Baculovirus is Identified as Morgellons

[jeany]

I'm picking up on Saponin again...

Saponins: Properties, Applications and Processing
www.redorbit.com/news/business/905822/saponins_properties_applications_and_processing/index.html

Saponins are a diverse group of compounds widely distributed in the plant kingdom, which are characterized by their structure containing a triterpene or steroid aglycone and one or more sugar chains.

While the main dietary sources of saponins are legumes (soybeans, chickpeas, mungbeans, peanuts, broad beans, kidney beans, lentils), they are also present in oats, allium species (leek, garlic), asparagus, tea, spinach, sugarbeet, and yam

Soap bark tree (Quillaja saponaria), fenugreek (Trigonella foenum-graceum), alfalfa (Medicago sativa), horse chestnut (Aesculus hippocastanum), licorice (Glycyrrhiza species such as Glycyrrhiza glabra), soapwort (Saponaria officinaux), Mojave yucca (Yucca schidigera), gypsophila genus (such as Gypsophila paniculata), sarsaparilla (Smilax regelii and other closely related species of Smilax genus) and ginseng (Panax genus) are the main non-food sources of saponins used in health and industrial applications.

The ability of saponins to swell and rupture erythrocytes causing a release of haemoglobin (the in vitro haemolytic activity) has been one of the most investigated properties of saponins.

* remember...G6PD - Gluthiaone - Iron overload - HeLa cells - Sickle Cell Anaemia?

The toxicity of saponins to insects (insecticidal activity), parasite worms (anthelmintic activity), molluscs (molluscicidal), and fish (piscidal activity) and their antifungal, antiviral, and antibacterial activity are well documented.

!!!The results of in vivo studies with rats, mice , and rabbits suggested that saponins are not absorbed in the alimentary channel but hydrolyzed to sapogenins by enzymatic action.

A study on the bioavailability of soyasaponins in humans showed that ingested soyasaponins had low absorbability in human intestinal cells and seem to be metabolized to soyasapogenol B by human intestinal microorganisms in vivo and excreted in the feces.

The safety of saponins of commonly used food and feedstuffs such as soybeans and alfalfa has been established by animal toxicology studies.

The safety of saponins (such as glycyrrhizic acid) or saponin-containing extracts (such as quillaja extracts) that are used as food additives has been the subject of thorough reviews.

Toxicological recommendations for glycyrrhizic acid are based on its effect of increasing mineralocorticoid activity, which in turn results in electrolyte imbalance due to sodium retention and potassium excretion, and water retention.

This effect though reversible can lead to elevated blood pressure if sustained.

Table 5 Lethality of quillaja saponins to CD-1 mice (Kensil and Marciani, 1991)

Saponins can impact the immune system through their adjuvant activity, their ability to improve effectiveness of orally administered vaccines by facilitating the absorption of large molecules, and their immunostimulatory effects.

Cholesterol-lowering activity of saponins...

Anticancer activity has been reported for a number of triterpene and steroid saponins including but not limited to soyasaponins.

Yucca and quillaja are classified as food additives in the US under section 172.50 (Natural Flavoring Substances and Natural Substances Used in Conjuction with Flavors) (US Food and Drug Administration, 2003).

Although quillaja and yucca are not considered Generally Recognized As Safe (GRAS) by the US Food and Drug Administration (FDA), they have been given GRAS designation by Flavor and Extract Manufacturers ' Association (FEMA) (FEMA #2973, and 3120 respectively).

There is a pending GRAS notice (GRN #165) received by FDA in 2005 from the American Beverage Association for quillaja extract (type 2) to be used as a foaming agent in semi-frozen carbonated and non-carbonated beverages at levels not exceeding 500 milligrams dry weight per kilogram beverage (US Food and Drug Administration, 2005a).

Saponins have been used as immunological adjuvants in veterinary vaccine formulations due to their immune enhancing properties since 1950s (Dalsgaard, 1974). Their use in human vaccines, however, has been limited by their complexity and toxicity.

The chemopreventive and chemother\apeutic activities of ginseng dammarane sapogenins have prompted the development of anticancer drugs which are at various stages of development

A new class of HIV drugs called Maturation Inhibitors (PA-457, in Phase 2 clinical trials) are being developed using betulinic acid derivatives.

Pharmaceutical compositions or plant extracts containing saponins have been patented for the prevention and/or treatment of a variety of conditions such as inflammation infection , alcoholism , pre- and post- menopausal symptoms , cardiovascular and cerebrovascular diseases such as coronary heart disease and hypertension, prophylaxis and dementia, ultraviolet damage including cataract, and carcinoma cutaneum, gastritis, gastric ulcer, and duodenal ulcer .

