Baculovirus is Identified as Morgellons

[Jill]

Kammy and all,

I believe that Lyme and Babesia tests would be 1st priority- I don't know how or where you could get a test for T parva- likely that the powers that be would not want it to be known that pathogen is loose in the states and elsewhere.

From what I've read, I believe the source of the T parva and the human connection is via- Theileria parva p67 antigen - vaccinne given to cattle and animals (dogs).
Could be via milk and/or meat?

tinyurl.com/lg78jw

The vaccine is the source of the baculovirus as well- it is used in the process of making the vaccine.

As I posted- there is also an insect connection to all of the above.

As to testing:

IGeneX, Inc.
797 San Antonio Rd., Palo Alto, CA 94303 USA
Tel. [phone] Fax. [phone]
www.igenex.com/

www.lymesite.com/reliable_testing.htm

Lyme, Babesia, Ehrlichia

Harvey and other doctors suggest Bowen Labs

Laboratory testing for Lyme disease is under continuing development, ... research involvement in pediatric hematology / oncology and Jo Anne Whittaker, ... The accuracy of this method was tested in two other laboratories with identical results. ... For Q-RIBb test info contact the Bowen Institute at: 727-937-9077; ...

(not sure if they are still in business?)


Ideas, Kmarie?

Jill

[Jill]

Sept 5, 2009 11:13:48 GMT -5 kammy said:

Where should this thread go from here?


We have a scientist that's coming in here shortly to look at this, they've already been down 25 different paths as to where to look, I'm sure they're already overwhelmed? What do we want them to concentrate on when they start their research?

Per above- T Parva -# 1 priority if Scientists are looking at this

Next- the other components found by Robert Smith that tie to
the remediation products for waste water/sludge and VOCS - primarily MTBE/TBA and BTEX-

Those would be :

Azoarcus_sp_EbN1

Ralstonia_solanacearum_BMI1000 Polynucleobacter_
Chromobacterium violaceum ATCC 12472,
Azoarcus_sp_BH72Dechloromonas_aromatica_RCB


All of which are found in PM 1 which is a remediation product for
MTBE- along with Agrobacterium Ti C 58

(see my links at MDR - on Robert Smith & MTBE)


All the above and more was ID'd by Robert Smith in a Morgellons sufferers specimens- the source would most likely be water.

[kammy]

Sept 5, 2009 12:37:35 GMT -5 felixwillford said:

Sept 5, 2009 12:03:48 GMT -5 kammy said:

I wonder if Lymes looks microscopically similar to Morgellons in any way, in other words, if we both have a baculovirus system happening within our diseases?

[/size] [/quote]

K,

Just thowing this out there ........ I have been reading the book LAB 257 that Becky McClain suggested. ( why would she suggest it if she didn't KNOW that it was TRUE about how they "play" with microbes and make BIG mistakes that get loose)

I believe we acquired this thru insects. Suebe once told me that her doctor told her the orgin would be from rodents? (FLEAS) Correct me if I am wrong Suebe!

I certainly have NO CLUE, but we do KNOW that the LYME/Borrelia Spirochete HAS definately been "manipulated". In Becky's video, she mentioned making microbe "mixes" with different viruses. (i.e. very easily could have been something like baculovirus, and the T. Parva) .... So it could be VERY possible that the reason some/most are testing positive for Lyme/borrelia is because of we have part of it.

The GFP is used for tracking, if they threw "Lyme/borrelia spirochete out there with and ticks / insects / spiders / gnats / flies / mosquitoes / mites / scabies and all FLEAS acquired "this new spirochete".... these INSECTS would pass the disease along with the GFP used for tracking would the GFP WOULD be found in us. (ROBERT SMITH)

I've been away for a while so I am not up to date on all that you have been researching...... (but I will catch up). I have more reading to do on the baculovirus.

