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Post by jj on Sept 6, 2009 10:58:04 GMT -5
Anybody talk to Robert Smith yet?
Think he would be fascinated to see what his own words and research really means through the eyes of others.
JJ
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Post by camv35s on Sept 6, 2009 11:11:18 GMT -5
Hi Jill I remember a few years ago reading the posts of a man that had all the symptoms of morgellons and he worked at a sewage treatment site Norcardioides sp JS 614 is also used in remediation of sludge/wastewater AND in PM 1- which is another BINGO for the MTBE remediation products - Norcardioides sp JS 614 is also tied to: Best regards Cam
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Post by Jill on Sept 6, 2009 11:33:49 GMT -5
Agree, CAM- and I recall the postings about the man at the wastewater treatment plant as well. Thanks for bringing up the info source. Didn't his wife post? I do recall her stating that the bacteria used at the wastewater treatment plant WAS, in her opinion what caused this condition. Is this the man you are referring to, Cam ? www.morgellons-disease.info/Sufferers-Shares/April-Sufferer.html |
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Post by camv35s on Sept 6, 2009 11:54:20 GMT -5
hi jill ,Thats the one, Its remarkable that we both remembered that post .Best regard cam
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Post by Jill on Sept 6, 2009 12:24:00 GMT -5
I had remembered a different version of the story- told elsewhere but I believe it is the same people. Good information, Cam- thanks again! www.morgellons-disease.info/Sufferers-Shares/April-Sufferer.htmlFair use Except
Steve at 39 years old stricken with a form of RA specialists cannot figure out.....he has NO HISTORY of RA in his family. His non-response to traditional treatments even qualifying for PROSORBA treatments a blood dialysis (once a week for 12 weeks) and Steve completed the treatment twice (a total of 24 treatments) and was considered disabled..........I believe this SAVED HIS LIFE the blood dialysis filtered these organisms from his body.....relieving his liver that was in crisis at the time.....otherwise he was ready to go down that is how sick he was..... |
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Post by kammy on Sept 6, 2009 18:17:05 GMT -5
It's possible that some of the scientists are finding 94 and 97% pathogenic ratios in regard to our disease, however, what makes a disease a disease by definition, is what all people within that group have in common.
That's like saying, as a simple example, 97% of the people with cancer have abnormal cells, however 3% don't. From the sounds of it, it appears that T parva is a co-infection of Morgellons, but, not a cause.
I chose the three main symptoms that we all in common and went from there. If a scientist isolates one of the three, let's say fibers, and chooses to concentrate their work in that area, they are only going to get a partial answer with their work, unless, by chance - the fibers are the cause of the other two. I have shown that the fibers are the end result of a life cycle, not the beginning.
I haven't heard of anyone culturing the fibers and finding the baculovirus capsids as a result. In other words, those doing research on the fibers to find out what Morgellons is - are going about their research backwards and will never see what brought the fibers about. Carnicom has captured some of the BV capsids, the seeds of creation, with his process, that is why he is on track and close to discovery.
I still haven't found Robert Smith's work, the same holds true for Nocardioides sp. JS614, and the Rattus Norvegicus= Kil1= 30 kDa unless the percentage is 100%, these would also be considered a co-infection if they have high numbers. Are either of these, or any other pathogens that he's found to be at 100%, Jill?
Not to say that there can't be a combination of factors to cause a disease, however all factors have to be in common in 100% of the cases.
Of course, we appreciate Robert Smith's work, he is putting us on the fast track to be able to identify the pathogens involved and therefore, hopefully get quicker treatments.
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Post by kammy on Sept 6, 2009 19:34:15 GMT -5
To Date, Morgellons Is: The other known part of Morgellons, which has been proven to be present 100% – is related to agrobacterium. This aspect has been positively identified by Vitaly Citovsky who has already stated that this is at 100% in all cases. www.i-sis.org.uk/agrobacteriumAndMorgellons.php"They found that “all Morgellons patients screened to date have tested positive for the presence of Agrobacterium, whereas this microorganism has not been detected in any of the samples derived from the control, healthy individuals.” Agrobacterium would also be considered a primary factor also, if all three of the symptoms that people with Morgellons have in common; fibers, specks and biofilm is met, if not, it is a secondary factor. So, by using this sound logic, we can say – Morgellons is a combination of a GM insect baculovirus system with the addition of agrobacterium. It is also possible that there is a 3rd and 4th factor involved, we don’t know yet, unless the other scientists researching are reporting their findings at 100%? It will be by unraveling each one of these factors to find them at 100% that will be the entire cause. In my opinion, I’ve done the hard part, identified the main system, all that needs to be done is to compare what is present with the GM insect BV system and agrobacterium and then see what remains at 100% in all cases to find the other cause factors. |
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Post by lilsissy on Sept 8, 2009 23:25:40 GMT -5
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Post by kammy on Sept 9, 2009 1:58:07 GMT -5
@http://www.youtube.com/watch?v=ikR4ZUd_HZA
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Post by kammy on Sept 9, 2009 8:13:42 GMT -5
@http://www.youtube.com/watch?v=cQGwboUzRwo
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Post by toni on Sept 9, 2009 10:37:04 GMT -5
Great video's Kammy.
The TSO music makes it like a beautiful event, haha
( I love that orchestra).
It sure is interesting for sure, and you've put together
a some great stuff.
Thank you.
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Post by kammy on Sept 9, 2009 20:14:10 GMT -5
TY, Toni... yw...