Jeany

[jeany]

Oleanolic Acid in Saponins

Oleanolic acid, one of the most common triterpene saponin aglycone, has also been reported to possess anti-viral (anti-HIV), anti- inflammatory, hepatoprotective, anti-ulcer, antibacterial, hypoglycaemic, anti-fertility, and anticariogenic activity, Anti-viral (anti-HIV), anticancer, antibacterial, antimalarial, anti-inflammatory, anthelmintic, and antioxidant properties have been demonstrated for betulinic acid and its derivatives.

www.redorbit.com/news/business/905822/saponins_properties_applications_and_processing/index.html

en.wikipedia.org/wiki/Oleanolic_acid

Oleanolic acid is a naturally occurring triterpenoid, widely distributed in food and medicinal plants, related to betulinic acid.

It can be found in Phytolacca americana (American pokeweed), and Syzygium spp, garlic, etc.

It is relatively non-toxic, antitumor, and hepatoprotective, as well as exhibiting antiviral properties.

List of phytochemicals in food

Saponins – soybeans, beans, other legumes, maize, alfalfa

Oleanolic acid - American pokeweed, honey mesquite, garlic, java apple, cloves, and many other Syzygium species.

Betulinic acid - Ber tree, white birch, tropical carnivorous plants Triphyophyllum peltatum and Ancistrocladus heyneanus, Diospyros leucomelas a member of the persimmon family, Tetracera boiviniana, the jambul (Syzygium formosanum), and many other Syzygium species.

Syzygium....Myrtaceae...Myrtle, clove, guava, feijoa, allspice, and eucalyptus .

Jeany

[jeany]

Eucalyptol
en.wikipedia.org/wiki/Eucalyptol

Eucalyptol comprises up to 90 percent of the essential oil of some species of the generic product Eucalyptus oil, hence the common name of the compound.

It is also found in camphor laurel, bay leaves, tea tree, mugwort, sweet basil, wormwood, rosemary, sage and other aromatic plant foliage.

Eucalyptol with a purity from 99.6 to 99.8 percent can be obtained in large quantities by fractional distillation of eucalyptus oil.

Although it can be used internally as a flavoring and medicine ingredient at very low doses, typical of many essential oils (volatile oils), eucalyptol is toxic if ingested at higher than normal doses.

Eucalyptol reduces inflammation and pain when applied topically.
It kills leukaemia cells in vitro.

Eucalyptol is used as an insecticide and insect repellent.

Eucalyptus essential oil is effective against airborne viruses and bacteria. Tests have proven that a spray containing 2% essential oil of Eucalyptus will kill 70% of staphylococci in the air. The oil can have a calming effect on the emotions. It will help clear the mind, assist in concentration and steady the nervous system. It will help people who are nervous.

www.ultimatewatermassage.com/oils/eucalyptus.htm

Eucalyptus used Internally
As a herbal supplement, eucalyptus is also used as:
an antibiotic
a diuretic - that is, it helps the kidney to expel water
as an anti-diabetic herb
an anti-tumor agent. The oils appear to have some anti-cancer properties.

Eucalyptus is known for a whole variety of health purposes, both when used as a herbal supplement, in a tea; and when its essential oil is used.

It relieves pain and and the aching of sore muscles, breaks up congestion in the lungs and bronchial passages, and reduces inflammation.

www.herbal-supplements.net/Eucalyptus_-_A_Herbal_Supplement_and_An_Oil.html

Uses based on tradition or theory

AIDS, alertness, antibacterial, antifungal, antimicrobial, antioxidant, antiviral, aromatherapy, astringent, athlete's foot, back pain, bronchitis, burns, cancer prevention, cancer treatment, chronic obstructive pulmonary disease (COPD), cleaning solvent, croup, deodorant, diabetes, diarrhea, dysentery, ear infections, emphysema, fever, flavoring, fragrance, herpes, hookworm, inflammation, inflammatory bowel disease, influenza, insect repellant, leukemia, liver protection, muscle/joint pain (applied to the skin), muscle spasm, nerve pain, onychomycosis (fungal infection), pain, parasitic infection, ringworm, runny nose, scabies, shingles, sinusitis, skin infections in children, snoring, stimulant, strains/sprains (applied to the skin), tuberculosis, urinary difficulties, urinary tract infection, whooping cough, wound healing.

!!!!Eucalyptus oil should be taken with caution, since small amounts of oil taken by mouth have resulted in severe and deadly reactions. For eucalyptus oil, doses of 0.05 to 0.2 milliliter or 0.3 to 0.6 gram daily have been used traditionally, but may cause toxic side effects. For infusions prepared with eucalyptus leaf, a quantity of 2 to 3 grams of eucalyptus leaf in 150 milliliters of water, three times a day, has been used traditionally, but may result in toxic side effects.

www.nlm.nih.gov/medlineplus/druginfo/natural/patient-eucalyptus.html

How to Use Essential Oils in Homemade Facial Masks - Video
www.livestrong.com/video/430-use-essential-oils-homemade-facial/

Organic Eukalyptus leaves, oil: www.mountainroseherbs.com/search/search.php?refine=y&keywords=Eucalyptus+globulus

In test tube studies, eucalyptus species have been shown to possess antibacterial actions against such organisms as Bacillus subtilis, as well as several strains of Streptococcus.