[/quote]

Hi K... must be nice sitting around reading a book?... I'm just teasing with you... I haven't read a book in I don't know when... if it's not out here on the Internet...

I haven't followed Robert Smith's work lately, I left off some time back with his analysis of the fibers... I'm hear he's making some good progress and all the researchers that are on the right track, should tie-in eventually. We can build on what we've found by looking at what they have verified, to build a stronger case for what Morgellons is? Maybe you or Jill has a link to Smith's work?

I've stated I'm almost Lymes illiterate, I've been concentrating in one place, does anyone know what form this borrelia spirochete starts out as, just wondering? I think it's very important to pay attention to Lymes, even if we think it doesn't concern us... from the new way insects are operating in nature, any insect disease needs to be of concern.

For those of us who don't know, what's the GFP, K or Jill?

[kammy]

Sept 5, 2009 13:09:28 GMT -5 Jill said:

From what I've read, I believe the source of the T parva and the human connection is via- Theileria parva p67 antigen - vaccinne given to cattle and animals (dogs).
Could be via milk and/or meat?

tinyurl.com/lg78jw

The vaccine is the source of the baculovirus as well- it is used in the process of making the vaccine.

Jill

vaccines again? We know they give the animals vaccines, have we looked at what role baculovirus have in animal vaccines, such as in the animals we eat? And, which ones they are?

We just saw Carnicom cook his (what I'm saying) is a baculovirus sphere up to boiling point... and might still have a viable cell? Would cooking our meat get rid of these baculovirus? If we can pass them from human to human under certain circumstances, are the farm animals passing them amongst each other, or are GM insects infecting the farm animals, too?

[Jill]

T Parva is vectored via a tick bite.

In the US- I believe that, for the most part, our animals have the
vaccine which I posted a few posts ago. My guess is that we get the T parva from milk and/or meat - IOW, from animals that have been vaccinated for T parva. Possible- insect bite- vector?

That would account for the baculovirus content and as Kmarie mentions, the GFP- Green fluorescent protein. They add that to
recombinants.

Robert Smith links:

www.thenmo.org/rSmith02.htm

www.thenmo.org/rSmith01.htm

[kammy]

Sept 5, 2009 13:17:24 GMT -5 Jill said:

Sept 5, 2009 11:13:48 GMT -5 kammy said:

Where should this thread go from here?


We have a scientist that's coming in here shortly to look at this, they've already been down 25 different paths as to where to look, I'm sure they're already overwhelmed? What do we want them to concentrate on when they start their research?

Per above- T Parva -# 1 priority if Scientists are looking at this

Next- the other components found by Robert Smith that tie to
the remediation products for waste water/sludge and VOCS - primarily MTBE/TBA and BTEX-

Those would be :

Azoarcus_sp_EbN1

Ralstonia_solanacearum_BMI1000 Polynucleobacter_
Chromobacterium violaceum ATCC 12472,
Azoarcus_sp_BH72Dechloromonas_aromatica_RCB


All of which are found in PM 1 which is a remediation product for
MTBE- along with Agrobacterium Ti C 58

(see my links at MDR - on Robert Smith & MTBE)


All the above and more was ID'd by Robert Smith in a Morgellons sufferers specimens- the source would most likely be water.

" Per above- T Parva -# 1 priority if Scientists are looking at this[/b][/size]
[/color]
Next- the other components found by Robert Smith that tie to
the remediation products for waste water/sludge and VOCS - primarily MTBE/TBA and BTEX-"...

T Parva is probably in the baculovirus mix, it's evidently shown as harden, callused, glue cones, within our lesions, correct me, if I'm wrong? Yes, we can say it's there, it's a small piece in a big puzzle. Is T Parva touching the water, moving around, landing on your food... the thing that is spreading our disease and keeps re-infecting us? No.

T Parva is probably evident in some of my photographs of some of these 'unknowns', I just don't know what I'm looking at yet. But, I can tell you that there is no ONE particular virus/bacteria/fungi/parasite within the main BV Sphere System that if it is removed is going to completely stop the system!