IDENTIFICATION OF LARVAE IN YOUR SAMPLES
My friend dropped by tonight and wanted me to take a look at some of her samples with the microscope that she had in-between tape that came out of her skin, I learned something and thought I would share it. What's been so difficult to recognize in our samples - is what's what? From studying the many different ways this 'insect' morphs or transforms itself, I can now identify a larvae in the 'out of skin' samples. You'll notice that in these two samples below, you'll see a pointed 'head' end and then a blunt 'tail' end? I have witnessed this happening in front of my eyes under the microscope, that's how I know. The 'insect' whenever it is stressed turns back into a crystal/carbon capsid that has an elongated shape, with a pointed head end and a blunt tail end, and is hard to identify otherwise. Seeing these shapes below are evidence of larvae in your samples: Larvae in transition @100x:   Larvae in transition @100x:   |
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Post by kammy on Sept 9, 2009 22:24:25 GMT -5
I found this article interesting from 1997 talking about how crayfish in Australia now have a baculoviral disease which they have named CdSPV for Cherax destructor systemic parvo-like virus. en.scientificcommons.org/35942512"A systemic parvo-like virus in the freshwater crayfish Cherax destructor (1997) Systemic Cowdry Type A inclusions (CAs) were observed in a moribund Cherax destructor collected at an aquaculture farm in South Australia. Inclusions were most common in the gills and were associated with multifocal necrosis of the main gill axis and lamellae. The hepatopancreas was necrotic; however, only one CA was observed in the interstitial tissues. CAs were associated with necrosis in the abdominal and gut musculature. CAs were also observed in the spongy connective tissues and the epicardium. Empty capsids (17.5 +/- 0.5 nm) and microfilaments were most commonly observed within these inclusions by transmission electron microscopy. Complete icosahedral viral particles (20.8 +/- 1.2 nm) were difficult to distinguish within the viroplasm, but were visualised better in aggregates between the viroplasm and the inner nuclear membrane. The nucleolus was closely associated with the developing viroplasm, and was hypertrophied and segregated into its fibrillar and granular components. The virus was named Cherax destructor systemic parvo-like virus (CdSPV) on the basis of its histopathology, cytopathology and morphology. CdSPV is the first systemic virus described in a freshwater crayfish." **It seems that the baculovirus has been in use for some time in the various science arenas, and the fact that they are known to produce " microfilaments" has gone overlooked by everyone in the science community studying this disease?... and they are possibly known to be made out of something that is not found in data banks... that this might be one of the first places to look to compare our 'fibers' to - by someone in the know? |
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Post by jeany on Sept 10, 2009 5:08:55 GMT -5
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Post by jeany on Sept 10, 2009 5:11:34 GMT -5
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Post by kammy on Sept 10, 2009 5:30:22 GMT -5
The PiggyBac Gene Vector System www.ars.usda.gov/SP2UserFiles/person/5129/DEVELOPMENT%20OF%20PIGGYBAC.pdf"The piggyBac gene vector system has been used successfully to genetically transform ten insect species from three different insect orders and is being applied to others. It is the only gene vector that has been successfully applied to Lepidoptera. In an effort to explore some of the parameters that should be considered when utilizing this gene vector system, we have explored methods to optimize the production of transformants using the piggyBac gene vector system." piggybac.bio.nd.edu/default.htm" Why is it called piggyBac?I’m sure that question has crossed the minds of everyone who hears the name. Well, here’s the story. When I first identified these elements as insertions in Baculovirus mutants I began by naming them IFPx, for Insertion in FP mutant “x”. Upon sequencing one of these insertions and finding that it had characteristic features of a transposon, I named it TFP3, or Transposon in FP3. This nomenclature was a bit boring, and it was hard to attract the attention of anyone outside of Baculovirology to the significance of these elements. A colleague reviewing one of my papers wrote in his review (yes, he did reveal himself afterwards) that I might get more attention for my work if I named these elements along the lines of Drosophila transposon nomenclature rather than the boring “IFP such and such”. I’d never though to do that before, but I sat down and tried to come up with a logical nomenclature based upon the characteristics of these elements. All of them inserted into infecting Baculovirus genomes, and were then carried around in these viral genomes. So I hit upon “piggyback” as the name for the one element that I was most certain would be intact and interesting. To indicate the relationship to Baculoviruses, I dropped the “k” and capitalized the “B”,hence piggyBac. I proceeded to name additional elements as tagalong, hitchhiker, and even clingon. The only problem here is I’m running out of names. I’ll have to resort to alternative languages to name future elements if they are discovered. I am indebted to my reviewer for making the suggestion. If I had not changed the name, there would be a lot less interest. The simplicity of movement of this element, and often high frequency of transformation, make it an attractive tool for genetic manipulation of insects. We believe that these properties can be exploited for an even broader range of invertebrate species, and perhaps even vertebrate species, and are hopeful that more attempts will be made to utilize the element for transgenic studies." |
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Post by jeany on Sept 10, 2009 14:14:58 GMT -5
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Post by lilsissy on Sept 12, 2009 21:29:49 GMT -5
bump
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Post by kammy on Sept 14, 2009 12:41:31 GMT -5
What family is T. parva in? Is this our relationship to Lymes? www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=11991958In earlier investigations of baculovirus biology, a protein called GP64 was identified as the envelope fusion protein of AcMNPV and its close relatives (2, 3, 42, 47). Subsequently, it was discovered that GP64 is closely related (∼30% identity at the amino acid level) to the envelope fusion protein encoded by members of the genus en.wikipedia.org/wiki/Thogotovirus Thogotovirus (family Orthomyxoviridae), which are pathogenic for ticks and can also infect vertebrates (22, 27, 29, 33). This indicated that an envelope fusion protein either had been transferred between a minus-sense RNA virus and a double-stranded DNA virus or was acquired independently by each of these viruses from another source, such as a conserved host gene (29) (Fig. ​(Fig.11FIG. 1.) |
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Post by Jill on Sept 14, 2009 18:58:52 GMT -5
Thanks for the link Jen- appreciate the info! Jill |
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