Eucalyptus oil (0.05-0.2 ml per day) can be taken internally by adults.11 It should always be diluted in warm water before consuming.

For eucalyptus leaf preparations, an infusion of 2-3 grams of the chopped leaves may be boiled in 150 ml of water and taken two times per day.

!!!!Eucalyptus oil needs to be used very cautiously since as little as 3.5 ml of the oil taken internally has proven fatal. It is best for people to discuss internal use with a qualified healthcare professional.

www.peacehealth.org/KBASE/cam/hn-2086009.htm

Jeany

[bannanny]

I just wanted to say I appreciate all the research you guys do even if I can't seem to follow it! You guys rock!

love you ~~ bannanny

[kammy]

TY, Banny, many may not realize that Jeany and I give this search a lot of time and effort? We could be doing other things with our time but we choose to do this - free and for the people.

I'm a little selfish in that I'm looking for something that will help me, nothing much has worked so far and by publishing what I find, it might help you, too? I'm always looking for cheap or natural remedies...

I came across this patent today and found it interesting:

"Systemic administration of NAC as an adjunct in the treatment of bioterror exposures such as anthrax, smallpox or radiation and for vaccination prophylaxis, and use in combination with DHEA for the treatment of smallpox and other viruses"

www.freepatentsonline.com/y2004/0022873.html

"The invention is for the combination and related methods of N-acetyl-cysteine oral, inhaled, or intravenous, or glutathione inhaled or intravenous, generally in combination with antibiotic and/or antiviral therapy to ameliorate the toxic effects of infection with materials used in Bioterror incidents such as Bacillus anthracis and smallpox virus, and alternatively, upon exposure to radiation, during testing, and vaccination, as treatment prior to treatment with antibiotic or antiviral therapy to ameliorate the toxic effects of infection and exposure with these organisms."

I think we've talked about the importance of NAC in many posts in helping with our symptoms, I'm seeing that they are also using DHEA and Selenium along with an antibiotic...

Maybe, we should look at DHEA and Selenium a little closer to see if we could add those supplements/foods for improvement?

[kammy]

DHEA

en.wikipedia.org/wiki/Dehydroepiandrosterone

"Studies have shown that DHEA is useful in patients with systemic lupus erythematosus. An application of the evidence was discussed by the U.S. Food and Drug Administration in 2001 and is available online.[6] This review also shows that cholesterol and other serum lipids decrease with the use of DHEA (mainly a decrease in HDL-cholesterol, the so-called "good cholesterol," and triglycerides can be expected in women,)"

**My HDL levels are low... is this saying it can make them even lower?

"Some in vitro studies have found DHEA to have an anti-proliferative or apoptotic effect on cancer cell lines.[18][19][20] The clinical significance of these findings, if any, is unknown. Higher levels of DHEA, in fact, have been correlated with an increased risk of developing breast cancer in both pre- and postmenopausal women.[21][22]

Regular exercise is known to increase DHEA production in the body.

In Canada, a prescription is required to buy DHEA."

**DHEA supplementation sounds controversial... I guess we need to exercise more to get it naturally? OH NOOOOO! lol

[kammy]

Selenium

en.wikipedia.org/wiki/Selenium

"Dietary selenium comes from nuts, cereals, meat, fish, and eggs. Brazil nuts are the richest ordinary dietary source (though this is soil-dependent, since the Brazil nut does not require high levels of the element for its own needs). In descending order of concentration, high levels are also found in kidney, tuna, crab, and lobster.[14][15]"

That makes twice I have seen Brazil nuts on a list as high in something we supposedly need... we need to eat more Brazil nutz!

(Oh, and when you purchase nuts, it's best to get them in the shell... in the shelling process (and who knows what they are using to shell them with?)... the oxidation process starts and the oils in the nuts can become quickly rancid.)

[jeany]

Jan 22, 2010 6:54:23 GMT -5 kammy said:

DHEA

en.wikipedia.org/wiki/Dehydroepiandrosterone

"Studies have shown that DHEA is useful in patients with systemic lupus erythematosus. An application of the evidence was discussed by the U.S. Food and Drug Administration in 2001 and is available online.[6] This review also shows that cholesterol and other serum lipids decrease with the use of DHEA (mainly a decrease in HDL-cholesterol, the so-called "good cholesterol," and triglycerides can be expected in women,)"

**My HDL levels are low... is this saying it can make them even lower?

"Some in vitro studies have found DHEA to have an anti-proliferative or apoptotic effect on cancer cell lines.[18][19][20] The clinical significance of these findings, if any, is unknown. Higher levels of DHEA, in fact, have been correlated with an increased risk of developing breast cancer in both pre- and postmenopausal women.[21][22]

Regular exercise is known to increase DHEA production in the body.