We might be able to eliminate it if we attack enough of these main pathogens at one time, we have seen that people can become somewhat healthy and symptom free with this disease.

If we have something that's in perpetual motion that's moving around the world, a never-ending do-loop in nature... we've got to figure out how to stop IT and eliminate IT from our bodies?

Our water is probably, already contaminated with this in most places? Are VOCS, MTBE/TBA and BTEX-... flying around, self perpetuating themselves, spreading Morgellons? No.

So, are they really priorities, afterall? I wouldn't want our only chance of a scientist looking off in 5 different directions as priorities while the "Silent One" is moving by the second to bring continued and further illness? And, our time possibly running out?...

Not to say, that all of these pathogens mentioned are not important to eliminate, yes, they are. The more we can eliminate - the better we will feel.

[Jill]

Sept 5, 2009 11:13:48 GMT -5 kammy said:

How long is it going to take a scientist to find the cancer part of the baculovirus system, acquire the demographics of how many of us and how quickly cancer is developing inside our disease,

Kammy,

The cancer aspect primarily stems (no pun) to the T parva and the blood issue, IMO, rather than the Baculovirus.

Real quick- a few thoughts:
As to T parva- and the spread of same- via Rhipicephalus appendiculatus tick:

Huge problem in Africa, incurring over 170 million a year in direct losses not to include indirect losses such as acaricides for tick control, chemotherapy and more.

www.library.wur.nl/wda/dissertations/dis3458.pdf

TICKS AND TICK-BORNE DISEASES (T & TBDs) IN AFRICA: Economic importance, control problems with acaricides and new hope based on use of Neem compounds.

Global losses: Global losses due to T & TBDs have been estimated at US$ 7 billion annually and approximately 80% of the world's cattle population of 1.2 billion are at risk from T & TBDs *** (McCosker, 1979). Unfortunately, over half of this occurs in Africa.

***((Ticks and Tick Bourne diseases))

80 % of the world's cattle population at risk



www.neemfoundation.org/neem-articles/neem-updates/vol-1v-no1/keeping-up-with-neem.html

Rhipicephalus appendiculatus was RE-DISTRIBUTED to 3 other Continents: America, Asia, Australia and therefore this tick would/could also be a vector to those countries as well.

The T parva alters- hijacks- the blood cells.



www.tigr.org/tdb/e2k1/tpa1/intro.shtml

Fair use

Excerpt:

The infected lymphocytes grow larger and begin to divide. As each enlarged lymphocyte, called a lymphoblast, divides, the schizont inside the*** lymphoblast also divides, ensuring infection of each of the two daughter cells produced by the dividing lymphoblast. The infected lymphocytes expand rapidly and spread throughout the lymphoid system of the animal,
giving rise to widespread destruction of hostcells.

Some of the schizonts develop into merozoite forms, which are released from the lymphocytes into the bloodstream, where they invade red blood cells (erythrocytes).

In the red cells, the parasites develop into forms called piroplasms, which are able to infect ticks. As ticks feed on animals infected with the parasite, they ingest red blood cells containing the piroplasms. Once in the tick gut, the parasites differentiate into male and female gamonts, which fuse to form zygotes. The zygotes differentiate into kinetes, which move to the salivary gland and enter a particular cell type. Here the parasites form sporoblasts, each of which give rise to 30,000 to 50,000 sporozoites, a parasite form able to infect animals. The sporozoites are introduced into a mammalian host along with tick saliva when the tick feeds, initiating a new cycle of parasite development.
end

tinyurl.com/mzdre9

Above Lymphoblasts- immature blood cells- goes to cancer- below


*** Lymphoblasts:



Go to page 7 - link below- for Theileria parasites 'hijack' the cellular machinery to drive lymphocyte proliferation.

