - it's just an odd coincidence that the Vietnam/Korean war guys have Morgellons and the VA hospital calls what they have "Agent Orange" or 'chloracne', to cover it up?
So, Bannanny, you basically just have the clear 'goo' that comes out of your hands? And, no lesions? Is that correct?
I said earlier that you are a very interesting 'specimen', in that I haven't heard of anyone else that emits a CLEAR 'goo', and that's all. I asked the others - if they had ever heard of anyone with just those symptoms. I find it interesting and fascinating that you are different than most. But, like I said, I'm the new kid on the block and what do I know? I don't any liquid-type 'goo' or biofilm lesions, either. I have read a lot threads with people describing their problems but, none like this.
As a youngster, my hands and feet would sweat quite a bit, to eliminate toxins, I guess, or because I was real active? But, as an adult, they don't sweat at all. I know we're not talking about your hands sweating here, but, my point is - the hands will eliminate toxins. One of things I noticed is that the Morgellons lesions don't easily grow on the palms of the hands. It's kinda' like you're a mystery inside a mystery.
Here's what I'll do. You prepare me a Petri Dish with some of your biofilm/goo in it and tape it shut - all the way around and put it in a couple of baggies and a padded envelope and mail it to me and let me study it... and I'll photograph it and let Jeany look at it... lol - and then she'll tell you what's what! lol - No, seriously, get the dish with agar... and then PM me when you do?
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Post by kammy on Apr 13, 2009 17:05:19 GMT -5
THE NEW QUORUM SENSING ANTIBIOTICS If you watch the Bonnie Bassler video in the post below, it says that ALL of these organisms (bacteria and fungus) are speaking AN INTERNATIONAL LANGUAGE.
That these new antibiotics disrupt the enzyme that's allowing them to speak to each other and amongst themselves. The enzyme that the bacteria has for communication is the same that the fungus has! It doesn't matter what the bacteria or fungus are - there's no need to identify them... really. They all have the same enzymes for communication - their key to living. This is amazing technology that's about to happen with this new antibiotic. AND, I BELIEVE, SOME ARE ALREADY ON THE MARKET.This information is in the post I left earlier.
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Post by jeany on Apr 13, 2009 17:26:49 GMT -5
Hey K-marie, english is my first language. I grew up in California. I speak both languages. Yes the actinomycetes are involved here...did you see my picture I've posted here? Jeany
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Post by jeany on Apr 13, 2009 17:29:53 GMT -5
From my other post: .......Three of the antibiotics tested, AZM, ceftazidime (CFT), and ciprofloxacin (CPR), were very active in the assay and were further examined for their effects on QS-regulated virulence factor productio... ceftazidime en.wikipedia.org/wiki/Ceftazidimeciprofloxacin en.wikipedia.org/wiki/CiprofloxacinAZM I'm not sure could be AZITHROMYCIN?? en.wikipedia.org/wiki/AzithromycinI have read that Lyme Bacteria and Chlaymydia pneumonae are both gramm negative. These quorum sensing antibiotics combat gramm negative bacterial infections. This could be the "clue" treating Lyme AND Morgellons Disease. I will talk about this with my Lyme Doc..wonder what he will say? And YES they are available...That's the GOOD news!! Jeany
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