In Canada, a prescription is required to buy DHEA."

**DHEA supplementation sounds controversial... I guess we need to exercise more to get it naturally? OH NOOOOO! lol

Yes, Kam, DHEA can be in fact quite dangerous! I've read, it 'interferes' with sexual hormones, causing problems in the gonads esp. for men.

A natural source of DHEA is wild yam.

Jeany

[jeany]

Jan 20, 2010 21:09:33 GMT -5 bannanny said:

I just wanted to say I appreciate all the research you guys do even if I can't seem to follow it! You guys rock!

love you ~~ bannanny

[kammy]

Where are we today in our search for the cause of the Morgellons disease? I'm going to use the Baculo Thread as a 'central-central' place to work from, as I have stated, we're doubtful if the original product that was created that contained a baculoviral system is what has caused ALL of Morgellons to happen and we can't be positive until it is confirmed. However, from what we can obviously see from my photographs and those of others, Morgellons is mimicking or is in a baculoviral system of sorts.

We're seeing where one aspect can adapt another, fungi and bacteria go hand in hand. We're seeing where parasites and fungi can lead to cancer, we're seeing cancer diagnosed with some of our members. Some of us have already been diagnosed as having atypical cells or in a pre-cancer state. How does cancer relate to Morgellons Disease, we don't know yet, cancer numbers are on the rise - it could just be a coincidence?

It is obviously unlikely that one product or event has caused Morgellons Disease, it appears to be a combination of factors coming together in certain individuals that makes it evident. Of course, we are looking at all avenues to see what we can recognize in existing photographs as to what is Morgellons Disease in what I am calling a 'bacuolvirus-like system' because we have to have known scientific terminology in order to describe what is happening and the baculovirus system fits.

Any doctor can look at our basic blood work numbers to see that something isn't quite right, and needs further investigation? I think it's very, very odd that our doctors have taken the stance of dismissing Morgellons symptoms, calling us DOP and sending us home to deal with this ourselves, don't you? And it seemed as if they all had a secret meeting somewhere and said this "DOP diagnosis" would be the protocol in our treatment, why would they do this - because Morgellons appears to be a vast disease covering too much medical ground and to test everyone for what's going on would bankrupt the Insurance Companies? Probably... They are probably waiting on the procedures and lab testing protocol from above before they dig into what is wrong with us, it's as if they've been given silent orders to wait, an unspoken understanding they have of how the medical system works. In the meantime, they have abandoned their Hippocratic Oath and their duty to the people in the name of preserving their system, their job security, who's paying the bulk of their over-inflated salaries?

So, we have sections of this disease that are all encompassing - we're seeing many different fungal co-infections involved as has already been reported in the Morgellons.org case study and Morgellons can be a co-infection of Lymes Disease, making all of this very complicated. We are attempting to segment the sections and work on them in their appropriate thread topics and then pull back in here what we believe is involved in the total baculoviral or baculovirus-like, Morgellons system.

The greatest mystery IMO, is what is our sphere? If we could identify what the main Morgellons Sphere is - we could easily draw so many sound conclusions and be closer to solving this mystery. I am going to take this photo below and see if I can find some biology forums and see if someone can give me an educated guess as to what this cell is and see what kind of answers I get, this is from a human lesion experiment cultured in nutrient agar. I'm asking you also if you know any biology students/professionals - to ask them their opinion and let's see what kind of answers we get? (Of course, I'm not going to mention 'Morgellons' in my query.)



This photograph at 100x causes me to ask many questions. It looks like a red blood cell, if it is a red blood cell - it is a 'giant cell'... ok, that part is easy, all we just need a positive ID from someone who is knowledgeable in that field. My next question is - what's that 'speck' of stuff that's resting on the cell, these specks are seen in a lot of specimens and in various other places? (These specks are a large portion of what is coming out of my ear non-stop, I can do photo comparisons to show this and talk about this more in depth later.) I am suspecting that this is the Bacillus thuringiensis crystal/agrobacterium part of our disease but have not researched this aspect yet in depth, I will probably do this in the 'black and white speck', or 'what's happening with our food' thread. (If you are interested in the B.T./agro aspect, please start a thread, I will come join you or we can revive an older one.) I believe the photo is damaging, especially if this is found to be a giant red blood cell because it shows the direct relationship of how these crystal specks are or can interact with our cells.