www.theileria.org/downloads/cmi_925.pdf


At above link- there are 7 links to cancer. Here are 2:

Fair use

Excerpt:
However, we are far from understanding the
entire AP-1-regulated transcriptional programme that is
important in cell transformation and cancer progression. It
has been shown that Theileria-infected T lymphocytes
have increased amounts of TfR (CD71) on their surface...
end excerpt

Excerpt:

Thus, in Theileria-infected lymphocytes and in human
ovarian and breast cancer cells, augmented expression of
two different members of the Rab11-family is associated
with a transformed cell phenotype. Moreover, we observed
that Rab11A was not only increased, but when
compared with Rab2 (see Fig. 1), was also preferentially
associated with the membrane fraction and consequently
in an active GTP-bound form.
end excerpt

[Jill]

Just a couple thoughts-

They USE Baculovirus in cancer treatment: ((3 examples))

www.patentstorm.us/patents/6342216/claims.html

US Patent 6342216 - Therapy of cancer by insect cells containing recombinant baculovirus encoding genes


www.ncbi.nlm.nih.gov/pubmed/16775347

Enhanced baculovirus-mediated transduction of human cancer cells by tumor-homing peptides.


www.wjgnet.com/1007-9327/14/5810.pdf

Suppression of gastric cancer growth by baculovirus vector-
mediated transfer of normal epithelial cell specific-1

Excerpt:
CONCLUSION: Baculovirus-mediated NES1 gene can
be used in gene therapy for GC.
end

GC= Gastric cancer

[kammy]

Sept 5, 2009 14:23:13 GMT -5 Jill said:

Sept 5, 2009 11:13:48 GMT -5 kammy said:

How long is it going to take a scientist to find the cancer part of the baculovirus system, acquire the demographics of how many of us and how quickly cancer is developing inside our disease,

Kammy,

The T parva alters- hijacks- the blood cells.



www.tigr.org/tdb/e2k1/tpa1/intro.shtml

Fair use

Excerpt:

The infected lymphocytes grow larger and begin to divide. As each enlarged lymphocyte, called a lymphoblast, divides, the schizont inside the*** lymphoblast also divides, ensuring infection of each of the two daughter cells produced by the dividing lymphoblast. The infected lymphocytes expand rapidly and spread throughout the lymphoid system of the animal,
giving rise to widespread destruction of hostcells.

Some of the schizonts develop into merozoite forms, which are released from the lymphocytes into the bloodstream, where they invade red blood cells (erythrocytes).

In the red cells, the parasites develop into forms called piroplasms, which are able to infect ticks. As ticks feed on animals infected with the parasite, they ingest red blood cells containing the piroplasms. Once in the tick gut, the parasites differentiate into male and female gamonts, which fuse to form zygotes. The zygotes differentiate into kinetes, which move to the salivary gland and enter a particular cell type. Here the parasites form sporoblasts, each of which give rise to 30,000 to 50,000 sporozoites, a parasite form able to infect animals. The sporozoites are introduced into a mammalian host along with tick saliva when the tick feeds, initiating a new cycle of parasite development.
end

tinyurl.com/mzdre9

Above Lymphoblasts- immature blood cells- goes to cancer- below

The above diagram looks very familiar as to the process that is being used by certain 'spheres' in my photographs. I have always thought that I am seeing more than one sphere, I'm not sure because they have several assigned duties. That's why I'm at a stopping place, my work needs to be turned over to a professional, at this point.

I haven't read Robert Smith's work, if he's saying that he's seeing T-Parva in all cases of Morgellons that he's analyzed, then that's good enough for me.

If Robert Smith is saying that T parva is the cause of Morgellons, ask him if he's hatched one of these T parva cells yet, and if it flew away?

[Jill]

Kammy,

I have no contact with Robert Smith at all. His work was posted on various links by Kmarie and we took a stab at it years ago- here on LB. Having more time now- I decided to take another stab at it. Turns out it's straight-forward for the most part.