I think we will be able to tie all of these various threads up into a logical sequence of events soon so that all is needed is further testing to verify or rule out what has happened to us and to give our doctors, scientists, labs more concise places to look in order to finally realize what our disease is and so that we can finally get the medical treatment we need and deserve. Please be patient as we are working with minimal means.

[kammy]

When looking at our mainstay crops that we consume daily on the black specks thread, when looking at wheat, I saw a plant fungus that only has very few microscopic photos to show what this fungus looks like microscopically, most are what it looks like on the plant leaf of other plants. I noticed that the similarities of what I've seen in a human lesion experiment are just too close to ignore.

It's obvious that whatever the Morgellons fungus is - is not easily seen with lab diagnostic testing because our results are all over the place, and even one, myself - showing no fungus. It seems that once we become 'Morgellons activated' we easily pick up other fungi to come along for the ride, probably due to low immunity. Some of us are showing plant fungi as our predominate fungus, I'm speculating that the Morgellons fungus is a plant fungus, it was more than likely introduced into our bodies by our food source. We can see that food scientists are adding fungi into our food processing, for insect control, fermentation, our livestock are eating them and for many varied reasons, I suspect - we need to be looking more in this direction?

I also did an experiment at one time by taking a healthy green leaf off a plant and touched it after touching my ear to see how toxic the stuff in my ear is. I didn't follow through with the experiment, I studied the leaf visually and noticed that it turned brown in the area where I had touched it, it had brown specks in the leaf such as this stock photo below. We have also seen where our cultured saliva grows the Morgellons fungus. There are no leaves here and no house plants to do an experiment on at this time of the winter, if any of you have an inclination to look - take a house plant leaf and spit on it or rub it with your lesion debris and see what the leaf looks like and post about it here? Most of the stock photos on plant fungi are of the leaves themselves and this could be why our fungus has alluded us?

(I also cultured organic bread before I left, I'll show photos of it shortly.)

Looking at plant diseases of wheat:

Septoria

en.wikipedia.org/wiki/Septoria

"Septoria are Ascomycete fungi that causes numerous leaf spot diseases on field crops, forages and many vegetables, and is responsible for yield losses.

Septoria leaf blotch is a fungal disease due to Septoria tritici, anamorph Mycosphaerella graminicola, that affects wheat and occasionally other grasses including barley. It is the major disease of wheat in the UK.

Septoria apiicola is the cause of late blight of celery. it is characterized by the production of conidia within pycnidia. The symptoms include chlorotic spots that turn brown and necrotic. Septoria apiicola can survive on seeds."

Septoria pycnidia

I'm microscopic images of cultured human Morgellons lesion samples to various stock photos of Septoria pycnidia plant disease.

vegetablemdonline.ppath.cornell.edu/PhotoPages/Cucurbit/Septoria/Septoria4.htm

Septoria Leaf and Fruit Spot of Cucurbits

vegetablemdonline.ppath.cornell.edu/factsheets/Cucurbit_Septoria.htm

Stock photos of Septoria pycnidia:

Notice how the lesions on the leaf look very similar in shape to Morgellons lesions:?

Stock Photo of Septoria on a Pumpkin

From a human Morgellons lesion, cultured in nutrient agar:

From a second human lesion experiment at 100x:

Septoria tritici

en.wikipedia.org/wiki/Septoria_tritici

"Septoria tritici is the causal agent of S. tritici leaf blotch, a foliar disease of wheat, and occasionally infects other grasses including barley. It is found in all wheat growing areas of the world[1] and is the major disease of wheat in the UK.[2] The teleomorph is Mycosphaerella graminicola (Fuckel) Schröter.

Morphology

Asexual state: Pycnidiospores are hyaline and threadlike and measure 1.7-3.4 x 39-86 μm, with 3 to 7 indistinct septations. Germiniation of pycnidiospores can be lateral or terminal. Cirrhi are milky white to buff. Sometimes in culture nonseptate, hyaline microspores, measuring 1-1.3 x 5-9 μm, occur outside pycnidia by yeastlike budding.[3]

Sexual state: Perithecia are subepidermal, globose, dark brown, and 68-114 μm in diameter. Asci measure 11-14 x 30-40 μm. Ascospores are hyaline, elliptical, and 2.5-4 x 9-16 μm, with two cells of unequal length.[3]"

Septoria tritici Blotch and Stagonospora nodorum Blotch

ohioline.osu.edu/ac-fact/0002.html

by Patrick E. Lipps and Dennis Mills
The Ohio State University

"Septoria and/or Stagonospora diseases can be found in nearly every wheat field in Ohio at some time during the growing season.

The Fungi Causing Leaf Blotch Diseases

Three different fungi cause blotch diseases: Stagonospora nodorum (Phaeosphaeria nodorum), Septoria tritici (perfect state Mycospharella graminicola) and Stagonospora avenae f. sp. triticea (Phaeosphaeria avenaria f. sp. triticea). Differences in spore shape and size separate species of Stagonospora from species of Septoria.