Theileria parva- is what it is. From what I'm reading, it is no doubt the cloned variety- from a vaccine to include the Baculovirus.

Other ID'd elements are tied to remediation products as I've mentioned. Several tie to the T parva.

T parva alone accounts for many if not most symptoms. Again, it is very doubtful that this is T parva as it is vectored from the ticks. That would cause a person to show rapid deterioration and early death. That is the other reason that it makes sense that the T parva ID'd by Smith is sourced from a vaccine- and consumed. Milk product or meat? IOW- a watered down variety.

I see where the Baculovirus has some interesting components.

Found the link you posted- where you can get tested? Or have specimens tested rather?

The feature of the Baculovirus- of interrupting w/ cell death
apoptosis is key, IMO. If the baculovirus is inhibiting cell death-
interfering with homeostasis according to abstracts- then my question is:

Is the debris we experience, clusters of cells? Possibly live cells?
Red blood cells?

Does the T parva, when it 'hijacks' the blood cells (per above post) does it create the artificial blood that Cliff Carnicom mentions?

More questions than answers....

[kammy]

Jill,

I'm going to go look at Robert Smith's work and see what he's up to, so I can understand better what you're trying to say. And, see what symptoms T parva is related to, specifically - it has to relate to 'specks', biofilm and fibers, to be considered what all people have in common. If it doesn't, then it is a secondary factor. If it does meet these criteria, then I have a photo of it, somewhere, unless it's elusive like the sponge.

I have an outer ear lesion that produces quite a bit of debris, I have pulled the debris out and photographed it, cultured it - there are no red blood cells in the debris in this type lesion. I don't know where the blood is that should be in this area of the ear? Live cells, most definitely, but, they are in a dessicated form.

I can't find where he says that all people with Morgellons have T parva, do you have a link, Jill? I see there's been Raman Graphs and chemical analysis done on the fibers - it's just a matter of one of them checking what they have against the GM fungus gnat frass, IMO. And, then whatever the differences are could be easily be determined by other known or suspected factors believed to be involved.

[lilsissy]

Sept 5, 2009 11:59:25 GMT -5 kammy said:

From Jen above:

"Development with no embryo.
also he wrote me that anabuse works on the zinc finger protein"

Who wrote this?

I've seen a reference to where anabuse might or helps with Morgellons. What do you know about this, Jen?

I wrote to Silentsuperbug sometime last year.

He recommended the soap and anabuse . He said if the Doctor asked me why Anabuse to tell him it work's on he zinc finger protein and it is anti-parasitic.

I myself do not know remember much of what I learned about the Zinc-finger protein but I always keep my eye open for it as a key word. I believe there is some literature about Z.F.P. and Lyme.

Apoptosis,
Cell death could possibly come into play to keep newly D.N.A. transformed cells from committing suicide or being killed and carried off , some cells commit suicide but I'm not sure if Human cells do.
I know slime mold cell commit suicide.

also,

I remember about 15 years ago, big reports all over the news came on about how scientist had discovered a way to stop aging ,
it would stop cells from dying off if (I remember right)
but we would die from cancer , I remember this for part for sure, CANCER ............ because we would then have cancer that sometimes develops that would not die off either.

If they could solve cancer it was said, then we could utilize their discovery and stop aging.

I do not know what this was all about for sure it has been a while .
I'm guessing it involved telomers and cell duplication.

Seems to me they would need a system to stop our killer cells if they are rearranging our D.N.A. , if a foreign code is giving by your new cell (now a D.N.A. transformed cell)
it must give the wrong code........ so what stops our body from killing them?



When I saw SilentsuperBug
....... no embryo....... it fit the development process. This poop is replicating without intimating with the oppisite sex , it seems. Self replication by imating that of nature but skipping the mating stage and embrionic envelope.