Stagonospora nodorum causes disease on leaves and glumes of the head, whereas Septoria tritici attacks leaves only.

ohioline.osu.edu/ac-fact/0002.html

"Symptoms of Septoria tritici Blotch

Wheat plants are susceptible to infection at any stage of development from seedlings to adult plants. Symptoms are usually detected on lower leaves in the fall and early spring, but as temperatures rise in late May, spread of Septoria tritici blotch decreases. Thus, Septoria tritici blotch is more common on lower leaves of plants than upper leaves. The initial symptoms are yellowish or chlorotic flecks usually on the lowermost leaves, especially those in contact with the soil. These flecks enlarge into irregular lesions, brown-to-reddish brown in color. As the lesions age, the centers become somewhat bleached with gray or ash-white centers. During this time, small, dark brown to black specks form in the center. These are pycnidia or spore producing bodies of the fungus. The presence of small, black pycnidia in lesions is the most reliable character for identifying the disease."

"Symptoms of Stagonospora nodorum Blotch

Symptoms usually appear within two or three weeks of head emergence. Leaf lesions begin as very dark brown flecks or spots, sometimes with a yellow halo. These small irregular lesions expand into oval light brown lesions with dark brown centers."

[kammy]

Is it possible for humans to contract Septoria tritici or a plant fungus disease?

student.biology.arizona.edu/honors2007/group03/thepaper.pdf

"New diseases of humans, animals and plants emerge regularly. Enhanced virulence on a new host can be facilitated by the acquisition of novel virulence factors. Interspecific gene transfer is known to be a source of such virulence factors in bacterial pathogens (often manifested as pathogenicity islands in the recipient organism1) and it has been speculated that interspecific transfer of virulence factors may occur in fungal pathogens2."

P. tritici-repentis

"Transfer of ToxA from S. nodorum to P. tritici-repentis In comparison to Stagonospora nodorum blotch caused by S. nodorum, tan-spot of wheat caused by P. tritici-repentis is a recently recognized disease. P. tritici-repentis formerly known as Helminthosporium tritici-vulgaris) was first identified in 1902 from grass species and in 1928 from wheat, but was described as a saprophyte or occasional pathogen17. It was not until 1941 that typical necrotic tan spot symptoms were first described18,19. Following these US reports, these symptoms were next noted in Australia and Africa in the early 1950s20,21. It is significant that tan spot was initially called yellow spot in both the United States and Australia, a presumably accurate description of mild chlorotic disease symptoms. The 1942 reports describe leaf spots with light brown centers and yellow borders, a symptom perfectly in line with a current infection involving ToxA-induced necrosis. These early reports may have captured the first emergence of a new form of P. tritici-repentis, containing interspecifically transferred ToxA. In contrast, S. nodorum has been recognized as an abundant, regular and serious wheat pathogen in many parts of the world since at least 1889 (ref. 22). ToxA is found in only 24% of S. nodorum isolates and 80% of P. tritici-repentis isolates (this study and refs. 12,13).

Currently, epidemics of both pathogens occur annually, and mixed infection on the same leaf is common. It is therefore likely that the two fungi regularly come into close contact. It is possible that conidial anastomosis tubes (CATs) form between these species, facilitating genetic exchange26. We have observed CAT formation in S. nodorum, and CATs have been observed in P. tritici-repentis (Lynda Ciuffetti, personal communication). The presence of a large region of almost identical DNA in both species, the presence on that region of a critical host range–enhancing toxin gene and its absence in related species is consistent with interspecific gene transfer. The pattern of high sequence diversity in S. nodorum and monomorphism in P. tritici-repentis and the recent emergence of the tan spot symptom lead us to suggest that the interspecific transfer of ToxA was from the former to the latter and occurred some time shortly before 1941. It remains possible that ToxA was transferred to P. tritici-repentis from a third, as yet undiscovered fungal (or non-fungal) species, but we have been unable to identify other possible donors. It is likely that ToxA-expressing P. tritici-repentis was globally distributed in shipments of infected grain, leading to widespread dissemination and setting the stage for the emergence of new highly pathogenic, tan spot–causing populations of P. triticirepentis in wheat fields worldwide."

We've mentioned the genetic factor as being one reason why some of us are Morgellons evident and others are not. Are we "turning into plants" as Stricker has stated?

Whether a plant gets blotch/spot disease is based on the genetics of the plant:

www.k-state.edu/wgrc/Publications/1999/6923.pdf

"Two major genes in wheat are responsible for resistance to tan spot (13,14)."

[kammy]

I've heard it expressed and expressed it myself that we're becoming more leery of eating the food. WELL... we have to eat something! After posting the article above about the wheat, I went and made a sandwich... I looked a little closer at the bread this time.... lol

We have to eat bread and grains and try to keep a healthy diet especially, since we're sick. The way I look at it, since I'm not getting noticeably better, is that I'm sick anyway, what difference does it make? lol Until I get the right medicines or treatment, I'm going to carry on like normal.

Until we know more, just use caution and as Toni is so observantly pointing out - look before you eat...!! We might to avoid foods with 'black specks' in them... especially those that aren't supposed to have spices in the ingredients! We have to use our common sense here and do the best we can.

[kammy]

Upon inquiring from what might be considered the world's leading expert on Dictyostelium, they pointed out that I could not possibly be seeing a baculoviral capsid from which an amobea was emerging due to the size of known baculovirus not being able to be seen with light microscopy.



"Dear xxxxxxx

Thought I should clarify my answer a little. It certainly can't be a baculoviral capsid you saw as they are way too small for you to see under a light microscope. It might have been a Dictyostelium (or another slime mould species) spore from which a slime mould amoeba emerged, but the image was too unclear to tell. This is possible but unlikely.

Regards
xxx xxxxx"

I have shown photos at 100x of what are believed to be "Giant Cells", I have shown at 100x what is appearing to act in the manner of a baculoviral system... and yet this is not possible due to the known size - this might lead us to believe that we are dealing with a mutation that has blown 'artifacts' and possibly our own blood cells up in size? Or, we're dealing with a whole new scientific entity which is mimicking some of the baculoviral characteristics that's visible microscopically on a "giant" scale? Very curious.

This unusual, too large, too small size variation has already been noted by Carnicom:

www.carnicom.com/blood1.htm

"A second observation is that more variation of size (not form, however) will occur than within human samples observed. This appears to be a result of the chemical environment that allows this reconstitution process to take place. The cells will change in size during observation on the microscope stage, and some of them will reach abnormally large diameters estimated up to approximately 20-25 microns. In addition, some of the cells will reconstitute to a smaller diameter than a human cell, down to a level of approximately 4 microns in diameter. The average size of the cells appears to concide closely with that of the human species, on the order of 6-8 microns in diameter."

[kammy]

While on Carnicom's site, I noticed he had 4 new papers out that were very interesting to read, we thank Cliff for his ongoing, and hard work:

www.carnicom.com/

[kammy]

I was up late last night reading Carnicom's site papers... noting some of the highlights below that possibly can help us, (neither Carnicom nor I are medical doctors qualified to give medical nor nutritional advice):

In his, "MORGELLONS : pH, CONDUCTIVITY, IONS & LIVE ANALYSIS, Jan 10 2010"

www.carnicom.com/ph6.htm

Carnicom mentions that the culture thrives in an acidic environment. We probably need to PH up?

He mentions that the filament (fungal) hyphae start with white to green to black. I mentioned that I was seeing white, black and then green/gray... we are close in that we are seeing three colors of hyphae and the colors are approximately the same. The various color of hyphae are all collectively believed to be the one Morgellons fungus, he mentions the age determines the color, I mentioned where it is in its life cycle when it enters the Petri Dish determines the color (another factor of age), that it can look different at different times, in which I showed the actual Petri Dishes from my experiments and the explanation about this multi-colored aspect on the "Kammy's Request" thread.

Carnicom is listing 4 ingredients as the Morgellons artifacts: "These are the encasing filament, the chlamydia-like organism, the mycoplasma-like (pleomorphic) organism and under certain condtions, the erythrocytic (red blood cell) form."

We need to look at closer about what might be happening in what he's noting by the Chlorine ion increasing and the Iron ion decreasing with the age of the culture. It is noted that the "chlamydia-like organism" uses iron as a nourishment source, this organism is suspected positively present by him testing using a dye on another of his pages.

I remember in my early research that in a group of Morgellons patients tested that 100% or a large number tested positive for chlamydia pneumonia, that this is likely a given part of Morgellons disease? Jeany tested positive for C. pneumonia when her blood analysis was initially tested and was on antibiotics for 10 weeks and 1 week intravenously to kill all the life stages. Here is a link showing Dr. Harvey's work in relation to Morgellons and c. pneumonia:

"Dr. Harvey's 2004 PHYSICIAN PROTOCOL BORRELIA-ASSOCIATED ILLNESSES CURRENT DIAGNOSIS AND TREATMENT

The role of Chlamydia Pneumoniae (what Dr. Harvey calls "chlamydophila pneumoniae" above) in Morgellons disease."

(link not good)

Information on Chlamydia Pneumoniae treatment: cpnhelp.org

C. pneumonia is a blood test that we should ask our doctors for from the indications. Keep in mind that C. pneumonia can be of the skin causing ulcers - our lesions can also be tested for this pathogen. Mycoplama should be tested for also according to his "A MECHANISM OF BLOOD DAMAGE" report.