This stuff loves keratin (our Hair) which is often used in cross-linking of fibers ,
the stacks remember?
It loves keratin and keratin loves it.. So they naturally ,
STICK TOGETHER LIKE MAGNETS,
KERATIN IS IS OFTEN USED IN CROSS-LINKING FIBERS.

You guys I'm sorry I am having one heck of a messed up day.

I will try to reread and catch-up.

Jen

[lilsissy]

this shows usefullness by Z.F.P.'s in D.N.A. transcripiton.
www.patentstorm.us/patents/6453242/description.html



TECHNICAL FIELD

The invention resides in the technical fields of bioinformatics, and protein engineering.

BACKGROUND

Zinc finger proteins (ZFPs)are proteins that can bind to DNA in a sequence-specific manner. Zinc fingers were first identified in the transcription



I do not know anything much about Prava but the first time I heard of a human parva I was a little shocked because I did not know it could infect humans untill last spring.

Jen

[lilsissy]

I don't think that to many can argue that our government wants to chip us . The push for the chip was slowed probably because they were shown to give cancer.

So it would seem a massive effort could be underway to eliminate cancer itself and make us more integrable with technology.
This could provide the benefit of a long life for those who will be chipped.

www.nanotech-now.com/news.cgi?story_id=34199

Now their would be two motives to rid cancer , microchips and long life .

[lilsissy]

What's this looks so familar,

cilium , antenna, embroynic developement,
healthfully.org/medicalscience/id16.html
S.S.B said,
Qurom sensing .
Is it the same as cell sensing ? Anybody?


here the Fly and the embryo are discussed at te cell surface.
www.pubmedcentral.nih.gov/articlerender.fcgi?artid=1206327

One thing I believe for me is certain,
there is D.N.A. Mutation going on so advanced and rapid that it would be from a lab.

[kammy]

www.i-sis.org.uk/isisnews/i-sisnews9-28.php

"Gene Therapy with Your Salads, Anyone?

A virus is simultaneously being genetically modified to kill insect pests and to transfer genes into human cells in gene therapy. Prof. Joe Cummins points to a major gap in biosafety regulation.

Do our biosafety regulators know that a certain virus is being genetically modified to control plant disease and to serve as a gene carrier or vector for human gene therapy? This is the baculovirus, a virus previously thought to infect only insects, but has since found to get into all kinds of mammalian cells including those of human beings. Farm workers spraying the crop with anti-insect baculovirus and the public eating the crop not properly washed may both become genetically modified as a result.

Pests that infect and cause disease symptoms in both crops and human cells have never been described. Yet natural viruses that infects and slowly kills insects are also known to infect humans, but the infected humans do not seem to have symptoms. However, when the virus is genetically modified to eradicate insect pests, it may cause disease symptoms in those spraying crops or eating the sprayed crop. The baculovirus manipulated for insect control and for human gene therapy have both proven genetically unstable, and are prone to recombination and deletion at high frequency [1]. Such genetic instability has been noted repeatedly by those studying the virus, the genetic instability of the virus makes toying with it like playing with explosives.

Natural baculovirus, in contrast, is very stable and may remain dormant in the environment for years before infecting insects. The virus alone has a relatively low killing power and slow action. When a gene for a potent toxin such as scorpion toxin or a gene affecting a juvenile hormone is added to the virus, it kills faster and fewer insects survive infection. Numerous field tests of modified virus sprayed on crops have been done, despite protests from the public.

Soon after GM baculovirus was developed for insect-control, the virus was discovered to be capable of infecting human liver cells and produced relatively little toxicity to the infected cells. For that reason, baculovirus vectors were developed to treat liver disease, and even to transfer genes to the human brain [2]. The fact that baculovirus can infect human liver or brain cells seems to have been ignored by those developing the virus for commercial pest control. There has been a great deal of pressure to hasten approval of the GM baculovirus for pest control especially in the United States and Canada, where human populations have already been used as guinea pigs for GM crops.

Ecological impacts of recombinant baculovirus insecticides have focussed on baculovirus containing scorpion toxin because that construction has been most widely used [3]. Impacts on non-target insects are simply extrapolated from findings on insects of related phylogeny, a practice that is full of pitfalls, for simply adding and deleting genes can change the host range of the resultant baculovirus in unpredictable ways [4]. Furthermore, the recombinant baculoviruses were very persistent, and capable of reshaping an ecosystem.

The scorpion toxins used with recombinant baculovirus have been selected to avoid toxicity to humans, and as much as possible, to non-target animals. However, the allergenicity other harmful effects in human liver infection has not yet been investigated.

Recombinant baculoviruses have also been constructed containing other genes, such as those coding for Bacillus thuringiensis (bt) toxins [5], which are known to produce allergic reactions in human beings and also harmful to rats [6]. A recombinant baculovirus has even been constructed containing an antisense fragment to the c-myc oncogene [7]. The c-myc oncogene is a modified form of an essential cellular gene. Thus, the antisense gene, which contains a DNA sequence complementary to the gene itself, may end up inactivating an essential cellular function.

Baculovirus vectors efficiently transfer genes into human liver cells [8, 9]. Hybrid baculovirus-adenovirus vectors have also been used to deliver genes to human cells [10].

In conclusion baculovirus vectors are being used to control insect pests because they are effective and persist for a long time in the environment. Baculovirus vectors are also being used in gene therapy of human liver and brain. These areas of research seem to exist as two solitudes and the risks of one are not evaluated in the context of the other. We may be treated to liver and brain gene therapy with our salad whether we need it or not."

(Do we need to start a Foods With Baculovirus Thread?)

[kammy]

Sept 5, 2009 18:41:47 GMT -5 @lilsissy said:

I wrote to Silentsuperbug sometime last year.

He recommended the soap and anabuse . He said if the Doctor asked me why Anabuse to tell him it work's on he zinc finger protein and it is anti-parasitic.


Jen

What kind of soap, hon? I must have missed it?

[lilsissy]

this kind,
picasaweb.google.com/SILENTSUPERBUG/CBL001DETTOL02#5236529546771364866


dettol

[Jill]

Jen,

In his list or protocol for Morgellons, Garth Nicolson also recommends Dettol. Must be good stuff?

www.immed.org/treatment%20considerations/2.2.09Updates/MorgellonsDis09b.rtf

Excerpt:
4. Do not use lotions or perfumes; instead, use acne products for skin treatments. Lotions or perfumes tend to spread the condition to uninvolved areas. Instead, use Neutrogena Rapid Clear acne treatment pads, Isotrex gel topical acne treatment (containing isotretinoin, www.walgreens.com/). Hand sanitizer gels should be used when removing debris in sores. Medicated soaps should be used, or soaps such as Dead Sea salt soap for acne or Dettol bar soap (http://www.carbolicsoap.com/dettol).
end excerpt

Agree with you on the crystal structure of the Baculovirus - and because it is, I'm certain, in the specimen that Robert Smith ID'd- via vaccine, no doubt.

And the Zinc fingers- with their "purposefully re-engineered DNA-binding specificities" seem to be how they are able to 'target' any cell type- plant, animal or human to perform genomic manipulation.

IOW- key to our condition. I come across the Zinc finger proteins in various materials- per Robert Smith's research much of it having to do with T parva.

www.zincfingers.org/default2.htm

My greatest concern is seeing how the T parva takes the condition to cancer.

As to the vector- it could be water and/or insects. Any insect. Which insect found to vector in any given area is insignificant because it will vary.
Flies-spiders-whatever. The important issue is what is being vectored.

More later...

[Jill]

Kammy,

Why I believe that Robert Smith's report is right on target.... all the components tie in- to both symptoms and what is known and proven. A little research and you find it all ties perfectly.