Carnicom is stating that the immune system is a critical factor in how quickly the organisms involved can change, we obviously need to boost our immune systems.

[kammy]

While looking for Dr. Harvey's work above, I found this interesting article for those who have asked about the 41 kda band that most/all? are seeing in their Morgellons or Lymes testing Western Blot analysis:

www.springboard4health.com/notebook/health_lyme_disease.html

"My Lab Results Would Have Been Considered Negative

After testing, I only had one band - number 41, which is the “flagellin” (or tail) of the spirochete, specific for Borrelia bacteria (Lyme), so I would have been told that I was negative."

[kammy]

Still looking for Dr. Harvey's work, I found this interesting about C. pneumoniae and cancer:

www3.interscience.wiley.com/journal/118849930/abstract?CRETRY=1&SRETRY=0

"ABSTRACT

Recently, Chlamydia pneumoniae-specific DNA and antigens were reported in the skin of patients with Mycosis fungoides (MF), the most common form of cutaneous T-cell lymphomas. In order to revalidate these data we analyzed skin sections of patients with MF for the expression of three different chlamydial antigens and C. pneumoniae DNA by immunohistochemistry and PCR according to previously described protocolls. Neither C. pneumoniae-specific DNA sequences nor antigens were detected in any of the skin biopses from 24 MF patients tested, suggesting that further studies are needed to establish any pathogenetic relevance of C. pneumoniae in MF."

Transmission of C. Pneumonaie

www.jstor.org/pss/30113160

[jeany]

Jan 27, 2010 16:19:50 GMT -5 kammy said:

Still looking for Dr. Harvey work, I found this interesting about C. pneumoniae and cancer:

www3.interscience.wiley.com/journal/118849930/abstract?CRETRY=1&SRETRY=0

"ABSTRACT

Recently, Chlamydia pneumoniae-specific DNA and antigens were reported in the skin of patients with Mycosis fungoides (MF), the most common form of cutaneous T-cell lymphomas. In order to revalidate these data we analyzed skin sections of patients with MF for the expression of three different chlamydial antigens and C. pneumoniae DNA by immunohistochemistry and PCR according to previously described protocolls. Neither C. pneumoniae-specific DNA sequences nor antigens were detected in any of the skin biopses from 24 MF patients tested, suggesting that further studies are needed to establish any pathogenetic relevance of C. pneumoniae in MF."

aha...this is interesting:

Mycosis fungoides (also known as Alibert-Bazin syndrome or granuloma fungoides), is the most common form of cutaneous T-cell lymphoma. It generally affects the skin, but may progress internally over time.

The name mycosis fungoides is somewhat misleading--it loosely means "mushroom-like fungal disease".

The disease, however, is not a fungal infection but rather a type of non-Hodgkin's lymphoma.

It was so named because Alibert described the skin tumors of a severe case as having a mushroom-like appearance.

Typical visible symptoms include rashlike patches, tumors, or lesions. Itching (pruritus) is common, perhaps in 20% of patients, and is not universal.

Diagnosis is sometimes difficult because the early phases of the disease often resemble eczema or even psoriasis.

Common treatments include simple sunlight, ultraviolet light, topical steroids, topical and systemic chemotherapies, local superficial radiotherapy, the histone deacetylase inhibitor vorinostat, total skin electron beam radiation, and biological therapies (e.g. interferons, retinoids, rexinoids).
Treatments are often used in combination.

en.wikipedia.org/wiki/Mycosis_fungoides

[jeany]

Jan 27, 2010 16:15:15 GMT -5 kammy said:

While looking for Dr. Harvey's work above, I found this interesting article for those who have asked about the 41 kda band that most/all? are seeing in their Morgellons or Lymes testing Western Blot analysis:

www.springboard4health.com/notebook/health_lyme_disease.html

"My Lab Results Would Have Been Considered Negative

After testing, I only had one band - number 41, which is the “flagellin” (or tail) of the spirochete, specific for Borrelia bacteria (Lyme), so I would have been told that I was negative."

From the link above:

First, the arthropod is not the exclusive vector of Lyme disease. In addition to ticks, Bb may be carried and transmitted by fleas, mosquitos, and mites.

Second, Lyme disease is not exclusively vector-borne.

Compelling evidence supports horizontal (sexual) and vertical (congenital) human-to-human transfer.

University of Wisconsin researchers state that dairy cattle and other food animals can be infected with B. burgdorferi and hence some raw foods of animal origin might be contaminated with the pathogen.

Recent findings indicate that the pathogen may be transmitted orally to laboratory animals, without an arthropod vector.

Thus, the possibility exists that Lyme disease can be a food infection.

.. evidence that the transmission of Bb is possible by blood transfusion.

People from Asia who come to me with the classic Lyme rash have been infected by fleas and gnats."