Also- because the man has 2 DEGREES. Master in Chemistry and BS in Genetics. And he is motivated to find the cause.

T parva is a huge part of this, I'm convinced. First off- Harriett Bishop in the last Morgellons Conference stated that 97% of those with this condition have Lyme Disease. Prior to that, Dr Harvey gave the percentage as 94%. So, we can be certain that Lyme disease is a huge part of the condition. IMO, the fibers are a co-infection of the Lyme- but either way- Lyme is a given.

T parva overlaps with 2 Lyme bands- Western blot- 28 kDa and 47kDa . Babesia also overlaps with T parva - band 47 kDa so there is another tie in.

Other components that Robert Smith ID'd- Nocardioides sp. JS614- That is a Deep Fungal infection and another BINGO.

Norcardioides sp JS 614 is also used in remediation of sludge/wastewater AND in PM 1- which is another BINGO for the MTBE remediation products - Norcardioides sp JS 614 is also tied to:

www.americanelements.com/mobr.html

Excerpt:

# Two closely related pathways of nicotine catabolism in Arthrobacter nicotinovorans and Nocardioides sp. strain JS614. Arch Microbiol. 2008 May;189(5):511-7. Epub 2007 Dec 11.

( Arthrobacter nicotinovorans is an aerobic bacterium isolated from the soil around tobacco plants)


Speaking of PM 1 the MTBE remediation product, 3 more components ID'd by RS are in that particular product which is most likely in 70-80% of drinking water- Nationwide, to include:


* Azoarcus sp. EbN1 and it's split gene: Has a crystal structure of mandelate racemace/muconate- lactonizing family protein...ethylbenzene
(counting Azoarcus as only 1 component w/Ralstonia-split gene)
Ralstonia solanacearum GMI1000

* Neisseria gonorrhoeae FA 1090

* Chromobacterium violaceum ATCC 12472

And, of course, Agrobacterium Ti C58 which, as we know, can do that Transkingdom
tDNA transfer......

Agrobacterium ti - ID'd by Citovsky



As if that were not enough, the 30 kDa fluroescent protein that RS refers to as the unknown protein just may be:

The ONLY HIT on the Protein Data Bank:

30 kDa FLUORESCENT

1KIL
Three-dimensional structure of the complexin/SNARE complex

((more on the above to follow- explains the 'coiled' effect per Citovsky and much more))

Also mentioned in the report- RS:

www.uniprot.org/uniprot/P04264

Fair use

Excerpt:
Recommended name:
Keratin, type II cytoskeletal 1
Alternative name(s):
Cytokeratin-1
Short name=CK-1
Keratin-1
Short name=K1
67 kDa cytokeratin
Hair alpha protein

Homo sapiens (Human) [Complete proteome]


www.antibodies-online.com/antibody/337186/Keratin+Type+II+Cytoskeletal+1+67-Kda+KRT1+Human/

(( Robert Smith's find below))

Keratin, Type II Cytoskeletal 1 67-Kda (KRT1) (Human) antibody


which- is the same weigh- 67 kDa as the T parva Vaccine:

www.freshpatents.com/Theileria-parva-dna-vaccines-dt20060921ptan20060210537.php

Fair use
Excerpt:

A 67 kDa glycoprotein (p67) from the surface of the T. parva sporozoite has been isolated


((could be co-incidence?))

The above, IMO, is key. The 67 kDa and the finding of RS.

Could it be that the T parva is ON our skin? Is that the substance that creates the biofilm? Does the other bacteria DRAWN to the biofilm ON our skin draw insects?

To what degree are we protected by the P35 Baculovirus (?) that inhibits cell death?

Is the debris coming out of our skin- due to the P35 (in the T parva vaccine)- the crystals? Cells ?

Is this what is in the air per Cliff Carnicom ? Artificial blood ? We know that
T parva alters- HIJACKS the blood cells. Does it alter them to the degree that they resemble artificial blood?

More later....